G. Vogl

Phosphatidylinositol 3-kinase VPS34 of Candida albicans is involved in filamentous growth, secretion of aspartic proteases, and intracellular detoxification.

S. Wölfl, A. Hartmann, A. Kitanovic, 2005, FEMS yeast research.

Immune evasion by acquisition of complement inhibitors: the mould Aspergillus binds both factor H and C4b binding protein.

A. Blom, P. Zipfel, M. Dierich, 2008, Molecular immunology.

The vesicle transport protein Vac1p is required for virulence of Candida albicans.

P. Zipfel, M. Nguyen, G. Vogl, 2006, Microbiology.

Comparison of Real-Time PCR Signal-Amplified In Situ Hybridization and Conventional PCR for Detection and Quantification of Human Papillomavirus in Archival Cervical Cancer Tissue

A. Staudach, G. Vogl, O. Dietze, 2004, Journal of Clinical Microbiology.

Angiogenic potential of ductal carcinoma in situ (DCIS) of human breast

G. Vogl, O. Dietze, C. Hauser-kronberger, 2005, Histopathology.

HER2 Is Unlikely to Be Involved in Directly Regulating Angiogenesis in Human Breast Cancer

G. Vogl, O. Dietze, C. Hauser-kronberger, 2006, Applied immunohistochemistry & molecular morphology : AIMM.

Candida albicans Hgt1p, a multifunctional evasion molecule: complement inhibitor, CR3 analogue, and human immunodeficiency virus-binding molecule.

K. Kuchler, M. Dierich, G. Vogl, 2011, The Journal of infectious diseases.