S. Boillée

发表

System xC- is a mediator of microglial function and its deletion slows symptoms in amyotrophic lateral sclerosis mice.

D. Seilhean, C. Escartin, Pinar Mesci, 2015, Brain : a journal of neurology.

Main path and byways: non‐vesicular glutamate release by system xc− as an important modifier of glutamatergic neurotransmission

A. Massie, S. Hewett, S. Boillée, 2015, Journal of neurochemistry.

New advances in Amyotrophic Lateral Sclerosis genetics: Towards gene therapy opportunities for familial and young cases.

M. Amador, S. Millecamps, S. Boillée, 2021, Revue neurologique.

New insights on the disease contribution of neuroinflammation in amyotrophic lateral sclerosis.

C. Lobsiger, S. Boillée, Aude Chiot, 2019, Current opinion in neurology.

Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice

Minh N. H. Nguyen, R. H. Brown, A. McMahon, 2003, Science.

Ultrasound-Induced Blood-Spinal Cord Barrier Opening in Rabbits.

F. Salachas, C. Lafon, J. Chapelon, 2019, Ultrasound in medicine & biology.

Toxicity from different SOD1 mutants dysregulates the complement system and the neuronal regenerative response in ALS motor neurons

D. Cleveland, C. Lobsiger, S. Boillée, 2007 .

Heterozygous Tbk1 loss has opposing effects in early and late stages of ALS in mice

S. Akira, A. Ludolph, T. Satoh, 2019, The Journal of experimental medicine.

Early defect in the expression of mouse sperm DnaJ 1, a member of the DNAJ/heat shock protein 40 chaperone protein family, in the spinal cord of the wobbler mouse, a murine model of motoneuronal degeneration

M. Junier, S. Fasano, R. Meccariello, 2002, Neuroscience.

Transforming growth factor alpha: a promoter of motoneuron survival of potential biological relevance.

M. Junier, S. Boillée, M. Coulpier, 2001, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Transforming Growth Factor α: A Promoter of Motoneuron Survival of Potential Biological Relevance

M. Junier, S. Boillée, M. Coulpier, 2001, The Journal of Neuroscience.

Mutations in SQSTM1 encoding p62 in amyotrophic lateral sclerosis: genetics and neuropathology

V. Meininger, F. Salachas, C. Cazeneuve, 2013, Acta Neuropathologica.

Mitochondrial defect in muscle precedes neuromuscular junction degeneration and motor neuron death in CHCHD10S59L/+ mouse

E. Génin, D. Bohl, J. Ricci, 2019, Acta Neuropathologica.

The NADPH oxidase Nox2 regulates VEGFR1/CSF‐1R‐mediated microglial chemotaxis and promotes early postnatal infiltration of phagocytes in the subventricular zone of the mouse cerebral cortex

P. Carmeliet, K. Krause, Carmen Ruiz de Almodóvar, 2013, Glia.

C1q induction and global complement pathway activation do not contribute to ALS toxicity in mutant SOD1 mice

C. Pozniak, D. Cleveland, C. Lobsiger, 2013, Proceedings of the National Academy of Sciences.

Phenotype difference between ALS patients with expanded repeats in C9ORF72 and patients with mutations in other ALS-related genes

V. Meininger, F. Salachas, A. Brice, 2012, Journal of Medical Genetics.

Mutations in UBQLN2 are rare in French amyotrophic lateral sclerosis

V. Meininger, F. Salachas, A. Brice, 2012, Neurobiology of Aging.

ALS: A Disease of Motor Neurons and Their Nonneuronal Neighbors

D. Cleveland, S. Boillée, C. V. Velde, 2006, Neuron.

Revisiting oxidative damage in ALS: microglia, Nox, and mutant SOD1.

D. Cleveland, S. Boillée, 2008, The Journal of clinical investigation.

The Amyotrophic Lateral Sclerosis M114T PFN1 Mutation Deregulates Alternative Autophagy Pathways and Mitochondrial Homeostasis

V. Meininger, F. Salachas, P. Ravassard, 2022, International journal of molecular sciences.

Genetic analysis of SS18L1 in French amyotrophic lateral sclerosis

V. Meininger, F. Salachas, P. Couratier, 2014, Neurobiology of Aging.

Screening of OPTN in French familial amyotrophic lateral sclerosis

V. Meininger, F. Salachas, A. Brice, 2011, Neurobiology of Aging.

Onset and Progression in Inherited ALS Determined by Motor Neurons and Microglia

G. Kollias, N. Copeland, N. Jenkins, 2006, Science.

Deletion of the inflammatory S100-A9/MRP14 protein does not influence survival in hSOD1G93A ALS mice

M. Heneka, D. Bohl, C. Lobsiger, 2021, Neurobiology of Aging.

Local and remote interactions between macrophages and microglia in neurological conditions.

S. Boillée, 2021, Current opinion in immunology.

Modifying macrophages at the periphery has the capacity to change microglial reactivity and to extend ALS survival

D. Bohl, D. Seilhean, P. de la Grange, 2020, Nature Neuroscience.

Astrocytes as determinants of disease progression in inherited amyotrophic lateral sclerosis

D. Gutmann, R. Takahashi, D. Cleveland, 2008, Nature Neuroscience.

ALS-causing SOD1 mutants generate vascular changes prior to motor neuron degeneration

R. Deane, M. O’Banion, B. Zlokovic, 2008, Nature Neuroscience.

Author manuscript, published in "Journal of Neural Transmission 2010;117(8):981-1000" DOI: 10.1007/s00702-010-0429-0 A G Barbeito et al. Motor neuron-immune interactions Motor neuron- immune interactions: the vicious circle of ALS

Pinar Mesci, S. Boillée, A. Barbeito, 2010 .

The wobbler mouse

M. Peschanski, M. Junier, S. Boillée, 2003, Molecular Neurobiology.

Gene therapy for ALS delivers

D. Cleveland, S. Boillée, 2004, Trends in Neurosciences.

Genetic screening of ANXA11 revealed novel mutations linked to amyotrophic lateral sclerosis

Y. Marie, V. Meininger, F. Salachas, 2020, Neurobiology of Aging.

Impact of a frequent nearsplice SOD1 variant in amyotrophic lateral sclerosis: optimising SOD1 genetic screening for gene therapy opportunities

Y. Marie, V. Meininger, F. Salachas, 2021, Journal of Neurology, Neurosurgery, and Psychiatry.

Mutant SOD1 in cell types other than motor neurons and oligodendrocytes accelerates onset of disease in ALS mice

L. Goldstein, O. Mikse, D. Cleveland, 2008, Proceedings of the National Academy of Sciences.

Effects of a Single Head Exposure to GSM-1800 MHz Signals on the Transcriptome Profile in the Rat Cerebral Cortex: Enhanced Gene Responses Under Proinflammatory Conditions

J. Edeline, P. Leveque, D. Arnaud-Cormos, 2020, Neurotoxicity Research.

Schwann cells expressing dismutase active mutant SOD1 unexpectedly slow disease progression in ALS mice

D. Cleveland, K. Yamanaka, C. Lobsiger, 2009, Proceedings of the National Academy of Sciences.

Novel UBQLN2 mutations linked to amyotrophic lateral sclerosis and atypical hereditary spastic paraplegia phenotype through defective HSP70-mediated proteolysis

V. Meininger, F. Salachas, M. Amador, 2017, Neurobiology of Aging.

Neurofilament accumulations in amyotrophic lateral sclerosis patients’ motor neurons impair axonal initial segment integrity

F. Salachas, S. Blanchard, D. Bohl, 2023, Cellular and Molecular Life Sciences.

Differential microglial response to progressive neurodegeneration in the murine mutant wobbler

M. Peschanski, S. Boillée, P. Dreyfus, 2001, Glia.

The contribution of the peripheral immune system to neurodegeneration

C. Lobsiger, S. Boillée, Félix Berriat, 2023, Nature Neuroscience.

Neurofilament accumulations in amyotrophic lateral sclerosis patients’ motor neurons impair axonal initial segment integrity

F. Salachas, S. Blanchard, D. Bohl, 2023, Cellular and Molecular Life Sciences.

Effects of a Single Head Exposure to GSM-1800 MHz Signals on the Transcriptome Profile in the Rat Cerebral Cortex: Enhanced Gene Responses Under Proinflammatory Conditions

J. Edeline, D. Arnaud-Cormos, M. Mallat, 2020, Neurotoxicity Research.

Motor neuron–immune interactions: the vicious circle of ALS

Pinar Mesci, S. Boillée, A. Barbeito, 2010, Journal of Neural Transmission.

Inaugural Article: Toxicity from different SOD1 mutants dysregulates the complement system and the neuronal regenerative response in ALS motor neurons