Bing Gong

发表

Nicotinamide riboside restores cognition through an upregulation of proliferator-activated receptor-γ coactivator 1α regulated β-secretase 1 degradation and mitochondrial gene expression in Alzheimer's mouse models

G. Pasinetti, L. Ho, M. Sastre, 2013, Neurobiology of Aging.

Ghrelin promotes midbrain neural stem cells differentiation to dopaminergic neurons through Wnt/β‐catenin pathway

Hong Jiang, Xixun Du, Bing Gong, 2020, Journal of cellular physiology.

Melatonin ameliorates myocardial ischemia/reperfusion injury in type 1 diabetic rats by preserving mitochondrial function: role of AMPK-PGC-1α-SIRT3 signaling

Bin Zhang, Shiqiang Yu, Bing Gong, 2017, Scientific Reports.

The role of ubiquitin C-terminal hydrolase L1 in neurodegenerative disorders.

E. Leznik, Bing Gong, 2007, Drug news & perspectives.

Germacrone cooperates with dexmedetomidine to alleviate high-fat diet-induced type 2 diabetes mellitus via upregulating AMPKα1 expression

Bing Gong, Haiyan Liu, Jun Wu, 2019, Experimental and therapeutic medicine.

Astragalus polysaccharide protects cardiomyocytes from 5-Fluorouracil-induced injury via decreasing ROS production

Bing Gong, Zhirong. Jiang, Dongyuan Wu, 2016 .

Targeting Ion Channels: An Important Therapeutic Implication in Gastrointestinal Dysmotility in Patients With Spinal Cord Injury

M. Korsten, Bing Gong, M. Radulovic, 2015, Journal of neurogastroenterology and motility.

Vitamin E Reversed Apoptosis of Cardiomyocytes Induced by Exposure to High Dose Formaldehyde During Mice Pregnancy.

Bing Gong, Xiaoxia Song, Zhirong. Jiang, 2017, International heart journal.

Epigenetic Mechanisms Linking Diabetes and Synaptic Impairments

Cheuk Y. Tang, G. Pasinetti, L. Ho, 2014, Diabetes.

SCFFbx2‐E3‐ligase‐mediated degradation of BACE1 attenuates Alzheimer’s disease amyloidosis and improves synaptic function

Yukiko Yoshida, Vahram Haroutunian, V. Haroutunian, 2010, Aging cell.

The neurological effects of ghrelin in brain diseases: Beyond metabolic functions

Hong Jiang, Xixun Du, Bing Gong, 2017, Neuroscience & Biobehavioral Reviews.

Role of standardized grape polyphenol preparation as a novel treatment to improve synaptic plasticity through attenuation of features of metabolic syndrome in a mouse model.

Cheuk Y. Tang, J. Simon, G. Pasinetti, 2013, Molecular nutrition & food research.

Identification of brain‐targeted bioactive dietary quercetin‐3‐O‐glucuronide as a novel intervention for Alzheimer's disease

J. Simon, G. Pasinetti, S. Percival, 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Brain-Targeted Proanthocyanidin Metabolites for Alzheimer's Disease Treatment

Sara M. Levens, A. Kania, S. Perlmutter, 2012, The Journal of Neuroscience.

Targeting multiple pathogenic mechanisms with polyphenols for the treatment of Alzheimer's disease-experimental approach and therapeutic implications

J. Schmeidler, G. Pasinetti, L. Ho, 2014, Front. Aging Neurosci..

The Ubiquitin-Proteasome System: Potential Therapeutic Targets for Alzheimer’s Disease and Spinal Cord Injury

C. Cardozo, Bing Gong, M. Figueiredo-Pereira, 2016, Front. Mol. Neurosci..

CTX-8573, an Innate-Cell Engager Targeting BCMA, is a Highly Potent Multispecific Antibody for the Treatment of Multiple Myeloma

Michael M. Schmidt, A. Oliphant, P. Bobrowicz, 2019, Blood.

Ubiquitin Hydrolase Uch-L1 Rescues β-Amyloid-Induced Decreases in Synaptic Function and Contextual Memory

O. Vitolo, O. Arancio, M. Shelanski, 2006, Cell.

Reversal of long-term dendritic spine alterations in Alzheimer disease models

O. Arancio, J. Pozueta, M. Shelanski, 2009, Proceedings of the National Academy of Sciences.

Persistent improvement in synaptic and cognitive functions in an Alzheimer mouse model after rolipram treatment.

O. Vitolo, O. Arancio, M. Shelanski, 2004, The Journal of clinical investigation.

Protection against β-amyloid induced abnormal synaptic function and cell death by Ginkgolide J

O. Vitolo, K. Nakanishi, O. Arancio, 2009, Neurobiology of Aging.

IVIG immunotherapy protects against synaptic dysfunction in Alzheimer's disease through complement anaphylatoxin C5a-mediated AMPA-CREB-C/EBP signaling pathway.

S. Barnum, G. Pasinetti, L. Ho, 2013, Molecular immunology.

Role of complement systems in IVIG mediated attenuation of cognitive deterioration in Alzheimer's disease.

S. Barnum, G. Pasinetti, Bing Gong, 2014, Current Alzheimer research.

Synaptic fatigue is more pronounced in the APP/PS1 transgenic mouse model of Alzheimer's disease.

Ottavio Arancio, O. Arancio, I. Ninan, 2005, Current Alzheimer research.

Childhood and adolescent obesity and long‐term cognitive consequences during aging

G. Pasinetti, L. Ho, Jun Wang, 2015, The Journal of comparative neurology.

Childhood and adolescent obesity and long‐term cognitive consequences during aging

G. Pasinetti, L. Ho, Jun Wang, 2015, The Journal of comparative neurology.

Cocoa extracts reduce oligomerization of amyloid-β: implications for cognitive improvement in Alzheimer's disease.

R. Blitzer, G. Pasinetti, M. Varghese, 2014, Journal of Alzheimer's disease : JAD.

G. Pasinetti, L. Ho, M. Sastre, 2013, Neurobiology of Aging.

Identification of brain‐targeted bioactive dietary quercetin‐3‐O‐glucuronide as a novel intervention for Alzheimer's disease

G. Pasinetti, S. Percival, L. Ho, 2013, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Brain-Targeted Proanthocyanidin Metabolites for Alzheimer's Disease Treatment

R. Dixon, C. Pavlides, D. Teplow, 2012, The Journal of Neuroscience.

Epigenetic Mechanisms Linking Diabetes and Synaptic Impairments

Cheuk Y. Tang, G. Pasinetti, L. Ho, 2014, Diabetes.