S. Ritter

发表

Leptin-sensitive neurons in the arcuate nucleus integrate activity and temperature circadian rhythms and anticipatory responses to food restriction.

H. Jansen, M. Wiater, Ai-jun Li, 2013, American journal of physiology. Regulatory, integrative and comparative physiology.

Hypothalamic and hindbrain NPY, AGRP and NE increase consummatory feeding responses

S. Ritter, K. Taylor, E. Lester, 2007, Physiology & Behavior.

Differential responsiveness of dopamine-beta-hydroxylase gene expression to glucoprivation in different catecholamine cell groups.

Ai-jun Li, S. Ritter, Qing Wang, 2006, Endocrinology.

Chemical Dissection of Brain Glucoregulatory Circuitry

T. Dinh, S. Ritter, K. Bugarith, 2005 .

The lipoprivic control of feeding is governed by fat metabolism, not by leptin or adipose depletion.

M. Wiater, S. Ritter, B. Hudson, 2010, Endocrinology.

Capsaicin‐induced neuronal degeneration: Silver impregnation of cell bodies, axons, and terminals in the central nervous system of the adult rat

T. Dinh, S. Ritter, Sue Ritter, 1988, The Journal of comparative neurology.

Neurochemical regulation of feeding in the rat: facilitation by alpha-noradrenergic, but not dopaminergic, receptor stimulants.

L. Stein, S. Ritter, D. Wise, 1975, Journal of comparative and physiological psychology.

Reduction of125I-angiotensin II binding sites in rat brain following monosodium glutamate treatment

S. Ritter, J. Harding, I. Rogulja, 1987, Brain Research.

Protein appetite is increased after central leptin-induced fat depletion.

M. Wiater, S. Ritter, B. Hudson, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

Plasma catecholamines in fasted and sucrose supplemented rats

S. Ritter, R. Mccarty, B. Weick, 1983, Physiology & Behavior.

Leptin receptor-expressing neurons in the ventromedial nucleus of the hypothalamus contribute to weight loss caused by fourth ventricle leptin infusions.

R. Harris, Ai-jun Li, S. Ritter, 2019, American journal of physiology. Endocrinology and metabolism.

Acute 2DG-induced glucoprivation or dexamethasone abolishes 2DG-induced glucoregulatory responses to subsequent glucoprivation.

N. Sanders, S. Ritter, 2001, Diabetes.

Deletion of GPR40 fatty acid receptor gene in mice blocks mercaptoacetate-induced feeding.

S. Wank, M. Wiater, Ai-jun Li, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Mercaptoacetate blocks fatty acid-induced GLP-1 secretion in male rats by directly antagonizing GPR40 fatty acid receptors.

Ai-jun Li, T. Dinh, S. Ritter, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Capsaicin abolishes lipoprivic but not glucoprivic feeding in rats.

S. Ritter, J. S. Taylor, J. Taylor, 1989, The American journal of physiology.

Vagal sensory neurons are required for lipoprivic but not glucoprivic feeding in rats.

S. Ritter, J. S. Taylor, J. Taylor, 1990, The American journal of physiology.

Feeding induced by pharmacological blockade of fatty acid metabolism is selectively attenuated by hindbrain injections of the galanin receptor antagonist, M40.

S. Ritter, F. Koegler, 1996, Obesity research.

Presence of galanin in rat vagal sensory neurons: evidence from immunohistochemistry and in situ hybridization.

N. Calingasan, S. Ritter, 1992, Journal of the autonomic nervous system.

NPAS2 deletion impairs responses to restricted feeding but not to metabolic challenges

M. Wiater, S. Ritter, Xiling Wu, 2010, Physiology & Behavior.

Localized glucoprivation of hindbrain sites elicits corticosterone and glucagon secretion.

Shayne F. Andrew, T. Dinh, S. Ritter, 2007, American journal of physiology. Regulatory, integrative and comparative physiology.

Hypoglycemia Detection

V. Routh, C. Donovan, S. Ritter, 2014 .

Activation of catecholamine neurons in the ventral medulla reduces CCK-induced hypophagia and c-Fos activation in dorsal medullary catecholamine neurons.

Ai-jun Li, S. Ritter, Qing Wang, 2018, American journal of physiology. Regulatory, integrative and comparative physiology.

Lesion of vagal afferent terminals impairs glucagon-induced suppression of food intake

S. Ritter, S. Weatherford, 1988, Physiology & Behavior.

The antiaggressive effect of lithium is abolished by area postrema lesion

K. Kelley, S. Ritter, J. McGlone, 1980, Physiology & Behavior.

The lipoprivic control of feeding is governed by fat metabolism, not by leptin or adipose depletion.

M. Wiater, S. Ritter, B. Hudson, 2010, Endocrinology.

Hypotensive hypovolemia and hypoglycemia activate different hindbrain catecholamine neurons with projections to the hypothalamus.

T. Dinh, S. Ritter, F. Flynn, 2006, American journal of physiology. Regulatory, integrative and comparative physiology.

Stimulation of insulin release and suppression of feeding by hepatic portal glucagon infusion in rats

S. Ritter, B. Weick, B. G. Weick, 1986, Physiology & Behavior.

Mercaptoacetate-induced feeding is impaired by central nucleus of the amygdala lesions

S. Ritter, B. Hutton, 1995, Physiology & Behavior.

Induction of Fos-like immunoreactivity (Fos-li) and stimulation of feeding by 2, 5-anhydro-d-mannitol (2, 5-AM) require the vagus nerve

M. Friedman, T. Dinh, S. Ritter, 1994, Brain Research.

Capsaicin attenuates hindbrain neuron responses to circulating cholecystokinin.

R. Ritter, D. Wingate, S. Ritter, 1989, The American journal of physiology.

Immunotoxic destruction of distinct catecholaminergic neuron populations disrupts the reproductive response to glucoprivation in female rats.

S. Ritter, H. I'anson, L. Sundling, 2003, Endocrinology.

Stimulation of feeding by three different glucose-sensing mechanisms requires hindbrain catecholamine neurons.

Ai-jun Li, T. Dinh, S. Ritter, 2014, American journal of physiology. Regulatory, integrative and comparative physiology.

Mercaptoacetate and fatty acids exert direct and antagonistic effects on nodose neurons via GPR40 fatty acid receptors.

Huan Zhao, Ai-jun Li, S. Ritter, 2014, American journal of physiology. Regulatory, integrative and comparative physiology.

Repeated 2-deoxy-D-glucose-induced glucoprivation attenuates Fos expression and glucoregulatory responses during subsequent glucoprivation.

N. Sanders, S. Ritter, Nicole Marie Sanders, 2000, Diabetes.

Galanin Injection into the Nucleus of the Solitary Tract Stimulates Feeding in Rats with Lesions of the Paraventricular Nucleus of the Hypothalamus

S. Ritter, F. Koegler, 1998, Physiology & Behavior.

Hindbrain catecholamine neurons control rapid switching of metabolic substrate use during glucoprivation in male rats.

M. Wiater, Ai-jun Li, T. Dinh, 2013, Endocrinology.

Immunolesion of norepinephrine and epinephrine afferents to medial hypothalamus alters basal and 2-deoxy-D-glucose-induced neuropeptide Y and agouti gene-related protein messenger ribonucleic acid expression in the arcuate nucleus.

G. Fraley, S. Ritter, 2003, Endocrinology.

Immunotoxic catecholamine lesions attenuate 2DG-induced increase of AGRP mRNA

G. Fraley, T. Dinh, S. Ritter, 2002, Peptides.

Aqueduct occlusion does not impair feeding induced by either third or fourth ventricle galanin injection.

S. Ritter, F. Koegler, 1997, Obesity research.

2-Deoxy-d-glucose and Mercaptoacetate Induce Different Patterns of Macronutrient Ingestion

S. Ritter, Joshua B Ritter, Liana Cromer, 1999, Physiology & Behavior.

Differential effects of infused nutrients on 2-deoxy-d-glucose- and 2-mercaptoacetate-induced feeding

S. Ritter, L. Singer, 1994, Physiology & Behavior.

Lateral parabrachial subnucleus lesions abolish feeding induced by mercaptoacetate but not by 2-deoxy-D-glucose.

N. Calingasan, S. Ritter, 1993, American Journal of Physiology.

Hypothalamic paraventricular nucleus lesions do not abolish glucoprivic or lipoprivic feeding

N. Calingasan, S. Ritter, 1992, Brain Research.

Nodose ganglionectomy reduces angiotensin II receptor binding in the rat brainstem

R. Speth, T. Dinh, S. Ritter, 1987, Peptides.

Magnitude of stress-induced brain norepinephrine depletion varies with age

S. Ritter, N. Pelzer, 1978, Brain Research.

Capsaicin-sensitivity and the sensory vagus: Do these exceptions prove or disprove the B-neuron rule for autonomic afferents?

R. Ritter, S. Ritter, 1990, Behavioral and Brain Sciences.

Minireview: The value of looking backward: the essential role of the hindbrain in counterregulatory responses to glucose deficit.

Ai-jun Li, T. Dinh, S. Ritter, 2011, Endocrinology.

Area postrema lesions block feeding induced by systemic injections of monosodium glutamate

S. Ritter, S. Stone, 1987, Physiology & Behavior.

S. Ritter, V. M. Reddy, S. S. Meharg, 1986, Physiology & Behavior.

Chapter 9 – GLUCOPRIVATION AND THE GLUCOPRIVIC CONTROL OF FOOD INTAKE

S. Ritter, 1986 .

Absence of lithium-induced taste aversion after area postrema lesion

K. Kelley, S. Ritter, J. McGlone, 1980, Brain Research.

Localization of hindbrain glucoreceptive sites controlling food intake and blood glucose

T. Dinh, S. Ritter, Yubei Zhang, 2000, Brain Research.

Sympathoadrenal responses to glucoprivation and lipoprivation in rats

A. Scheurink, S. Ritter, 1993, Physiology & Behavior.

Self-stimulation reward pathways: norepinephrine vs dopamine.

L. Stein, J. Belluzzi, C. Wise, 1974, Journal of psychiatric research.

Glucoprivation increases expression of neuropeptide Y mRNA in hindbrain neurons that innervate the hypothalamus

Ai-jun Li, S. Ritter, 2004, The European journal of neuroscience.

Plasma catecholamines: Exaggerated elevation is associated with stress susceptibility

R. Ritter, S. Ritter, B. Weick, 1980, Physiology & Behavior.

Absence of glucoprivic feeding after stress suggests impairment of noradrenergic neuron function

R. Ritter, S. Ritter, N. Pelzer, 1978, Brain Research.

Self-stimulation of noradrenergic cell group (A6) in locus coeruleus of rats.

L. Stein, S. Ritter, 1973, Journal of comparative and physiological psychology.

Alloxan-induced glucoprivic feeding deficits are blocked by D-glucose and amygdalin

S. Ritter, J. Murnane, 1985, Pharmacology Biochemistry and Behavior.

SELF-STIMULATION REWARD PATHWAYS: NOREPINEPHRINE VS DOPAMINE

L. Stein, J. Belluzzi, C. Wise, 1975 .

Substantia nigra self-stimulation: dependence on noradrenergic reward pathways.

J. Belluzzi, L. Stein, C. Wise, 1975, Behavioral biology.

Self-stimulation in the mesencephalic trajectory of the ventral noradrenergic bundle.

L. Stein, S. Ritter, S Ritter, 1974, Brain research.

Monitoring and Maintenance of Brain Glucose Supply

S. Ritter, 2017 .

Evidence that hindbrain astrocytes in the rat detect low glucose with a glucose transporter 2-phospholipase C-calcium release mechanism.

S. Ritter, Susan J. Burke, R. Rogers, 2020, American journal of physiology. Regulatory, integrative and comparative physiology.

Feeding Behavior: Neural and Humoral Controls

R. Ritter, C. Barnes, S. Ritter, 1987 .

Repeated Pharmacogenetic Catecholamine Neuron Activation in the Ventrolateral Medulla Attenuates Subsequent Glucoregulatory Responses

Ai-jun Li, S. Ritter, Qing Wang, 2020, Diabetes.

Selective Pharmacogenetic Activation of Catecholamine Subgroups in the Ventrolateral Medulla Elicits Key Glucoregulatory Responses

Ai-jun Li, S. Ritter, Qing Wang, 2018, Endocrinology.

Hindbrain catecholamine neurons control multiple glucoregulatory responses

Ai-jun Li, T. Dinh, S. Ritter, 2006, Physiology & Behavior.

Simultaneous Silencing of Npy and Dbh Expression in Hindbrain A1/C1 Catecholamine Cells Suppresses Glucoprivic Feeding

Ai-jun Li, T. Dinh, S. Ritter, 2009, The Journal of Neuroscience.

Immunotoxin lesion of hypothalamically projecting norepinephrine and epinephrine neurons differentially affects circadian and stressor-stimulated corticosterone secretion.

Alan G Watts, C. Pedrow, A. G. Watts, 2003, Endocrinology.

Immunotoxic destruction of distinct catecholamine subgroups produces selective impairment of glucoregulatory responses and neuronal activation

T. Dinh, S. Ritter, Sue Ritter, 2001, The Journal of comparative neurology.

Glucagon satiety: Diurnal variation after hepatic branch vagotomy or intraportal alloxan

S. Ritter, S. Weatherford, 1986, Brain Research Bulletin.

Glucagon-induced inhibition of feeding is impaired by hepatic portal alloxan injection.

S. L. Stone, S. Ritter, S. Weatherford, 1986, The American journal of physiology.

Cerebroventricular dilation in spontaneously hypertensive rats (SHRs) is not attenuated by reduction of blood pressure

T. Dinh, S. Ritter, S. Stone, 1988, Brain Research.

Progressive postnatal dilation of brain ventricles in spontaneously hypertensive rats

T. Dinh, S. Ritter, 1986, Brain Research.

Leptin-sensitive neurons in the arcuate nuclei contribute to endogenous feeding rhythms.

Bethany R. Smith, H. Jansen, Ai-jun Li, 2012, American journal of physiology. Regulatory, integrative and comparative physiology.

Hyperphagia and obesity produced by arcuate injection of NPY–saporin do not require upregulation of lateral hypothalamic orexigenic peptide genes

Ai-jun Li, T. Dinh, S. Ritter, 2008, Peptides.

Hindbrain catecholamine neurons modulate the growth hormone but not the feeding response to ghrelin.

S. Ritter, A. Emanuel, 2010, Endocrinology.

Prior optic nerve transection reduces capsaicin‐induced degeneration in rat subcortical visual structures

T. Dinh, S. Ritter, 1991, The Journal of comparative neurology.

Hindbrain catecholamine neurons mediate consummatory responses to glucoprivation

S. Ritter, Sue Ritter, Bryan Hudson, 2004, Physiology & Behavior.

Disparate effects of infused nutrients on delayed glucoprivic feeding and hypothalamic norepinephrine turnover

S. Ritter, S. Bellin, 1981, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Insulin-induced elevation of hypothalamic norepinephrine turnover persists after glucorestoration unless feeding occurs

S. Ritter, S. Bellin, 1981, Brain Research.

Circadian integration of sleep-wake and feeding requires NPY receptor-expressing neurons in the mediobasal hypothalamus.

T. Dinh, S. Ritter, S. Simasko, 2011, American journal of physiology. Regulatory, integrative and comparative physiology.

Abbreviated title: Mediobasal Hypothalamus Integrates Sleep/Wake and Feeding

T. Dinh, S. Ritter, S. Simasko, 2011 .

Functional expression of neuropeptide Y receptors in human neuroblastoma cells

Ai-jun Li, S. Ritter, 2005, Regulatory Peptides.

2-mercaptoacetate and 2-deoxy-d-glucose induce Fos-like immunoreactivity in rat brain

T. Dinh, S. Ritter, 1994, Brain Research.

Basomedial hypothalamic injections of neuropeptide Y conjugated to saporin selectively disrupt hypothalamic controls of food intake.

R. Speth, Ai-jun Li, T. Dinh, 2005, Endocrinology.

Response of catecholaminergic neurons in the mouse hindbrain to glucoprivic stimuli is astrocyte dependent

D. McDougal, S. Ritter, R. Rogers, 2018, American journal of physiology. Regulatory, integrative and comparative physiology.

Capsaicin: A Probe for Studying Specific Neuronal Populations in Brain and Retina

T. Dinh, S. Ritter, 1992 .

2-Deoxy-D-glucose but not 2-mercaptoacetate increases Fos-like immunoreactivity in adrenal medulla and sympathetic preganglionic neurons.

A. Scheurink, S. Ritter, L. Singer, 1995, Obesity research.

Glucose homeostasis and sympathoadrenal activity in mercaptoacetate-treated rats

A. Scheurink, S. Ritter, A. Steffens, 1995, Physiology & Behavior.

The role of gustatory and postingestive signals in the termination of delayed glucoprivic feeding and hypothalamic norepinephrine turnover

S. Ritter, N. Pelzer, S. Bellin, 1981, The Journal of neuroscience : the official journal of the Society for Neuroscience.

Hindbrain cytoglucopenia-induced increases in systemic blood glucose levels by 2-deoxyglucose depend on intact astrocytes and adenosine release.

S. Ritter, R. Rogers, G. Hermann, 2016, American journal of physiology. Regulatory, integrative and comparative physiology.

Capsaicin‐induced neuronal degeneration in the brain and retina of preweanling rats

T. Dinh, S. Ritter, 1990, The Journal of comparative neurology.

Subgroups of hindbrain catecholamine neurons are selectively activated by 2-deoxy-d-glucose induced metabolic challenge

I. Llewellyn-Smith, T. Dinh, S. Ritter, 1998, Brain Research.

The effects of low-, medium-, and high-fat diets on 2-deoxy-D-glucose- and mercaptoacetate-induced feeding

S. Ritter, L. K. Singer-Koegler, P. Magluyan, 1996, Physiology & Behavior.

Orexin-A enhances feeding in male rats by activating hindbrain catecholamine neurons.

Ai-jun Li, S. Ritter, Qing Wang, 2015, American journal of physiology. Regulatory, integrative and comparative physiology.

Hindbrain Catecholamine Neurons Activate Orexin Neurons During Systemic Glucoprivation in Male Rats.

R. L. Brown, Ai-jun Li, S. Ritter, 2015, Endocrinology.

2-Deoxy-D-glucose, but not mercaptoacetate, increases food intake in decerebrate rats.

S. Ritter, Rebecca A. Darling, 2009, American journal of physiology. Regulatory, integrative and comparative physiology.

Hindbrain glucoregulatory mechanisms: Critical role of catecholamine neurons in the ventrolateral medulla

Ai-jun Li, S. Ritter, Qing Wang, 2019, Physiology & Behavior.

Participation of Hindbrain AMP-Activated Protein Kinase in Glucoprivic Feeding

Ai-jun Li, S. Ritter, Qing Wang, 2011, Diabetes.

Doses of Cholecystokinin and Glucagon That Combine to Inhibit Feeding Do Not Affect Gastric Emptying in the Rat

S. Ritter, S. Weatherford, 1989 .

Protection against stress-induced brain norepinephrine depletion after repeated 2-deoxy-D-glucose administration

R. Ritter, S. Ritter, 1977, Brain Research.

Fourth ventricular alloxan injection causes feeding but not hyperglycemia in rats

S. Ritter, M. Strang, 1982, Brain Research.

Effects of Metabolic Blockade on Macronutrient Selection

S. Ritter, F. Koegler, M. F. Wiater, 1999 .

2. Hypoglycemia Detection.

V. Routh, C. Donovan, S. Ritter, 2012, Translational endocrinology & metabolism.

Response of catecholaminergic neurons in the mouse hindbrain to glucoprivic stimuli is astrocyte dependent

D. McDougal, S. Ritter, R. Rogers, 2018, American journal of physiology. Regulatory, integrative and comparative physiology.

Low-dose near-celiac arterial cholecystokinin suppresses food intake in rats.

R. Ritter, N. Calingasan, S. Ritter, 1992, The American journal of physiology.

Intraventricular alloxan impairs feeding to both central and systemic glucoprivation

S. Ritter, J. Murnane, 1985, Physiology & Behavior.

Hyperphagia and obesity produced by arcuate injection of NPY–saporin do not require upregulation of lateral hypothalamic orexigenic peptide genes

Ai-jun Li, T. Dinh, S. Ritter, 2008, Peptides.

Participation of noradrenergic cell groups A 1 and A2 in glucoregulation

T. Dinh, S. Ritter, J. Haynes, 2006 .

Feeding Behavior: Neural and Humoral Controls

R. Ritter, C. Barnes, S. Ritter, 1987 .

Glucoprivic feeding is impaired by lateral or fourth ventricular alloxan injection.

S. Ritter, E. Ladenheim, J. Murnane, 1982, The American journal of physiology.