P. Goñi-Oliver

发表

Chronic lithium administration to FTDP‐17 tau and GSK‐3β overexpressing mice prevents tau hyperphosphorylation and neurofibrillary tangle formation, but pre‐formed neurofibrillary tangles do not revert

J. Lucas, J. Ávila, F. Hernández, 2006, Journal of neurochemistry.

Excitotoxicity induced by kainic acid provokes glycogen synthase kinase-3 truncation in the hippocampus

J. Ávila, D. Henshall, F. Hernández, 2015, Brain Research.

Calpain regulates N-terminal interaction of GSK-3β with 14-3-3ζ, p53 and PKB but not with axin

J. Ávila, F. Hernández, P. Goñi-Oliver, 2011, Neurochemistry International.

Calpain‐mediated truncation of GSK‐3 in post‐mortem brain samples

J. Ávila, F. Hernández, P. Goñi-Oliver, 2009, Journal of neuroscience research.

N-terminal Cleavage of GSK-3 by Calpain

J. Lucas, J. Ávila, F. Hernández, 2007, Journal of Biological Chemistry.

Lithium, a Potential Protective Drug in Alzheimer’s Disease

J. Lucas, J. Ávila, F. Hernández, 2008, Neurodegenerative Diseases.

Tau Kinase I Overexpression Induces Dentate Gyrus Degeneration

J. Lucas, J. Ávila, F. Hernández, 2010, Neurodegenerative Diseases.

Tau as a molecular marker of development, aging and neurodegenerative disorders.

J. Ávila, F. Hernández, P. Goñi-Oliver, 2008, Current aging science.

Hippocampal neuronal subpopulations are differentially affected in double transgenic mice overexpressing frontotemporal dementia and parkinsonism linked to chromosome 17 tau and glycogen synthase kinase-3β

J. Lucas, F. Hernández, T. Engel, 2008, Neuroscience.

PTEN suppresses axon outgrowth by down-regulating the level of detyrosinated microtubules

Carsten Schultz, Rainer Müller, Jan Schmoranzer, 2018, PloS one.

The role of GSK3 in Alzheimer disease

Jesús Avila, J. Lucas, J. Ávila, 2009, Brain Research Bulletin.