R. Finn

发表

CFAP54 is required for proper ciliary motility and assembly of the central pair apparatus in mice

G. Witman, C. McKenzie, B. Craige, 2015, Molecular biology of the cell.

Altered glutamate receptor function in the cerebellum of the Ppt1−/− mouse, a murine model of infantile neuronal ceroid lipofuscinosis

R. Finn, D. Pearce, A. Kovács, 2012, Journal of neuroscience research.

Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3 Δex7/8 -knock-in mouse model of juvenile neuronal ceroid lipofuscinosis

R. Finn, D. Pearce, A. Kovács, 2011, Neurochemistry International.

Treatment of the Ppt1–/– Mouse Model of Infantile Neuronal Ceroid Lipofuscinosis With the N-methyl-d-aspartate (NMDA) Receptor Antagonist Memantine

R. Finn, D. Pearce, A. Kovács, 2013, Journal of child neurology.

Strain-dependent brain defects in mouse models of primary ciliary dyskinesia with mutations in Pcdp1 and Spef2

R. Finn, C. Evans, C. Evans, 2014, Neuroscience.

Strain-specific differences in brain gene expression in a hydrocephalic mouse model with motile cilia dysfunction

Claudia C. Preston, R. Faustino, C. McKenzie, 2018, Scientific Reports.

Strain-specific differences in brain gene expression in a hydrocephalic mouse model with motile cilia dysfunction

Claudia C. Preston, R. Faustino, C. McKenzie, 2018, Scientific Reports.

Altered sensitivity to excitotoxic cell death and glutamate receptor expression between two commonly studied mouse strains

R. Finn, D. Pearce, A. Kovács, 2010, Journal of neuroscience research.