J. Doorbar

J. Doorbar, K. Cuschieri, K. Kavanagh, 2018, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

Title : Stratification of HPV-Induced Cervical Pathology using the Virally-Encoded Molecular Marker E 4 in Combination with p 16 and MCM

W. Quint, A. Singer, R. Jach, 2015 .

Comprehensive control of human papillomavirus infections and related diseases.

M. Poljak, D. Forman, J. Cuzick, 2013, Vaccine.

One virus, one lesion—individual components of CIN lesions contain a specific HPV type

W. Quint, C. van Hoof, F. Struyf, 2012, The Journal of pathology.

Role of E6 in Maintaining the Basal Cell Reservoir during Productive Papillomavirus Infection

Ke-Jia Zheng, J. Doorbar, N. Egawa, 2022, Journal of virology.

Delta-Like Ligand–Notch1 Signaling Is Selectively Modulated by HPV16 E6 to Promote Squamous Cell Proliferation and Correlates with Cervical Cancer Prognosis

G. Kenter, R. D. de Menezes, J. Doorbar, 2021, Cancer Research.

Expression of p16 and HPV E4 on biopsy samples and methylation of FAM19A4 and miR124‐2 on cervical cytology samples in the classification of cervical squamous intraepithelial lesions

W. Quint, J. Ordi, D. Jenkins, 2020, Cancer medicine.

Title : Investigating Diagnostic Problems of CIN 1 and 2 Associated with High-Risk HPV by Combining the Novel Molecular Biomarker PanHPV E 4 with P 16 ink 4 a

Wim, H. Huras, K. Okoń, 2015 .

Analysis of HPV1 E4 complexes and their association with keratins in vivo.

S. Napthine, E. Medcalf, J. Doorbar, 1996, Virology.

Synthesis of viral DNA and late capsid protein L1 in parabasal spinous cell layers of naturally occurring benign warts infected with human papillomavirus type 1.

T. Iftner, K. Egawa, J. Doorbar, 2000, Virology.

Characterization of events during the late stages of HPV16 infection in vivo using high-affinity synthetic Fabs to E4.

S. Napthine, G. Winter, N. Coleman, 1997, Virology.

Improved detection reveals active β-papillomavirus infection in skin lesions from kidney transplant recipients

S. Landolfo, J. Doorbar, M. Gariglio, 2014, Modern Pathology.

Identification of the nuclear localization signal of human papillomavirus type 16 L1 protein.

I. Frazer, L. Crawford, C. McLean, 1991, Virology.

Biology of papillomavirus replication in infected epithelium

K. Raj, J. Doorbar, 2007 .

The E1 (cid:217) E4 Protein of Human Papillomavirus Type 16 Associates with a Putative RNA Helicase through Sequences in Its C Terminus

R. Elston, S. Stacey, S. Napthine, 2000 .

The CXCL12/CXCR4 Signaling Pathway: A New Susceptibility Factor in Human Papillomavirus Pathogenesis

Pasquale Cutolo, C. Deback, F. Bachelerie, 2016, PLoS pathogens.

Comprehensive control of human papillomavirus infections and related diseases.

M. Poljak, D. Forman, J. Cuzick, 2013, Vaccine.

Identification of proteins encoded by the L1 and L2 open reading frames of human papillomavirus 1a

J. Doorbar, P. H. Gallimore, 1987, Journal of virology.

Deregulation of E‐cadherin by human papillomavirus is not confined to high‐risk, cancer‐causing types

I. Nindl, J. Doorbar, M. Hibma, 2010, The British journal of dermatology.

Integrated analysis of cervical squamous cell carcinoma cohorts from three continents reveals conserved subtypes of prognostic significance

Mark N. Wass, Ian G. Reddin, Nerissa K. Kirkwood, 2021, Nature Communications.

Principles of epithelial homeostasis control during persistent human papillomavirus infection and its deregulation at the cervical transformation zone.

Yuwen Chen, J. Doorbar, N. Egawa, 2021, Current opinion in virology.

Host control of human papillomavirus infection and disease.

J. Doorbar, 2017, Best practice & research. Clinical obstetrics & gynaecology.

The high-risk HPV E6 target scribble (hScrib) is required for HPV E6 expression in cervical tumour-derived cell lines

L. Banks, J. Doorbar, P. Massimi, 2016, Papillomavirus research.

HPV8-E6 Interferes with Syntenin-2 Expression through Deregulation of Differentiation, Methylation and Phosphatidylinositide-Kinase Dependent Mechanisms

K. Hofmann, B. Akgül, J. Doorbar, 2017, Front. Microbiol..

The Viral E4 Protein Is Required for the Completion of the Cottontail Rabbit Papillomavirus Productive Cycle In Vivo

N. Christensen, N. Cladel, J. Doorbar, 2004, Journal of Virology.

Identification of a G(2) arrest domain in the E1 wedge E4 protein of human papillomavirus type 16.

J. Millar, K. Raj, J. Doorbar, 2002, Journal of virology.

Biology of the Human Papillomavirus Life Cycle: The Basis for Understanding the Pathology of PreCancer and Cancer

M. Stoler, C. Bergeron, J. Doorbar, 2020 .

E6AP is important for HPV E6’s role in regulating epithelial homeostasis and its loss impairs keratinocyte commitment to differentiation

Ke-Jia Zheng, J. Doorbar, N. Egawa, 2022, bioRxiv.

Grading immunohistochemical markers p16INK4a and HPV E4 identifies productive and transforming lesions caused by low‐ and high‐risk HPV within high‐grade anal squamous intraepithelial lesions

E. Pirog, D. Jenkins, J. Doorbar, 2019, British Journal of Dermatology.

The human papillomavirus type 11 E1/\E4 protein is not essential for viral genome amplification.

M. Howett, J. Doorbar, L. Budgeon, 2006, Virology.

Human papillomavirus type 16 E1circumflexE4 contributes to multiple facets of the papillomavirus life cycle.

P. Lambert, J. Doorbar, Tomomi Nakahara, 2005, Journal of virology.

Refining our understanding of cervical neoplasia and its cellular origins

J. Doorbar, H. Griffin, 2019, Papillomavirus research.

Latent papillomavirus infections and their regulation.

J. Doorbar, 2013, Current opinion in virology.

Sol-Gel Ceramic Pigments

A. Atkinson, J. Doorbar, D. Segal, 1998 .

Investigating Diagnostic Problems of CIN1 and CIN2 Associated With High-risk HPV by Combining the Novel Molecular Biomarker PanHPVE4 With P16INK4a

W. Quint, R. Jach, H. Huras, 2015, The American journal of surgical pathology.

Life Cycle Heterogeneity in Animal Models of Human Papillomavirus-Associated Disease

K. Sotlar, N. Christensen, W. Liu, 2002, Journal of Virology.

Human papillomavirus molecular biology and disease association

J. Doorbar, N. Egawa, H. Griffin, 2015, Reviews in medical virology.

Detection of viral DNA and E4 protein in basal keratinocytes of experimental canine oral papillomavirus lesions.

M. Stanley, D. Anderson, J. Doorbar, 2001, Virology.

Human Papillomavirus Type 16 E1∧E4-Induced G2 Arrest Is Associated with Cytoplasmic Retention of Active Cdk1/Cyclin B1 Complexes

P. Lambert, J. Millar, K. Raj, 2005, Journal of Virology.

E1∧E4 Protein of Human Papillomavirus Type 16 Associates with Mitochondria

P. Beard, K. Raj, J. Doorbar, 2004, Journal of Virology.

Functional Analysis of the Human Papillomavirus Type 16 E1∧E4 Protein Provides a Mechanism for In Vivo and In Vitro Keratin Filament Reorganization

P. McIntosh, M. Omary, P. Walker, 2004, Journal of Virology.

Detection of novel splicing patterns in a HPV16-containing keratinocyte cell line.

M. Stanley, T. Crook, L. Banks, 1990, Virology.

Sequence close to the N-terminus of L2 protein is displayed on the surface of bovine papillomavirus type 1 virions.

W. Liu, J. Doorbar, L. Gissmann, 1997, Virology.

Sequence divergence yet conserved physical characteristics among the E4 proteins of cutaneous human papillomaviruses.

J. Doorbar, P. Gallimore, P. H. Gallimore, 1989, Virology.

The Biology of Papillomavirus Latency

G. Maglennon, J. Doorbar, 2012, The open virology journal.

The papillomavirus life cycle.

J. Doorbar, 2005, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

Molecular biology of human papillomavirus infection and cervical cancer.

J. Doorbar, John Doorbar, 2006, Clinical science.

Human Papillomaviruses; Epithelial Tropisms, and the Development of Neoplasia

K. Egawa, J. Doorbar, N. Egawa, 2015, Viruses.

Human Beta‐papillomavirus infection and keratinocyte carcinomas

M. Feltkamp, J. Bouwes Bavinck, R. Genders, 2015, The Journal of pathology.

The biology and life-cycle of human papillomaviruses.

Wim Quint, W. Quint, M. Stanley, 2012, Vaccine.

E4 Antibodies Facilitate Detection and Type-Assignment of Active HPV Infection in Cervical Disease

W. Quint, C. van Hoof, F. Struyf, 2012, PloS one.

Structural Analysis Reveals an Amyloid Form of the Human Papillomavirus Type 16 E1∧E4 Protein and Provides a Molecular Basis for Its Accumulation

P. McIntosh, K. Raj, J. Eccleston, 2008, Journal of Virology.

Human Papillomavirus Type 16 E1∧E4 Contributes to Multiple Facets of the Papillomavirus Life Cycle

P. Lambert, J. Doorbar, Tomomi Nakahara, 2005, Journal of Virology.

Mouse Papillomavirus L1 and L2 Are Dispensable for Viral Infection and Persistence at Both Cutaneous and Mucosal Tissues

S. Brendle, N. Christensen, N. Cladel, 2021, Viruses.

Pathogenesis of Papillomaviruses in Humans

J. Doorbar, 2015 .

Epitope-mapped monoclonal antibodies against the HPV16E1--E4 protein.

L. Crawford, N. Coleman, D. Davies, 1992, Virology.

Human papillomavirus infection and induction of neoplasia: a matter of fitness.

J. Doorbar, C. Kranjec, Christian Kranjec, 2016, Current opinion in virology.

Depletion of Langerhans Cells in Human Papillomavirus Type 16-Infected Skin Is Associated with E6-Mediated Down Regulation of E-Cadherin

J. Doorbar, B. Bäckström, M. Hibma, 2003, Journal of Virology.

Model systems of human papillomavirus‐associated disease

J. Doorbar, 2016, The Journal of pathology.

Identification of a G2 Arrest Domain in the E1∧E4 Protein of Human Papillomavirus Type 16

J. Millar, K. Raj, J. Doorbar, 2002, Journal of Virology.

Whole tissue cervical mapping of HPV infection: Molecular evidence for focal latent HPV infection in humans

W. Quint, H. Svanholm, J. Doorbar, 2019, Papillomavirus research.

Immunosuppression Facilitates the Reactivation of Latent Papillomavirus Infections

P. McIntosh, G. Maglennon, J. Doorbar, 2013, Journal of Virology.

ONCOGENIC HUMAN PAPILLOMAVIRUSES

T. Kanda, P. Gariglio, J. Doorbar, 2012, The open virology journal.

Papillomavirus Life Cycle Organization and Biomarker Selection

J. Doorbar, John Doorbar, 2007, Disease markers.

Specific interaction between HPV-16 E1–E4 and cytokeratins results in collapse of the epithelial cell intermediate filament network

L. Crawford, C. McLean, J. Doorbar, 1991, Nature.

Identification of the human papilloma virus‐1a E4 gene products.

J. Doorbar, R. Grand, P. H. Gallimore, 1986, The EMBO journal.

Organization of Human Papillomavirus Productive Cycle during Neoplastic Progression Provides a Basis for Selection of Diagnostic Markers

Lesley Morris, Nicholas Coleman, P. Lambert, 2003, Journal of Virology.

Expression of Betapapillomavirus Oncogenes Increases the Number of Keratinocytes with Stem Cell-Like Properties

I. Mackenzie, B. Akgül, H. Pfister, 2013, Journal of Virology.

Presence of human papillomavirus in semen of healthy men is firmly associated with HPV infections of the penile epithelium.

J. Berkhof, C. Meijer, P. Snijders, 2015, Fertility and sterility.

Comprehensive control of human papillomavirus infections and related diseases.

M. Plummer, M. Poljak, J. Ferlay, 2013, Vaccine.

The E4 protein; structure, function and patterns of expression.

J. Doorbar, 2013, Virology.

Phosphorylation of the human papillomavirus type 1 E4 proteins in vivo and in vitro.

J. Doorbar, K. Smith, R. Grand, 1989, Virology.

Intrabody strategies for the treatment of human papillomavirus-associated disease

J. Doorbar, H. Griffin, 2007, Expert opinion on biological therapy.

Expression of papillomavirus L1 proteins regulated by authentic gene codon usage is favoured in G2/M-like cells in differentiating keratinocytes.

I. Frazer, Liang Zhao, K. Zhao, 2010, Virology.

Humans with inherited T cell CD28 deficiency are susceptible to skin papillomaviruses but are otherwise healthy

Mark M. Davis, J. Casanova, L. Notarangelo, 2021, Cell.

Analysis of HPV‐1 E4 gene expression using epitope‐defined antibodies.

L. Crawford, J. Doorbar, P. H. Gallimore, 1988, The EMBO journal.

Verrucous pilar cysts infected with beta human papillomavirus

A. Mir, Richard C. Wang, J. Doorbar, 2020, Journal of cutaneous pathology.

α- and β-papillomavirus infection in a young patient with an unclassified primary T-cell immunodeficiency and multiple mucosal and cutaneous lesions.

R. Badolato, R. Kumar, J. Doorbar, 2014, Journal of the American Academy of Dermatology.

The low-risk papillomaviruses.

J. Doorbar, N. Egawa, Nagayasu Egawa, 2017, Virus research.

Inhibition of papillomavirus protein function in cervical cancer cells by intrabody targeting.

R. Elston, G. Winter, J. Doorbar, 2006, Journal of molecular biology.

Carcinogenic human papillomavirus infection

Nicolas Wentzensen, Mark Schiffman, Carole Fakhry, 2016, Nature Reviews Disease Primers.

p53 controls expression of the DNA deaminase APOBEC3B to limit its potential mutagenic activity in cancer cells

Simak Ali, R. Coombes, M. Periyasamy, 2017, Nucleic acids research.

University of Dundee p 53 controls expression of the DNA deaminase APOBEC 3 B to limit its potential mutagenic activity in cancer cells

Simak Ali, M. Periyasamy, F. Fuller-Pace, 2017 .

Summary from an international cancer seminar focused on human papillomavirus (HPV)-positive oropharynx cancer, convened by scientists at IARC and NCI.

M. Carrington, F. Bray, S. Chanock, 2020, Oral oncology.

Serine/Arginine-Rich Splicing Factor 3 and Heterogeneous Nuclear Ribonucleoprotein A1 Regulate Alternative RNA Splicing and Gene Expression of Human Papillomavirus 18 through Two Functionally Distinguishable cis Elements

Zhi-Ming Zheng, J. Doorbar, M. Ajiro, 2016, Journal of Virology.

Etiological role of human papillomavirus infection for inverted papilloma of the bladder

T. Sasagawa, J. Doorbar, Y. Kitagawa, 2011, Journal of medical virology.

Modulation of basal cell fate during productive and transforming HPV‐16 infection is mediated by progressive E6‐driven depletion of Notch

R. Mahmood, J. Doorbar, H. Griffin, 2017, The Journal of pathology.

HPV E4 expression and DNA hypermethylation of CADM1, MAL, and miR124-2 genes in cervical cancer and precursor lesions

Peter J. F. Snijders, Chris J. L. M. Meijer, Johannes Berkhof, 2018, Modern Pathology.

The Role of Human Papillomaviruses and Polyomaviruses in BRAF-Inhibitor Induced Cutaneous Squamous Cell Carcinoma and Benign Squamoproliferative Lesions

M. Feltkamp, G. Inman, C. Proby, 2018, Front. Microbiol..

Human Papillomavirus type 16 Causes a Defined Subset of Conjunctival In Situ Squamous Cell Carcinomas

W. Quint, R. Mahmood, I. Rennie, 2019, Modern Pathology.

Stabilization of HPV16 E6 protein by PDZ proteins, and potential implications for genome maintenance.

L. Banks, K. Raj, J. Doorbar, 2011, Virology.

G2/M cell cycle arrest in the life cycle of viruses

J. Doorbar, Clare E Davy, Clare E. Davy, 2007, Virology.

HPV16 E1--E4 protein is phosphorylated by Cdk2/cyclin A and relocalizes this complex to the cytoplasm.

P. McIntosh, J. Doorbar, M. Ayub, 2006, Virology.

Human Papillomavirus Type 8 Interferes with a Novel C/EBPβ-Mediated Mechanism of Keratinocyte CCL20 Chemokine Expression and Langerhans Cell Migration

S. Smola, H. Pfister, C. Wickenhauser, 2012, PLoS pathogens.

A novel interaction between the human papillomavirus type 16 E2 and E1--E4 proteins leads to stabilization of E2.

P. McIntosh, J. Doorbar, Y. Soneji, 2009, Virology.

Human Papillomavirus Type 16 E 1 ∧ E 4-Induced G 2 Arrest Is Associated with Cytoplasmic Retention of Active Cdk 1 / Cyclin B 1 Complexes

P. Lambert, J. Millar, K. Raj, 2005 .

The E1∧E4 Protein of Human Papillomavirus Type 16 Associates with a Putative RNA Helicase through Sequences in Its C Terminus

R. Elston, S. Stacey, S. Napthine, 2000, Journal of Virology.

The identification of a conserved binding motif within human papillomavirus type 16 E6 binding peptides, E6AP and E6BP.

R. Elston, S. Napthine, J. Doorbar, 1998, The Journal of general virology.

Roles for E1-independent replication and E6-mediated p53 degradation during low-risk and high-risk human papillomavirus genome maintenance

T. Kiyono, J. Doorbar, Tomomi Nakahara, 2019, PLoS pathogens.

HPV16 and 18 genome amplification show different E4-dependence, with 16E4 enhancing E1 nuclear accumulation and replicative efficiency via its cell cycle arrest and kinase activation functions

R. Mahmood, J. Doorbar, N. Egawa, 2017, PLoS pathogens.

Performance of HPV E4 and p16INK4a biomarkers in predicting regression of cervical intraepithelial neoplasia grade 2 (CIN2): protocol for a historical cohort study

W. Quint, M. Stoler, D. Jenkins, 2022, BMJ open.

Characterization of cervical biopsies of women with HIV and HPV co-infection using p16ink4a, ki-67 and HPV E4 immunohistochemistry and DNA methylation

C. Meijer, J. Doorbar, L. Rozendaal, 2020, Modern Pathology.

Infection to Cancer—Finding Useful Biomarkers for Predicting Risk of Progression to Cancer

C. Meijer, J. Doorbar, A. Lorincz, 2020 .

Stratification of HPV-Induced Cervical Pathology using the Virally-Encoded Molecular Marker E4 in Combination with p16 or MCM

W. Quint, A. Singer, R. Jach, 2015, Modern Pathology.

Molecular Basis for Advances in Cervical Screening

H. Cubie, J. Doorbar, 2012, Molecular Diagnosis.

Role of Calpain in the Formation of Human Papillomavirus Type 16 E1^E4 Amyloid Fibers and Reorganization of the Keratin Network

P. McIntosh, J. Doorbar, Clare E Davy, 2011, Journal of Virology.

Global prevention and management of HPV related diseases : the pressing challenges and the compelling opportunities

M. Poljak, D. Forman, J. Cuzick, 2014 .

Comprehensive control of human papillomavirus infections and related diseases.

M. Poljak, D. Forman, J. Cuzick, 2013, Vaccine.

Inhibition of E6-induced degradation of its cellular substrates by novel blocking peptides.

R. Elston, P. McIntosh, L. Banks, 2004, Journal of molecular biology.

Pro-metastatic gene expression, immune evasion and an altered HPV spectrum characterize an aggressive subtype of cervical cancer

Ian G. Reddin, Nerissa K. Kirkwood, S. Henderson, 2020 .

Natural History and Biology of Human Papillomaviruses

J. Doorbar, 2016 .

Dynamics of papillomavirus in vivo disease formation & susceptibility to high-level disinfection-Implications for transmission in clinical settings.

R. Crawford, J. Yarwood, J. Doorbar, 2021, EBioMedicine.

Characterization of skin lesions induced by skin‐tropic α‐ and β‐papillomaviruses in a patient with epidermodysplasia verruciformis

J. Bouwes Bavinck, R. Genders, J. Doorbar, 2014, The British journal of dermatology.

Characterization of beta papillomavirus E4 expression in tumours from Epidermodysplasia Verruciformis patients and in experimental models.

H. Pfister, S. Landolfo, J. Doorbar, 2012, Virology.

Loss of epidermal Langerhans cells occurs in human papillomavirus alpha, gamma, and mu but not beta genus infections.

I. Nindl, J. Doorbar, H. Yoon, 2010, The Journal of investigative dermatology.

The early detection of cervical cancer. The current and changing landscape of cervical disease detection

R. Crawford, J. Doorbar, N. Egawa, 2020, Cytopathology : official journal of the British Society for Clinical Cytology.

The preference of women living with HIV for the HPV self-sampling of urine at a rural HIV clinic in Uganda

L. Atuyambe, J. Doorbar, Aslam Shiraz, 2022, Southern African Journal of Infectious Diseases.

Identification of the skin virome in a boy with widespread human papillomavirus‐2‐positive warts that completely regressed after administration of tetravalent human papillomavirus vaccine

F. Mignone, A. Matteelli, J. Doorbar, 2015, The British journal of dermatology.

Identification of the skin virome in a young boy with widespread HPV2-positive warts that completely regressed after administration of tetravalent human papillomavirus vaccine Running title: HPV2 variant in warts and vaccine outcome

F. Mignone, A. Matteelli, J. Doorbar, 2022 .

Cervical cell lift: A novel triage method for the spatial mapping and grading of precancerous cervical lesions

P. Sasieni, R. Crawford, J. Doorbar, 2022, EBioMedicine.

Erratum: Improved detection reveals active β-papillomavirus infection in skin lesions from kidney transplant recipients (Modern Pathology (2014) 27 (917) DOI:10.1038/modpathol.2014.2)

S. Landolfo, J. Doorbar, M. Gariglio, 2014 .

Reconstruction of Human Papillomavirus Type 16-Mediated Early-Stage Neoplasia Implicates E6/E7 Deregulation and the Loss of Contact Inhibition in Neoplastic Progression

N. Coleman, I. Roberts, J. Doorbar, 2012, Journal of Virology.

E1^E4-mediated keratin phosphorylation and ubiquitylation: a mechanism for keratin depletion in HPV16-infected epithelium

P. McIntosh, J. Doorbar, H. Griffin, 2010, Journal of Cell Science.

HPV-18 E2^E4 chimera: 2 new spliced transcripts and proteins induced by keratinocyte differentiation.

J. Gunaratne, F. Bachelerie, J. Doorbar, 2012, Virology.

Interaction of the Human Papillomavirus E6 Oncoprotein with Sorting Nexin 27 Modulates Endocytic Cargo Transport Pathways

D. Pim, L. Banks, J. Manzo-Merino, 2016, PLoS pathogens.

Mus musculus papillomavirus 1 E8^E2 represses expression of late protein E4 in basal-like keratinocytes via NCoR/SMRT-HDAC3 co-repressor complexes to enable wart formation in vivo

T. Iftner, F. Stubenrauch, R. Roden, 2023, mBio.

Pro-metastatic gene expression, immune evasion and an altered HPV spectrum characterize an aggressive subtype of cervical cancer

Ian G. Reddin, Nerissa K. Kirkwood, S. Henderson, 2020, bioRxiv.

HPV E6 inhibits E6AP to regulate epithelial homeostasis by modulating keratinocyte differentiation commitment and YAP1 activation

Ke-Jia Zheng, J. Doorbar, N. Egawa, 2023, PLoS pathogens.

Molecular Oncology: Cervical cancer

J. Doorbar, 2013 .

Inhibition of HPV-16 E7 oncogenic activity by HPV-16 E2

L. Banks, N. Gammoh, J. Doorbar, 2009, Oncogene.

The human Papillomavirus twilight zone – Latency, immune control and subclinical infection

J. Doorbar, 2023, Tumour virus research.

Phosphorylation of the Human Papillomavirus Type 16 E1^E4 Protein at T57 by ERK Triggers a Structural Change That Enhances Keratin Binding and Protein Stability

P. McIntosh, S. Howell, J. Doorbar, 2009, Journal of Virology.

Continuous Ink-Jet Printing Using Sol-Gel “Ceramic” Inks

A. Atkinson, J. Doorbar, A. Hudd, 1997 .

Whole tissue cervical mapping of HPV infection: Molecular evidence for focal latent HPV infection in humans

W. Quint, H. Svanholm, J. Doorbar, 2019, Papillomavirus research.

Human Papillomaviruses

J. Doorbar, C. John, Clare E. Davy, 2006, Methods in Molecular Medicine.

One virus, one lesion—individual components of CIN lesions contain a specific HPV type

W. Quint, C. van Hoof, F. Struyf, 2012, The Journal of pathology.

Human Beta‐papillomavirus infection and keratinocyte carcinomas

M. Feltkamp, J. Bouwes Bavinck, R. Genders, 2015, The Journal of pathology.

Host control of human papillomavirus infection and disease.

J. Doorbar, 2017, Best practice & research. Clinical obstetrics & gynaecology.

Loss of epidermal Langerhans cells occurs in human papillomavirus alpha, gamma, and mu but not beta genus infections.

I. Nindl, J. Doorbar, H. Yoon, 2010, The Journal of investigative dermatology.

Persistence of viral DNA in the epithelial basal layer suggests a model for papillomavirus latency following immune regression

P. McIntosh, G. Maglennon, J. Doorbar, 2011, Virology.

Risk stratification of cervical disease using detection of human papillomavirus (HPV) E4 protein and cellular MCM protein in clinical liquid based cytology samples

J. Doorbar, K. Cuschieri, K. Kavanagh, 2018, Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology.

The prevalence and clinical relevance of HPV in semen

J. Berkhof, C. Meijer, P. Snijders, 2022 .

Structural Analysis Reveals an Amyloid Form of the Human Papillomavirus Type 16 E1∧E4 Protein and Provides a Molecular Basis for Its Accumulation

P. McIntosh, K. Raj, J. Eccleston, 2008, Journal of Virology.

Investigating Diagnostic Problems of CIN1 and CIN2 Associated With High-risk HPV by Combining the Novel Molecular Biomarker PanHPVE4 With P16INK4a

W. Quint, R. Jach, K. Okoń, 2015, The American journal of surgical pathology.

Expression of papillomavirus L1 proteins regulated by authentic gene codon usage is favoured in G2/M-like cells in differentiating keratinocytes.

I. Frazer, Liang Zhao, K. Zhao, 2010, Virology.