J. Wegiel

发表

Contribution of glial cells to the development of amyloid plaques in Alzheimer’s disease

J. Wegiel, V. Venkataraman, R. Nagele, 2004, Neurobiology of Aging.

Stereological study of the neuronal number and volume of 38 brain subdivisions of subjects diagnosed with autism reveals significant alterations restricted to the striatum, amygdala and cerebellum

J. Wegiel, W. Brown, T. Wisniewski, 2014, Acta Neuropathologica Communications.

Neuronal nucleus and cytoplasm volume deficit in children with autism and volume increase in adolescents and adults

P. Hof, J. Wegiel, W. Brown, 2015, Acta Neuropathologica Communications.

Abnormal Intracellular Accumulation and Extracellular Aβ Deposition in Idiopathic and Dup15q11.2-q13 Autism Spectrum Disorders

Thomas Wisniewski, Edwin H. Cook, Michael Flory, 2012, PloS one.

Brain-region–specific alterations of the trajectories of neuronal volume growth throughout the lifespan in autism

J. Wegiel, W. Brown, T. Wisniewski, 2014, Acta Neuropathologica Communications.

Origin and turnover of microglial cells in fibrillar plaques of APPsw transgenic mice

J. Wegiel, R. Rubenstein, M. Osuchowski, 2003, Acta Neuropathologica.

Media from Rhabdomyosarcoma and Neuroblastoma Cell Cultures Stimulate in Vitro Aggregation and Fibrillization of Amyloid Beta-Protein

H. Wiśniewski, A. Chauhan, V. Chauhan, 1996, Neurochemical Research.

Fibrillar amyloid beta-protein forms a membrane-like hydrophobic domain.

A. Chauhan, V. Chauhan, J. Wegiel, 2001, Neuroreport.

Partial Agenesis and Hypoplasia of the Corpus Callosum in Idiopathic Autism

J. Wegiel, W. Brown, T. Wisniewski, 2017, Journal of neuropathology and experimental neurology.

The neuropathology of autism: defects of neurogenesis and neuronal migration, and dysplastic changes

Thomas Wisniewski, W. Ted Brown, Ira L. Cohen, 2010, Acta Neuropathologica.

The role of overexpressed DYRK1A protein in the early onset of neurofibrillary degeneration in Down syndrome

J. Wegiel, B. Reisberg, T. Wisniewski, 2008, Acta Neuropathologica.

Deficit of corpus callosum axons, reduced axon diameter and decreased area are markers of abnormal development of interhemispheric connections in autistic subjects

J. Wegiel, T. Wisniewski, V. Martínez‐Cerdeño, 2018, Acta neuropathologica communications.

Link Between DYRK1A Overexpression and Several-Fold Enhancement of Neurofibrillary Degeneration With 3-Repeat Tau Protein in Down Syndrome

J. Wegiel, B. Reisberg, M. D. de Leon, 2011, Journal of neuropathology and experimental neurology.

Neuronal nucleus and cytoplasm volume deficit in children with autism and volume increase in adolescents and adults

P. Hof, J. Wegiel, W. Brown, 2015, Acta neuropathologica communications.

Developmental deficits and staging of dynamics of age associated Alzheimer’s disease neurodegeneration and neuronal loss in subjects with Down syndrome

J. Wegiel, M. D. de Leon, W. Silverman, 2022, Acta neuropathologica communications.

Cells of monocyte/microglial lineage are involved in both microvessel amyloidosis and fibrillar plaque formation in APPsw tg mice

J. Wegiel, R. Rubenstein, H. Imaki, 2004, Brain Research.

Intraneuronal Aβ immunoreactivity is not a predictor of brain amyloidosis-β or neurofibrillary degeneration

T. Lehtimäki, J. Wegiel, B. Reisberg, 2007, Acta Neuropathologica.

Significant neuronal soma volume deficit in the limbic system in subjects with 15q11.2-q13 duplications

E. Cook, J. Wegiel, M. Casanova, 2015, Acta neuropathologica communications.

Brain-region–specific alterations of the trajectories of neuronal volume growth throughout the lifespan in autism

J. Wegiel, W. Brown, T. Wisniewski, 2014, Acta neuropathologica communications.

Contribution of olivofloccular circuitry developmental defects to atypical gaze in autism

E. Azmitia, J. Wegiel, Probal Banerjee, 2013, Brain Research.

Differences Between the Pattern of Developmental Abnormalities in Autism Associated With Duplications 15q11.2-q13 and Idiopathic Autism

Thomas Wisniewski, Boleslaw Lach, Edwin H. Cook, 2012, Journal of neuropathology and experimental neurology.

Type, Topography, and Sequelae of Neuropathological Changes Shaping Clinical Phenotype of Autism

J. Wegiel, W. Brown, T. Wisniewski, 2009 .

Clinicopathological Staging of Dynamics of Neurodegeneration and Neuronal Loss in Alzheimer Disease.

J. Wegiel, B. Reisberg, M. D. de Leon, 2020, Journal of neuropathology and experimental neurology.

Multiregional Age-Associated Reduction of Brain Neuronal Reserve Without Association With Neurofibrillary Degeneration or &bgr;-Amyloidosis

J. Wegiel, B. Reisberg, M. D. de Leon, 2017, Journal of neuropathology and experimental neurology.

Delayed Development of the Claustrum in Autism

J. Wegiel, W. Brown, T. Wisniewski, 2014 .

Stereological study of the neuronal number and volume of 38 brain subdivisions of subjects diagnosed with autism reveals significant alterations restricted to the striatum, amygdala and cerebellum

J. Wegiel, W. Brown, T. Wisniewski, 2014, Acta neuropathologica communications.

Clinicopathological Stratification of Idiopathic Autism and Autism with 15q11.2–q13 Duplications

E. Cook, J. Wegiel, W. Brown, 2013 .

The role of reduced expression of fragile X mental retardation protein in neurons and increased expression in astrocytes in idiopathic and syndromic autism (duplications 15q11.2‐q13)

J. Wegiel, W. Brown, T. Wisniewski, 2018, Autism research : official journal of the International Society for Autism Research.

Deficit of corpus callosum axons, reduced axon diameter and decreased area are markers of abnormal development of interhemispheric connections in autistic subjects

J. Wegiel, T. Wisniewski, V. Martínez‐Cerdeño, 2018, Acta Neuropathologica Communications.

Contribution of the serotonergic system to developmental brain abnormalities in autism spectrum disorder.

J. Wegiel, E. London, Jerzy Wegiel, 2024, Autism research : official journal of the International Society for Autism Research.