T. Biden

发表

Lysosomal acid lipase and lipophagy are constitutive negative regulators of glucose-stimulated insulin secretion from pancreatic beta cells

P. Meikle, P. Helquist, N. Mellett, 2013, Diabetologia.

Short-term inhibition of autophagy benefits pancreatic β-cells by augmenting ether lipids and peroxisomal function, and by countering depletion of n-3 polyunsaturated fatty acids after fat-feeding

P. Meikle, K. Chu, T. Biden, 2020, Molecular metabolism.

Oleate disrupts cAMP signaling, contributing to potent stimulation of pancreatic β-cell autophagy

P. Meikle, K. Chu, T. Biden, 2018, The Journal of Biological Chemistry.

Onset of Stimulated Degranulation of II and Coincides with the β Protein Kinase C Is Mediated by 1917 Chain IIA on Ser Phosphorylation of Nonmuscle Myosin Heavy Hughes

J. Stehn, W. E. Hughes, T. Kranenburg, 2006 .

Phosphorylation of Nonmuscle Myosin Heavy Chain IIA on Ser1917 Is Mediated by Protein Kinase CβII and Coincides with the Onset of Stimulated Degranulation of RBL-2H3 Mast Cells1

J. Stehn, W. E. Hughes, T. Kranenburg, 2006, The Journal of Immunology.

Synergistic cytotoxicity between tamoxifen and the plant toxin persin in human breast cancer cells is dependent on Bim expression and mediated by modulation of ceramide metabolism

R. Sutherland, A. Butt, T. Biden, 2007, Molecular Cancer Therapeutics.

G protein-coupled receptors

J. Shine, T. Biden, T. Iismaa, 1992, Current Biology.

G Protein-Coupled Receptors, Pathology and Therapeutic Strategies

J. Shine, T. Biden, T. Iismaa, 1995 .

Ceramide Generation Is Sufficient to Account for the Inhibition of the Insulin-stimulated PKB Pathway in C2C12 Skeletal Muscle Cells Pretreated with Palmitate*

C. Schmitz‐Peiffer, T. Biden, D. L. Craig, 1999, The Journal of Biological Chemistry.

PKCδ Blues for the β-Cell

C. Schmitz‐Peiffer, T. Biden, 2010, Diabetes.

Protein Kinase Cι Activity Is Necessary for Bcr-Abl-mediated Resistance to Drug-induced Apoptosis*

A. Fields, L. Jamieson, L. Carpenter, 1999, Journal of Biological Chemistry.

Inhibition of PKCα induces a PKCδ-dependent apoptotic program in salivary epithelial cells

L. Carpenter, M. Reyland, T. Biden, 2003, Cell Death and Differentiation.

Inhibition of PKCalpha induces a PKCdelta-dependent apoptotic program in salivary epithelial cells.

L. Carpenter, M. Reyland, T. Biden, 2003, Cell death and differentiation.

Increased fatty acid desaturation and enhanced expression of stearoyl coenzyme A desaturase protects pancreatic beta-cells from lipoapoptosis.

D. R. Laybutt, W. E. Hughes, T. Biden, 2005, Diabetes.

Chronic hyperglycemia, independent of plasma lipid levels, is sufficient for the loss of beta-cell differentiation and secretory function in the db/db mouse model of diabetes.

D. R. Laybutt, T. Biden, M. C. Åkerfeldt, 2005, Diabetes.

Muscle lipid accumulation and protein kinase C activation in the insulin-resistant chronically glucose-infused rat.

E. Kraegen, D. R. Laybutt, N. Ruderman, 1999, American journal of physiology. Endocrinology and metabolism.

Alteration of Endoplasmic Reticulum Lipid Rafts Contributes to Lipotoxicity in Pancreatic β-Cells*

P. Meikle, J. Weir, J. Cantley, 2013, The Journal of Biological Chemistry.

Sweet and Sour β-Cells: ROS and Hif1α Induce Warburg-Like Lactate Production During Type 2 Diabetes

J. Cantley, T. Biden, 2013, Diabetes.

Reduced endoplasmic reticulum (ER)-to-Golgi protein trafficking contributes to ER stress in lipotoxic mouse beta cells by promoting protein overload

D. R. Laybutt, T. Biden, C. Bartley, 2009, Diabetologia.

Protein Kinase C Epsilon Deletion in Adipose Tissue, but Not in Liver, Improves Glucose Tolerance.

D. James, P. Meikle, M. Febbraio, 2019, Cell metabolism.

Chronic effects of fatty acids on pancreatic beta-cell function: new insights from functional genomics.

W. E. Hughes, T. Biden, A. K. Busch, 2004, Diabetes.

Expression profiling of palmitate- and oleate-regulated genes provides novel insights into the effects of chronic lipid exposure on pancreatic beta-cell function.

T. Biden, G. Denyer, A. K. Busch, 2002, Diabetes.

Malonyl CoA as a metabolic switch and a regulator of insulin sensitivity.

E. Kraegen, D. R. Laybutt, N. Ruderman, 1998, Advances in experimental medicine and biology.

Reversal of chronic alterations of skeletal muscle protein kinase C from fat-fed rats by BRL-49653.

E. Kraegen, C. L. Browne, C. Schmitz‐Peiffer, 1997, American journal of physiology. Endocrinology and metabolism.

Ca2+ regulates the inositol tris/tetrakisphosphate pathway in intact and broken preparations of insulin-secreting RINm5F cells.

C. Wollheim, T. Biden, 1986, The Journal of biological chemistry.

Protein Kinase Cδ Activation by Interleukin-1β Stabilizes Inducible Nitric-oxide Synthase mRNA in Pancreatic β-Cells*

L. Carpenter, T. Biden, D. Cordery, 2000, The Journal of Biological Chemistry.

The role of cytosolic free calcium in the generation of inositol 1,4,5-trisphosphate and inositol 1,3,4-trisphosphate in HL-60 cells. Differential effects of chemotactic peptide receptor stimulation at distinct Ca2+ levels.

K. Krause, W. Schlegel, A. Monod, 1986, The Journal of biological chemistry.

Nutrient Stimulation Results in a Rapid Ca2+-dependent Threonine Phosphorylation of Myosin Heavy Chain in Rat Pancreatic Islets and RINm5F Cells*

T. Biden, R. Ludowyke, Justine R. Wilson, 1998, The Journal of Biological Chemistry.

Dilinoleoyl-phosphatidic acid mediates reduced IRS-1 tyrosine phosphorylation in rat skeletal muscle cells and mouse muscle

N. Turner, J. Burchfield, T. Mitchell, 2007, Diabetologia.

Acute reversal of lipid-induced muscle insulin resistance is associated with rapid alteration in PKC-theta localization.

G. Cooney, E. Kraegen, C. Schmitz‐Peiffer, 2000, American journal of physiology. Endocrinology and metabolism.

Inhibition of protein kinase C delta protects rat INS-1 cells against interleukin-1beta and streptozotocin-induced apoptosis.

L. Carpenter, T. Biden, D. Cordery, 2002, Diabetes.

Inhibition of Protein Kinase C δ Protects Rat INS-1 Cells Against Interleukin-1β and Streptozotocin-Induced Apoptosis

L. Carpenter, T. Biden, D. Cordery, 2002 .

Inhibition of glycogen synthesis by fatty acid in C2C12 muscle cells is independent of PKC-α, -ε, and -θ

C. Schmitz‐Peiffer, T. Biden, D. L. Craig, 2002 .

Protein Kinase C Function in Muscle, Liver, and β-Cells and Its Therapeutic Implications for Type 2 Diabetes

C. Schmitz‐Peiffer, T. Biden, 2008, Diabetes.

Differential regulation of adaptive and apoptotic unfolded protein response signalling by cytokine-induced nitric oxide production in mouse pancreatic beta cells

G. Cooney, D. R. Laybutt, T. Biden, 2011, Diabetologia.

Lipotoxic endoplasmic reticulum stress, β cell failure, and type 2 diabetes mellitus

K. Chu, T. Biden, E. Boslem, 2014, Trends in Endocrinology & Metabolism.

Deletion of PKCε Selectively Enhances the Amplifying Pathways of Glucose-Stimulated Insulin Secretion via Increased Lipolysis in Mouse β-Cells

J. Burchfield, M. Leitges, C. Schmitz‐Peiffer, 2009, Diabetes.

Diverse roles for protein kinase C δ and protein kinase C ε in the generation of high-fat-diet-induced glucose intolerance in mice: regulation of lipogenesis by protein kinase C δ

G. Cooney, J. Burchfield, M. Leitges, 2009, Diabetologia.

Regulation of Ca2+ transport by isolated organelles of a rat insulinoma. Studies with endoplasmic reticulum and secretory granules.

M. Prentki, C. Wollheim, T. Biden, 1984, The Journal of biological chemistry.

The diverse roles of protein kinase C in pancreatic beta-cell function.

J. Burchfield, L. Carpenter, C. Schmitz‐Peiffer, 2008, Biochemical Society transactions.

Active transport of myo-inositol in rat pancreatic islets.

C. Wollheim, T. Biden, 1986, The Biochemical journal.

Rapid increases in inositol 1,4,5-trisphosphate, inositol 1,3,4,5-tetrakisphosphate and cytosolic free Ca2+ in agonist-stimulated pancreatic acini of the rat. Effect of carbachol, caerulein and secretin.

R. Bruzzone, T. Biden, E. Trimble, 1987, The Biochemical journal.

Cross-talk between the unfolded protein response and nuclear factor-κB signalling pathways regulates cytokine-mediated beta cell death in MIN6 cells and isolated mouse islets

D. R. Laybutt, T. Biden, J. Chan, 2012, Diabetologia.

Bad News for β-Cell Apoptosis

H. Thomas, T. Biden, 2009, Diabetes.

Targeting triglyceride/fatty acid cycling in β-cells as a therapy for augmenting glucose-stimulated insulin secretion

J. Cantley, T. Biden, 2010, Islets.

Inhibition of PKCepsilon improves glucose-stimulated insulin secretion and reduces insulin clearance.

G. Cooney, D. R. Laybutt, J. Burchfield, 2007, Cell metabolism.

Cytokine-Induced β-Cell Death Is Independent of Endoplasmic Reticulum Stress Signaling

D. R. Laybutt, C. King, H. McGuire, 2008, Diabetes.

Inositol 1,4,5-trisphosphate mobilizes intracellular Ca2+ from permeabilized insulin-secreting cells.

M. Berridge, M. Prentki, C. Wollheim, 1984, The Biochemical journal.

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

Mark R. Marten, Erinna F. Lee, Dana L. Miller, 2021, Autophagy.

Second messenger function of inositol 1,4,5-trisphosphate. Early changes in inositol phosphates, cytosolic Ca2+, and insulin release in carbamylcholine-stimulated RINm5F cells.

C. Wollheim, T. Biden, 1986, The Journal of biological chemistry.

Regulation by membrane potential of phosphatidylinositol hydrolysis in pancreatic islets.

T. Biden, A. Davison, M. L. Prugue, 1993, The Journal of biological chemistry.

Protein kinase Czeta activation mediates glucagon-like peptide-1-induced pancreatic beta-cell proliferation.

M. Prentki, J. Buteau, C. Rhodes, 2001, Diabetes.

A redistribution of actin and myosin IIA accompanies Ca2+‐dependent insulin secretion

J. Wilson, T. Biden, R. Ludowyke, 2001, FEBS letters.

Disruption of beta cell acetyl-CoA carboxylase-1 in mice impairs insulin secretion and beta cell mass

D. James, P. Meikle, K. Hoehn, 2018, Diabetologia.

Signal Transduction in Insulin Secretion:

C. Wollheim, T. Biden, 1986 .

Signal transduction in insulin secretion: comparison between fuel stimuli and receptor agonists.

C. Wollheim, T. Biden, 1986, Annals of the New York Academy of Sciences.

Protein kinase Cdelta activation by interleukin-1beta stabilizes inducible nitric-oxide synthase mRNA in pancreatic beta-cells.

L. Carpenter, T. Biden, D. Cordery, 2001, The Journal of biological chemistry.

High-fat diet increases autophagic flux in pancreatic beta cells in vivo and ex vivo in mice

G. Ramm, K. Chu, T. Biden, 2015, Diabetologia.

Acetyl-CoA carboxylase-1 is a critical regulator of beta cell size and proliferation

D. James, K. Hoehn, J. Cantley, 2013 .

Secretin stimulates cyclic AMP and inositol trisphosphate production in rat pancreatic acinar tissue by two fully independent mechanisms.

R. Bruzzone, R. B. Merrifield, D. Andreu, 1987, Proceedings of the National Academy of Sciences of the United States of America.

Structural and biochemical studies of human galanin: NMR evidence for nascent helical structures in aqueous solution.

M. Morris, G. King, C. L. Browne, 1995, Biochemistry.

Ca2+-mediated generation of inositol 1,4,5-triphosphate and inositol 1,3,4,5-tetrakisphosphate in pancreatic islets. Studies with K+, glucose, and carbamylcholine.

C. Wollheim, W. Schlegel, T. Biden, 1987, The Journal of biological chemistry.

Glucose Homeostasis in Mice Is Transglutaminase 2 Independent

R. Graham, G. Cooney, J. Burchfield, 2013, PloS one.

Evidence for Selective Coupling of α1-Adrenergic Receptors to Phospholipase C-β1 in Rat Neonatal Cardiomyocytes*

E. Woodcock, T. Biden, S. Matkovich, 2001, The Journal of Biological Chemistry.

Cross-talk between muscarinic- and adenosine-receptor signalling in the regulation of cytosolic free Ca2+ and insulin secretion.

C. L. Browne, T. Biden, 1993, The Biochemical journal.

Sodium requirement for insulin release: putative role in regulation of intracellular pH.

C. Wollheim, T. Biden, D. Janjic, 1986, The American journal of physiology.

Phospholipase C-γ Mediates the Hydrolysis of Phosphatidylinositol, but Not of Phosphatidylinositol 4,5-Bisphoshate, in Carbamylcholine-stimulated Islets of Langerhans*

M. Blewitt, T. Biden, Justine R. Wilson, 2001, The Journal of Biological Chemistry.

PKCδ Is Required for Mitochondrial-dependent Apoptosis in Salivary Epithelial Cells*

L. Carpenter, M. Reyland, M. Humphries, 2001, The Journal of Biological Chemistry.

Cell Surface Receptors and the G Protein-Coupled Receptor Superfamily

J. Shine, T. Biden, T. Iismaa, 1995 .

Structural Determinants of Receptor Function

J. Shine, T. Biden, T. Iismaa, 1995 .

Rapid mobilization of Ca2+ from rat insulinoma microsomes by inositol-1,4,5-trisphosphate

M. Berridge, M. Prentki, C. Wollheim, 1984, Nature.

Endoplasmic reticulum stress contributes to beta cell apoptosis in type 2 diabetes

J. Kench, A. Biankin, D. R. Laybutt, 2007, Diabetologia.

Failure of the Adaptive Unfolded Protein Response in Islets of Obese Mice Is Linked With Abnormalities in β-Cell Gene Expression and Progression to Diabetes

D. R. Laybutt, T. Biden, J. Chan, 2013, Diabetes.

Regulation of mitochondrial metabolism in murine skeletal muscle by the medium‐chain fatty acid receptor Gpr84

N. Turner, G. Cooney, A. Cooper, 2019, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Identification of fatty acid binding protein 4 as an adipokine that regulates insulin secretion during obesity

D. James, N. Turner, D. Fazakerley, 2014, Molecular metabolism.

Protein Kinase Cζ Activation Mediates Glucagon-Like Peptide-1–Induced Pancreatic β-Cell Proliferation

M. Prentki, J. Buteau, C. Rhodes, 2001 .

Secretin induces rapid increases in inositol trisphosphate, cytosolic Ca2+ and diacylglycerol as well as cyclic AMP in rat pancreatic acini.

R. Bruzzone, R. Farese, T. Biden, 1986, The Biochemical journal.

Time-dependent effects of Prkce deletion on glucose homeostasis and hepatic lipid metabolism on dietary lipid oversupply in mice

M. Raftery, N. Turner, M. Raftery, 2011, Diabetologia.

Improved glucose homeostasis and enhanced insulin signalling in Grb14‐deficient mice

David E James, Roger J Daly, Christopher J Ormandy, 2004, The EMBO journal.

A role for protein phosphatase 2A-like activity, but not atypical protein kinase Czeta, in the inhibition of protein kinase B/Akt and glycogen synthesis by palmitate.

L. Carpenter, C. Schmitz‐Peiffer, T. Biden, 2001, Diabetes.

PKCα Is Activated But Not Required During Glucose-Induced Insulin Secretion From Rat Pancreatic Islets

L. Carpenter, P. Poronnik, T. Biden, 2004 .

Alterations in the Expression and Cellular Localization of Protein Kinase C Isozymes ε and θ Are Associated With Insulin Resistance in Skeletal Muscle of the High-Fat–Fed Rat

E. Kraegen, D. Chisholm, C. L. Browne, 1997, Diabetes.

A comprehensive lipidomic screen of pancreatic β-cells using mass spectroscopy defines novel features of glucose-stimulated turnover of neutral lipids, sphingolipids and plasmalogens

Peter J. Meikle, P. Meikle, N. Mellett, 2016, Molecular metabolism.

Treatment of type 2 diabetes with the designer cytokine IC7Fc

P. Meikle, M. Febbraio, D. Drucker, 2019, Nature.

Inhibition of PKCɛ Improves Glucose-Stimulated Insulin Secretion and Reduces Insulin Clearance

G. Cooney, D. R. Laybutt, J. Burchfield, 2007 .

Guanine nucleotides induce Ca2+-independent insulin secretion from permeabilized RINm5F cells.

C. Wollheim, T. Biden, L. Vallar, 1987, The Journal of biological chemistry.

Effects of ketone bodies on insulin release and islet-cell metabolism in the rat.

K. Taylor, T. Biden, 1983, The Biochemical journal.

Inositol 1,4,5-trisphosphate and intracellular Ca2+ homeostasis in clonal pituitary cells (GH3). Translocation of Ca2+ into mitochondria from a functionally discrete portion of the nonmitochondrial store.

C. Wollheim, W. Schlegel, T. Biden, 1986, The Journal of biological chemistry.

Molecular cloning and characterization of PKC iota, an atypical isoform of protein kinase C derived from insulin-secreting cells.

C. Schmitz‐Peiffer, T. Biden, L A Selbie, 1993, The Journal of biological chemistry.

Activated protein kinase C alpha associates with annexin VI from skeletal muscle.

C. L. Browne, C. Schmitz‐Peiffer, T. Biden, 1998, The Biochemical journal.

Roles of ceramide and sphingolipids in pancreatic β-cell function and dysfunction

P. Meikle, T. Biden, E. Boslem, 2012, Islets.

Quantitative and functional characterization of muscarinic receptor subtypes in insulin-secreting cell lines and rat pancreatic islets.

J. Wilson, L. Carpenter, T. Biden, 2000, Diabetes.

SMOC1 is a glucose-responsive hepatokine and therapeutic target for glycemic control

L. Selth, D. Samocha-Bonet, G. Steinberg, 2020, Science Translational Medicine.

Signaling Through G Protein-Coupled Receptors

J. Shine, T. Biden, T. Iismaa, 1995 .

Synergistic Interaction of Y1-Neuropeptide Y and -Adrenergic Receptors in the Regulation of Phospholipase C, Protein Kinase C, and Arachidonic Acid Production (*)

J. Shine, H. Herzog, C. L. Browne, 1995, The Journal of Biological Chemistry.

Calcium-calmodulin stimulates inositol 1,4,5-trisphosphate kinase activity from insulin-secreting RINm5F cells.

J. Cox, C. Wollheim, T. Biden, 1987, The Journal of biological chemistry.

Structural subtypes of the dopamine D2 receptor are functionally distinct: expression of the cloned D2A and D2B subtypes in a heterologous cell line.

T. Biden, L. Selbie, J. Shine, 1992, Molecular Endocrinology.

Heparan sulfate proteoglycans in beta cells provide a critical link between endoplasmic reticulum stress, oxidative stress and type 2 diabetes

S. Bornstein, D. R. Laybutt, C. Parish, 2021, PloS one.

A lipidomic screen of palmitate-treated MIN6 β-cells links sphingolipid metabolites with endoplasmic reticulum (ER) stress and impaired protein trafficking.

P. Meikle, D. R. Laybutt, T. Biden, 2011, The Biochemical journal.

Sulfonylureas as Concomitant Insulin Secretagogues and NLRP3 Inflammasome Inhibitors

K. Schroder, M. Cooper, A. Robertson, 2017, ChemMedChem.

Phospholipase Cδ1 does not mediate Ca2+ responses in neonatal rat cardiomyocytes

E. Woodcock, T. Biden, C. J. Mitchell, 2003 .

Alterations in the expression and cellular localization of protein kinase C isozymes epsilon and theta are associated with insulin resistance in skeletal muscle of the high-fat-fed rat.

E. Kraegen, D. Chisholm, C. L. Browne, 1997, Diabetes.

Deletion of protein kinase Cδ in mice modulates stability of inflammatory genes and protects against cytokine-stimulated beta cell death in vitro and in vivo

D. R. Laybutt, M. Leitges, L. Carpenter, 2011, Diabetologia.

PKCdelta blues for the beta-cell.

C. Schmitz‐Peiffer, T. Biden, 2010, Diabetes.

Protein-coupled Receptors

J. Shine, T. Biden, John Shine, 1996 .

High dietary fat and sucrose result in an extensive and time-dependent deterioration in health of multiple physiological systems in mice

Nolan J. Hoffman, B. Vissel, D. James, 2018, The Journal of Biological Chemistry.

A Selective Look at Autophagy in Pancreatic β-Cells

E. Walker, G. Pearson, T. Biden, 2021, Diabetes.

Stimulation of hepatic inositol 1,4,5-trisphosphate kinase activity by Ca2+-dependent and -independent mechanisms.

J. Altin, T. Biden, F. Bygrave, 1988, The Biochemical journal.

Role of endoplasmic reticulum stress induction by the plant toxin, persin, in overcoming resistance to the apoptotic effects of tamoxifen in human breast cancer cells

R. Nicholson, R. Sutherland, E. Musgrove, 2013, British Journal of Cancer.

G protein-coupled receptor (GPR)40-dependent potentiation of insulin secretion in mouse islets is mediated by protein kinase D1

P. MacDonald, M. Prentki, E. Olson, 2012, Diabetologia.

Akt Mediates Insulin-stimulated Phosphorylation of Ndrg2

G. Corthals, W. E. Hughes, J. Burchfield, 2004, Journal of Biological Chemistry.

Neuropeptide Y and regulation of the cardiovascular system.

E. Potter, J. Shine, H. Herzog, 1994, Journal of hypertension. Supplement : official journal of the International Society of Hypertension.

Effects of glucose and D-3-hydroxybutyrate on human pancreatic islet cell function.

I. Campbell, C. Rhodes, T. M. Szopa, 1985, Clinical science.

Phospholipase D1 Regulates Secretagogue-stimulated Insulin Release in Pancreatic β-Cells*

Ping Huang, Ping Huang, W. E. Hughes, 2004, Journal of Biological Chemistry.

An antisense of protein kinase C-zeta inhibits proliferation of human airway smooth muscle cells.

D. Cook, L. Carpenter, P. Poronnik, 2000, American journal of respiratory cell and molecular biology.

Reversal of chronic alterations of skeletal muscle protein kinase C from fat-fed rats by BRL-49653.

E. Kraegen, C. L. Browne, C. Schmitz‐Peiffer, 1997, The American journal of physiology.

Evidence for selective coupling of alpha 1-adrenergic receptors to phospholipase C-beta 1 in rat neonatal cardiomyocytes.

E. Woodcock, T. Biden, S. Matkovich, 2001, The Journal of biological chemistry.

Exposure to depolarizing concentrations of K+ inhibits hormonally-induced calcium influx in rat liver.

J. Altin, T. Biden, F. Bygrave, 1988, Biochemical and biophysical research communications.

PKC alpha is activated but not required during glucose-induced insulin secretion from rat pancreatic islets.

L. Carpenter, P. Poronnik, T. Biden, 2004, Diabetes.

Regulation of inositol 1,4,5-trisphosphate metabolism in insulin-secreting RINm5F cells.

C. Wollheim, T. Biden, L. Vallar, 1988, The Biochemical journal.

Evidence for phosphatidylinositol hydrolysis in pancreatic islets stimulated with carbamoylcholine. Kinetic analysis of inositol polyphosphate metabolism.

T. Biden, A. Davison, M. L. Prugue, 1992, The Biochemical journal.

A lipidomic screen of palmitate-treated MIN6 β-cells links sphingolipid metabolites with endoplasmic reticulum (ER) stress and impaired protein trafficking.

D. R. Laybutt, T. Biden, M. Fuller, 2011, The Biochemical journal.

Phosphorylation of Nonmuscle Myosin Heavy Chain IIA on Ser Is Mediated by Protein Kinase C II and Coincides with the Onset of Stimulated Degranulation of RBL-2H3 Mast Cells

C. Schmitz‐Peiffer, T. Biden, Z. Elgundi, 2006 .

Increases in phosphorylation of the myosin II heavy chain, but not regulatory light chains, correlate with insulin secretion in rat pancreatic islets and RINm5F cells.

J. Wilson, T. Biden, R. Ludowyke, 1999, Diabetes.

Activated protein kinase C a associates with annexin VI from skeletal muscle

C. Schmitz‐Peiffer, T. Biden, Carol L. Browne, 1998 .

Alteration of Endoplasmic Reticulum Lipid Rafts Contributes to Lipotoxicity in Pancreatic β-Cells*

P. Meikle, J. Weir, J. Cantley, 2013, The Journal of Biological Chemistry.

Protein Kinase C Epsilon Deletion in Adipose Tissue, but Not in Liver, Improves Glucose Tolerance.

D. James, P. Meikle, M. Febbraio, 2019, Cell metabolism.