S. Rutschmann

发表

ENU-induced phenovariance in mice: inferences from 587 mutations

Daniel L. Popkin, Zhengfan Jiang, B. Beutler, 2012, BMC Research Notes.

Efficient T cell activation via a Toll-Interleukin 1 Receptor-independent pathway.

B. Beutler, A. Theofilopoulos, K. Hoebe, 2006, Immunity.

Rab27a is a key component of the secretory machinery of azurophilic granules in granulocytes.

B. Beutler, D. Munafó, S. Rutschmann, 2007, The Biochemical journal.

Transport of LDL-derived cholesterol from the NPC 1 compartment to the ER involves the trans-Golgi network and the SNARE protein complex

Blasius, S. Pettersson, E. Moresco, 2008 .

C1q restrains autoimmunity and viral infection by regulating CD8+ T cell metabolism

N. Thielens, P. Ashton‐Rickardt, J. Strid, 2018, Science.

A Toll-Like Receptor 2-Responsive Lipid Effector Pathway Protects Mammals against Skin Infections with Gram-Positive Bacteria

V. Nizet, Zhengfan Jiang, B. Beutler, 2005, Infection and Immunity.

Dissecting immunity by germline mutagenesis

S. Rutschmann, Onjee Choi, 2012, Immunology.

Enhanced sensitivity to DSS colitis caused by a hypomorphic Mbtps1 mutation disrupting the ATF6-driven unfolded protein response

D. Brenner, B. Beutler, Xiaohong Li, 2009, Proceedings of the National Academy of Sciences.

Hypopigmentation and Maternal-Zygotic Embryonic Lethality Caused by a Hypomorphic Mbtps1 Mutation in Mice

B. Beutler, E. Moresco, Xiaohong Li, 2012, G3: Genes | Genomes | Genetics.

Dual Activation of the Drosophila Toll Pathway by Two Pattern Recognition Receptors

M. Belvin, J. Hoffmann, D. Ferrandon, 2003, Science.

N‐Ethyl‐N‐nitrosourea mutagenesis in the mouse provides strong genetic and in vivo evidence for the role of the Caspase Recruitment Domain (CARD) of CARD‐MAGUK1 in T regulatory cell development

P. Ashton‐Rickardt, E. Salisbury, S. Rutschmann, 2014, Immunology.

Slc15a4, AP-3, and Hermansky-Pudlak syndrome proteins are required for Toll-like receptor signaling in plasmacytoid dendritic cells

B. Beutler, Xiaohong Li, P. Georgel, 2010, Proceedings of the National Academy of Sciences.

A Deficiency in Nucleoside Salvage Impairs Murine Lymphocyte Development, Homeostasis, and Survival

E. Lam, D. Heathcote, P. Ashton‐Rickardt, 2012, The Journal of Immunology.

Genetic analysis of innate resistance to mouse cytomegalovirus (MCMV).

Zhengfan Jiang, B. Beutler, P. Georgel, 2005, Briefings in functional genomics & proteomics.

Role of Drosophila IKKγ in a Toll-independent antibacterial immune response

R. Zhou, J. Hoffmann, D. Ferrandon, 2000, Nature Immunology.

The Rel protein DIF mediates the antifungal but not the antibacterial host defense in Drosophila.

J. Hoffmann, C. Hétru, J. Reichhart, 2000, Immunity.

CD36 is a sensor of diacylglycerides

Thomas Hartung, T. Hartung, B. Beutler, 2005, Nature.

Human IL-13 production is negatively influenced by CD3 engagement. Enhancement of IL-13 production by cyclosporin A.

L. Aarden, J. Wijdenes, S. Rutschmann, 1996, Journal of immunology.

Genetic analysis of host resistance: Toll-like receptor signaling and immunity at large.

Zhengfan Jiang, B. Beutler, K. Hoebe, 2006, Annual review of immunology.

Cutting Edge: The Toll Pathway Is Required for Resistance to Gram-Positive Bacterial Infections in Drosophila1

D. Ferrandon, S. Rutschmann, A. Kilinç, 2002, The Journal of Immunology.

Dissecting innate immunity by germline mutagenesis

K. Hoebe, S. Rutschmann, 2008, Immunology.

Microfluorometer assay to measure the expression of beta-galactosidase and green fluorescent protein reporter genes in single Drosophila flies.

J. Hoffmann, D. Ferrandon, S. Rutschmann, 2001, BioTechniques.

Inflammation and autoimmunity caused by a SHP1 mutation depend on IL-1, MyD88, and a microbial trigger

B. Beutler, A. Theofilopoulos, S. Pettersson, 2008, Proceedings of the National Academy of Sciences.

Jinx, an MCMV susceptibility phenotype caused by disruption of Unc13d: a mouse model of type 3 familial hemophagocytic lymphohistiocytosis

B. Beutler, K. Hoebe, E. Janssen, 2008, The Journal of experimental medicine.

Jinx, an MCMV susceptibility phenotype caused by disruption of Unc13d: a mouse model of type 3 familial hemophagocytic lymphohistiocytosis

B. Beutler, K. Hoebe, E. Janssen, 2007, The Journal of Experimental Medicine.

Role of Drosophila IKK gamma in a toll-independent antibacterial immune response.

R. Zhou, J. Hoffmann, D. Ferrandon, 2000, Nature immunology.

Disruption of copper homeostasis due to a mutation of Atp7a delays the onset of prion disease

E. Masliah, B. Beutler, Xin Du, 2012, Proceedings of the National Academy of Sciences.

The protein LEM promotes CD8+ T cell immunity through effects on mitochondrial respiration

A. Oxenius, Isobel S Okoye, R. Abdi, 2015, Science.

Details of Toll-like receptor:adapter interaction revealed by germ-line mutagenesis.

I. Wilson, A. Olson, Zhengfan Jiang, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Analysis of the MCMV resistome by ENU mutagenesis

B. Beutler, K. Hoebe, P. Georgel, 2006, Mammalian Genome.

PanR1, a Dominant Negative Missense Allele of the Gene Encoding TNF-α (Tnf), Does Not Impair Lymphoid Development1

B. Dahiyat, B. Beutler, J. Zalevsky, 2006, The Journal of Immunology.

Effects of a new protein kinase C inhibitor CGP 41251 on T cell functions: inhibition of activation, growth, and target cell killing.

P. Erb, S. Rutschmann, S. Alkan, 1993, Cellular Immunology.

Corrections

Blasius, S. Pettersson, E. Moresco, 2012, British Journal of Ophthalmology.

Hypomorphic mutation in the site-1 protease Mbtps1 endows resistance to persistent viral infection in a cell-specific manner.

Daniel L. Popkin, B. Beutler, S. Kunz, 2011, Cell host & microbe.

Inflammation and autoimmunity caused by a SHP1 mutation depend on IL-1, MyD88, and a microbial trigger (Proceedings of the National Academy of Sciences of the United States of America (2008) 105, 39, (15028-15033) DOI: 10.1073/pnas.0806619105)

B. Beutler, A. Theofilopoulos, S. Pettersson, 2008 .

P. Ashton‐Rickardt, E. Salisbury, S. Rutschmann, 2014, Immunology.