W. Yin

发表

Mutation of CFAP57, a protein required for the asymmetric targeting of a subset of inner dynein arms in Chlamydomonas, causes primary ciliary dyskinesia

D. Conrad, P. Sears, L. Ostrowski, 2020, PLoS genetics.

Quantitative Proteomic Analysis of Human Airway Cilia Identifies Previously Uncharacterized Proteins of High Abundance.

L. Ostrowski, M. Goshe, K. Blackburn, 2017, Journal of proteome research.

Conditional deletion of dnaic1 in a murine model of primary ciliary dyskinesia causes chronic rhinosinusitis.

L. Ostrowski, W. O'Neal, T. Rogers, 2010, American journal of respiratory cell and molecular biology.

Restoring ciliary function to differentiated Primary Ciliary Dyskinesia cells with a lentiviral vector

L. Ostrowski, J. Olsen, T. Rogers, 2014, Gene Therapy.

Lectin conjugate-directed gene transfer to airway epithelial cells.

P. Cheng, W. Yin, 1994, Biochemical and biophysical research communications.

Exome sequencing identifies mutations in CCDC114 as a cause of primary ciliary dyskinesia.

Emily H Turner, J. Shendure, D. Nickerson, 2013, American journal of human genetics.

Reduced mucociliary clearance in old mice is associated with a decrease in Muc5b mucin.

L. Ostrowski, T. Rogers, W. Yin, 2016, American journal of physiology. Lung cellular and molecular physiology.

Mice with a Deletion of Rsph1 Exhibit a Low Level of Mucociliary Clearance and Develop a PCD Phenotype.

P. Sears, L. Ostrowski, T. Rogers, 2019, American journal of respiratory cell and molecular biology.

Mice with a Deletion of Rsph1 Exhibit a Low Level of Mucociliary Clearance and Develop a Primary Ciliary Dyskinesia Phenotype

P. Sears, L. Ostrowski, T. Rogers, 2019 .

Mutations in RSPH1 cause primary ciliary dyskinesia with a unique clinical and ciliary phenotype.

J. Shendure, D. Nickerson, M. Rosenfeld, 2014, American journal of respiratory and critical care medicine.

Mutations in SPAG1 cause primary ciliary dyskinesia associated with defective outer and inner dynein arms.

Emily H Turner, J. Shendure, D. Nickerson, 2013, American journal of human genetics.

Lack of GAS2L2 Causes PCD by Impairing Cilia Orientation and Mucociliary Clearance.

Laura E. Herring, P. Sears, L. Ostrowski, 2019, American journal of human genetics.

Secretory Cells Dominate Airway CFTR Expression and Function in Human Airway Superficial Epithelia.

L. Ostrowski, B. Stripp, S. Randell, 2020, American journal of respiratory and critical care medicine.

Identification of an ectodomain within the LAR protein tyrosine phosphatase receptor that binds homophilically and activates signalling pathways promoting neurite outgrowth

Tao Yang, W. Yin, F. Longo, 2005, The European journal of neuroscience.

Cryo-electron tomography reveals ciliary defects underlying human RSPH1 primary ciliary dyskinesia

L. Ostrowski, D. Nicastro, Jianfeng Lin, 2014, Nature Communications.

Continuous mucociliary transport by primary human airway epithelial cells in vitro.

P. Sears, L. Ostrowski, W. Yin, 2015, American journal of physiology. Lung cellular and molecular physiology.

Expression of a Truncated Form of ODAD1 Associated with an Unusually Mild Primary Ciliary Dyskinesia Phenotype

P. Sears, L. Ostrowski, F. Hildebrandt, 2022, International journal of molecular sciences.

Structural insights into the cause of human RSPH4A primary ciliary dyskinesia

P. Sears, L. Ostrowski, D. Nicastro, 2021, Molecular biology of the cell.

Expression of CFTR from a ciliated cell-specific promoter is ineffective at correcting nasal potential difference in CF mice

L. Ostrowski, T. Rogers, W. Yin, 2007, Gene Therapy.