M. Pozza

发表

Role of NMDA Receptor Subtypes in Governing the Direction of Hippocampal Synaptic Plasticity

M. Sheng, Y. T. Wang, Y. Auberson, 2004, Science.

Phaclofen: a GABAB blocker reduces long-duration inhibition in the neocortex.

M. Pozza, H. Olpe, G. Karlsson, 1988, European journal of pharmacology.

Selected amino acids, dipeptides and arylalkylamine derivatives do not act as allosteric modulators at GABAB receptors.

S. Urwyler, M. Pozza, J. Mosbacher, 2004, European journal of pharmacology.

The potency of the novel tachykinin receptor antagonist CGP49823 at rat and gerbil motoneurones in vitro.

R. H. Evans, M. Pozza, S. Ofner, 1998, European journal of pharmacology.

[3H]CGP 61594, the first photoaffinity ligand for the glycine site of NMDA receptors

M. Honer, H. Mohler, M. Pozza, 1999, Neuropharmacology.

GABAB autoreceptors regulate the induction of LTP

G. Collingridge, C. Davies, M. Pozza, 1991, Nature.

CGP 37849 and CGP 39551: novel and potent competitive N‐methyl‐d‐aspartate receptor antagonists with oral activity

G. Fagg, M. Pozza, M. Schmutz, 1990, British journal of pharmacology.

N,N′-Dicyclopentyl-2-methylsulfanyl-5-nitro-pyrimidine-4,6-diamine (GS39783) and Structurally Related Compounds: Novel Allosteric Enhancers of γ-Aminobutyric AcidB Receptor Function

S. Urwyler, W. Froestl, M. Pozza, 2003, Journal of Pharmacology and Experimental Therapeutics.

The Influence Of Rufinamide On Sodium Currents And Action Potential Firing In Rodent Neurons: 3.062

M. Pozza, M. Schmutz, M. McLean, 2005 .

CGP 35348: a centrally active blocker of GABAB receptors.

G. Fagg, R. G. Hall, W. Froestl, 1990, European journal of pharmacology.

The tritocerebral commissure ‘dwarf’ (TCD): a major GABA-immunoreactive descending interneuron in the locust

J. Bacon, M. Pozza, B. H. Peters, 1988, Journal of Comparative Physiology A.

Epilepsy, Hyperalgesia, Impaired Memory, and Loss of Pre- and Postsynaptic GABAB Responses in Mice Lacking GABAB(1)

C. Lüscher, P. Kelly, W. Spooren, 2001, Neuron.

Valproate enhances GABA-A mediated inhibition of locus coeruleus neurones in vitro

M. Pozza, M. Schmutz, H. Olpe, 1988, Naunyn-Schmiedeberg's Archives of Pharmacology.

Effects of clozapine, haloperidol and iloperidone on neurotransmission and synaptic plasticity in prefrontal cortex and their accumulation in brain tissue: an in vitro study

M. Pozza, A. Enz, H. Olpe, 2003, Neuroscience.

[3H]CGP 39653: a new N-methyl-D-aspartate antagonist radioligand with low nanomolar affinity in rat brain.

G. Fagg, M. Williams, M. Pozza, 1991, European journal of pharmacology.

Reevaluation of ACEA 1021 as an antagonist at the strychnine-insensitive glycine site of the N-methyl-d-aspartate receptor

K. Lingenhöhl, M. Pozza, 1998, Neuropharmacology.

Molecular distinction of three N-methyl-D-aspartate-receptor subtypes in situ and developmental receptor maturation demonstrated with the photoaffinity ligand 125I-labeled CGP 55802A.

D. Laurie, P. Seeburg, T. Marti, 1993, Proceedings of the National Academy of Sciences of the United States of America.

Phosphinic Acid Analogues of GABA. Part 2. Selective, Orally Active GABAB Antagonists.

W. Froestl, H. Riezen, M. Pozza, 1995 .

GABAA and GABAB receptors in locus coeruleus: effects of blockers.

M. Pozza, H. Olpe, M. Steinmann, 1988, European Journal of Pharmacology.

The neurotensin analog xenopsin excites nigral dopamine neurons.

M. Pozza, S. Bischoff, H. Olpe, 1988, European journal of pharmacology.

diamine (GS39783) and Structurally Related Compounds: Novel Allosteric Enhancers of -Aminobutyric Acid B Receptor Function

S. Urwyler, W. Froestl, M. Pozza, 2003 .

Electrophysiological characterization of CGP6873OA a N-methyl-D-aspartate antagonist acting at the strychnine-insensitive glycine site

K. Lingenhöhl, K. Zimmermann, M. Pozza, 2000, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

Electrophysiological characterization of a novel potent and orally active NMDA receptor antagonist: CGP 37849 and its ethylester CGP 39551.

G. Fagg, M. Pozza, H. Olpe, 1990, European journal of pharmacology.

Glutamate-induced activation of rat locus coeruleus increases CA1 pyramidal cell excitability

M. Pozza, P. Waldmeier, H. Olpe, 1985, Neuroscience Letters.

The actions of orally active GABAB receptor antagonists on GABAergic transmission in vivo and in vitro.

W. Froestl, M. Pozza, H. Bittiger, 1993, European journal of pharmacology.

Excitatory Acoustic Responses in the Inferior Colliculus of the Rat are Increased by GABAB Receptor Blockade

K. Lingenhöhl, M. Pozza, M. Vaughn, 1996, Neuropharmacology.

Electrophysiological studies on the hippocampus and prefrontal cortex assessing the effects of amyloidosis in amyloid precursor protein 23 transgenic mice

L. Danober, K. Wiederhold, M. Pozza, 2003, Neuroscience.

Comparison of antagonist potencies at pre‐ and post‐synaptic GABAB receptors at inhibitory synapses in the CA1 region of the rat hippocampus

C. Davies, N. A. Manuel, W. Froestl, 1999, British journal of pharmacology.

CGP 55845A: A potent antagonist of GABAb receptors in the CA1 region of rat hippocampus

G. Collingridge, C. Davies, M. Pozza, 1993, Neuropharmacology.

Comparative investigations on the actions of ACTH1–24, somatostatin, neurotensin, substance P and vasopressin on locus coeruleus neuronal activity in vitro

H. Haas, M. Pozza, H. Olpe, 1987, Naunyn-Schmiedeberg's Archives of Pharmacology.

Cholinomimetics induce θ rhythm and reduce hippocampal pyramidal cell excitability

C. Mondadori, A. Glatt, M. Pozza, 1987 .

GABAB receptor antagonists:New tools and potential new drugs

W. Froestl, M. Pozza, P. Waldmeier, 1996 .

ModuEation of the NMDA receptor by D-serine in the cortex and the spinal cord, in vitro

G. Fagg, M. Pozza, U. Wicki, 1990 .