M. Di Giambattista

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Factor VIII Inhibitors in Previously Treated Haemophilia A Patients with a Double Virus-inactivated Plasma Derived Factor VIII Concentrate

J. Arnout, J. Vermylen, K. Peerlinck, 1997, Thrombosis and Haemostasis.

Identification of a single base change in ribosomal RNA leading to erythromycin resistance.

P. Vannuffel, E. A. Morgan, M. Di Giambattista, 1992, The Journal of biological chemistry.

The role of rRNA bases in the interaction of peptidyltransferase inhibitors with bacterial ribosomes.

P. Vannuffel, M. Di Giambattista, C. Cocito, 1992, The Journal of biological chemistry.

Inhibition of protein synthesis by streptogramins and related antibiotics.

P. Vannuffel, M. Di Giambattista, C. Cocito, 1997, The Journal of antimicrobial chemotherapy.

Formation, isolation, and identification of products from the inactivation of virginiamycin M1 by Actinoplanes utahensis.

P. Vannuffel, P. Helquist, M. Di Giambattista, 1994, The Journal of antibiotics.

Affinity labeling of the virginiamycin S binding site on bacterial ribosome.

M. Di Giambattista, C. Cocito, A. Pecher, 1990, Biochemistry.

Action of erythromycin and virginiamycin S on polypeptide synthesis in cell-free systems.

M. Di Giambattista, C. Cocito, G. Chinali, 1988, Biochimica et biophysica acta.

Localization of virginiamycin S binding site on bacterial ribosome by fluorescence energy transfer.

M. Di Giambattista, C. Cocito, W. Möller, 1986, Biochemistry.

Chemical probing of a virginiamycin M-promoted conformational change of the peptidyl-transferase domain.

P. Vannuffel, M. Di Giambattista, C. Cocito, 1994, Nucleic acids research.

Interaction between virginiamycin S and ribosomes is partly provided by a salt bridge with a Mg2+ ion.

K. Clays, Y. Engelborghs, M. Di Giambattista, 1991, Biochemistry.

Hypoxia enables B19 erythrovirus to yield abundant infectious progeny in a pluripotent erythroid cell line.

Y. De Launoit, M. Di Giambattista, R. Laub, 2004, Journal of virological methods.

Analysis of the reversible binding of virginiamycin M to ribosome and particle functions after removal of the antibiotic.

M. Di Giambattista, C. Cocito, E. Nyssen, 1989, Biochimica et biophysica acta.

Inhibition of polypeptide synthesis in cell-free systems by virginiamycin S and erythromycin. Evidence for a common mode of action of type B synergimycins and 14-membered macrolides.

M. Di Giambattista, C. Cocito, G. Chinali, 1988, Biochimica et biophysica acta.

Modification of Factor VIII in Therapeutic Concentrates after Virus Inactivation by Solvent-Detergent and Pasteurisation

M. Di Giambattista, R. Laub, T. Barrowcliffe, 1998, Thrombosis and Haemostasis.

Does quantification of virus transcription provide a real measure of parvovirus B19 infectivity?

Y. De Launoit, M. Di Giambattista, R. Laub, 2006, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

An assay for parvovirus B19 neutralizing antibodies based on human hepatocarcinoma cell lines

Y. De Launoit, M. Di Giambattista, R. Laub, 2004, Transfusion.

Prevalence of human erythrovirus B19 DNA in healthy Belgian blood donors and correlation with specific antibodies against structural and non‐structural viral proteins

M. Di Giambattista, R. Laub, B. Latour, 2003, Vox sanguinis.

Inhibitors in German Hemophilia A Patients Treated with a Double Virus Inactivated Factor VIII Concentrate Bind to the C2 Domain of FVIII Light Chain

H. Brackmann, H. Lenk, M. Di Giambattista, 1999, Thrombosis and Haemostasis.

Mapping of natural anti-factor VIII antibodies in plasma pools from healthy donors: use of rationally designed synthetic peptides.

M. Di Giambattista, R. Laub, T. Branckaert, 2001, Biologicals : journal of the International Association of Biological Standardization.

The molecular basis of the inhibitory activities of type A and type B synergimycins and related antibiotics on ribosomes.

M. Di Giambattista, C. Cocito, G. Chinali, 1989, The Journal of antimicrobial chemotherapy.

A fluorescence lifetime study of virginiamycin S using multifrequency phase fluorometry.

K. Clays, A. Persoons, Y. Engelborghs, 1991, Biochemistry.

Kinetics of binding of macrolides, lincosamides, and synergimycins to ribosomes.

Y. Engelborghs, M. Di Giambattista, C. Cocito, 1987, The Journal of biological chemistry.

Analysis of fluorescence quenching of ribosome-bound virginiamycin S.

Y. Engelborghs, M. Di Giambattista, C. Cocito, 1984, Journal of Biological Chemistry.

Ribosome protection by tRNA derivatives against inactivation by virginiamycin M: evidence for two types of interaction of tRNA with the donor site of peptidyl transferase.

M. Di Giambattista, C. Cocito, G. Chinali, 1987, Biochemistry.

Continuous-flow UVC irradiation: a new, effective, protein activity-preserving system for inactivating bacteria and viruses, including erythrovirus B19.

Y. De Launoit, F. Sandras, M. Di Giambattista, 2004, Journal of virological methods.

Action of ions and pH on the binding of virginiamycin S to ribosomes.

M. Di Giambattista, C. Cocito, 1983, Biochimica et biophysica acta.

The lasting ribosome alteration produced by virginiamycin M disappears upon removal of certain ribosomal proteins.

M. Di Giambattista, C. Cocito, P. Moureau, 1983, Biochimica et biophysica acta.

Polychlorinated biphenyls and organochlorine pesticides are eliminated from therapeutic Factor VIII and immunoglobulin concentrates and reduced in albumin by plasma fractionation

A. Covaci, P. Schepens, M. Di Giambattista, 2002, Vox sanguinis.

Competition between erythromycin and virginiamycin for in vitro binding to the large ribosomal subunit.

M. Di Giambattista, C. Cocito, R. Parfait, 1981, Biochimica et biophysica acta.

In silico prediction of FVIII epitopes recognised by natural autoantibodies in polyvalent immunoglobulin concentrates.

M. Di Giambattista, R. Laub, G. Kemball-Cook, 2007, Molecular immunology.