T. Veveris-Lowe

Kallikrein 4 (hK4) and prostate-specific antigen (PSA) are associated with the loss of E-cadherin and an epithelial-mesenchymal transition (EMT)-like effect in prostate cancer cells.

J. Clements, T. Veveris-Lowe, M. Lawrence, 2005, Endocrine-related cancer.

The Journal of Clinical Endocrinology & Metabolism Printed in U.S.A. Copyright © 2000 by The Endocrine Society Placental Growth Hormone (GH), GH-Binding Protein, and Insulin-Like Growth Factor Axis in Normal, Growth- Retarded, and Diabetic Pregnancies: Co

D. Crane, K. Ho, F. Chan, 2022 .

Co-expression of GH and GHR isoforms in prostate cancer cell lines.

T. Veveris-Lowe, A. Herington, L. Chopin, 2002, Growth hormone & IGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society.

Growth hormone binding protein in normal and aneuploid pregnancy: a paradoxical decrease in trisomy 18

T. Veveris-Lowe, E. Wallace, D. D'Antona, 2001, BJOG : an international journal of obstetrics and gynaecology.

Seminal fluid characterization for male fertility and prostate cancer: kallikrein-related serine proteases and whole proteome approaches.

J. Clements, R. Gardiner, T. Veveris-Lowe, 2007, Seminars in thrombosis and hemostasis.

Hormone-dependent cancers: new approaches to identification of potential diagnostic and/or therapeutic biomarkers

J. Clements, T. Veveris-Lowe, L. Bui, 2010 .

Epithelial-Mesenchymal Transition in Prostate Cancer and the Potential Role of Kallikrein Serine Proteases

J. Clements, T. Veveris-Lowe, M. Lawrence, 2007, Cells Tissues Organs.

Placental growth hormone is not suppressed by oral glucose loading in normal human pregnancy.

D. Cowley, A. Cotterill, A. Russell, 2002, Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et metabolisme.

The role of kallikrein-related peptidases in prostate cancer: potential involvement in an epithelial to mesenchymal transition

J. Clements, T. Veveris-Lowe, M. Lawrence, 2006, Biological chemistry.

Kallikrein 4 (hK4) and prostate-specific antigen (PSA) are associated with the loss of E-cadherin and an epithelial-mesenchymal transition (EMT)-like effect in prostate cancer cells.

J. Clements, T. Veveris-Lowe, M. Lawrence, 2005, Endocrine-related cancer.

Expression of PSA-RP2, an alternatively spliced variant from the PSA gene, is increased in prostate cancer tissues but the protein is not secreted from prostate cancer cells

Robert Gardiner, H. Samaratunga, J. Clements, 2010, Biological chemistry.