É. Joly

Adipose Triglyceride Lipase Is Implicated in Fuel- and Non-fuel-stimulated Insulin Secretion*

M. Prentki, R. Zechner, N. Ruderman, 2009, The Journal of Biological Chemistry.

An improved PCR-mutagenesis strategy for two-site mutagenesis or sequence swapping between related genes.

E. Joly, É. Joly, R. Kirsch, 1998, Nucleic acids research.

Glucose represses PPARαgene expression via AMP‐activated protein kinase but not via p38 mitogen‐activated protein kinase in the pancreatic β‐cell

M. Prentki, N. Ruderman, L. Witters, 2009, Journal of diabetes.

THE BRET2/ARRESTIN ASSAY IN STABLE RECOMBINANT CELLS: A PLATFORM TO SCREEN FOR COMPOUNDS THAT INTERACT WITH G PROTEIN-COUPLED RECEPTORS (GPCRS)*

S. Angers, B. Houle, L. Bertrand, 2002, Journal of receptor and signal transduction research.

Islet beta cell failure in the 60% pancreatectomised obese hyperlipidaemic Zucker fatty rat: severe dysfunction with altered glycerolipid metabolism without steatosis or a falling beta cell mass

J. Leahy, M. Prentki, M. Latour, 2009, Diabetologia.

Oleate Promotes the Proliferation of Breast Cancer Cells via the G Protein-coupled Receptor GPR40*

M. Prentki, É. Joly, Y. Langelier, 2005, Journal of Biological Chemistry.

Hormone-sensitive lipase has a role in lipid signaling for insulin secretion but is nonessential for the incretin action of glucagon-like peptide 1.

M. Prentki, G. Mitchell, B. Corkey, 2004, Diabetes.