F. Ji

Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures.

T. Yagi, M. Sanbo, H. Asada, 1996, Biochemical and biophysical research communications.

GABA and histogenesis in fetal and neonatal mouse brain lacking both the isoforms of glutamic acid decarboxylase

K. Obata, F. Ji, N. Kanbara, 1999, Neuroscience Research.

Development of the GABA system in organotypic culture of hippocampal and cerebellar slices from a 67-kDa isoform of glutamic acid decarboxylase (GAD67)-deficient mice

K. Obata, F. Ji, 1999, Neuroscience Research.

Postnatal development of a GABA deficit and disturbance of neural functions in mice lacking GAD65

K. Obata, S. Shibata, O. Stork, 2000, Brain Research.

Synaptic localization of the 67,000 mol. wt isoform of glutamate decarboxylase and transmitter function of GABA in the mouse cerebellum lacking the 65,000 mol. wt isoform

H. Mitoma, K. Obata, S. Konishi, 1999, Neuroscience.

Reduction of extracellular GABA in the mouse amygdala during and following confrontation with a conditioned fear stimulus

K. Obata, O. Stork, F. Ji, 2002, Neuroscience Letters.