A. Ladd

A. Ladd, Andrea N. Ladd, L. Sheffield, 1997, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

CELF-mediated alternative splicing is required for cardiac function during early, but not later, postnatal life.

M. Penn, A. Ladd, K. Brimacombe, 2009, Journal of molecular and cellular cardiology.

Conserved developmental alternative splicing of muscleblind-like (MBNL) transcripts regulates MBNL localization and activity

A. Ladd, Andrea N. Ladd, F. Terenzi, 2010, RNA biology.

The neurofibromatosis type I pre-mRNA is a novel target of CELF protein-mediated splicing regulation

H. Lou, Hui-Ming Zhu, M. N. Hinman, 2009, Nucleic acids research.

Cardiac Tissue-Specific Repression of CELF Activity Disrupts Alternative Splicing and Causes Cardiomyopathy

T. Cooper, G. Taffet, C. Hartley, 2005, Molecular and Cellular Biology.

Dynamic balance between activation and repression regulates pre‐mRNA alternative splicing during heart development

T. Cooper, M. Swanson, A. Ladd, 2005, Developmental dynamics : an official publication of the American Association of Anatomists.

CUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease

A. Ladd, 2013, Molecular and Cellular Neuroscience.

Identification of Targets of CUG-BP, Elav-Like Family Member 1 (CELF1) Regulation in Embryonic Heart Muscle

A. Ladd, Y. Blech-Hermoni, Ryan J. Coram, 2016, PloS one.

The importance of CELF control: molecular and biological roles of the CUG‐BP, Elav‐like family of RNA‐binding proteins

A. Ladd, Andrea N. Ladd, Twishasri Dasgupta, 2012, Wiley interdisciplinary reviews. RNA.

Alternative splicing of the neurofibromatosis type 1 pre-mRNA is regulated by the muscleblind-like proteins and the CUG-BP and ELAV-like factors

H. Lou, A. Ladd, Andrea N. Ladd, 2012, BMC Molecular Biology.

The Ethylene Response Mediator ETR1 from Arabidopsis Forms a Disulfide-linked Dimer (*)

A. Bleecker, A. Ladd, Andrea N. Ladd, 1995, The Journal of Biological Chemistry.

Muscleblind‐like 1 is a negative regulator of TGF‐β‐dependent epithelial–mesenchymal transition of atrioventricular canal endocardial cells

A. Ladd, K. Brimacombe, Kyle R Brimacombe, 2009, Developmental dynamics : an official publication of the American Association of Anatomists.

Probable microvertebrates, vertebrate-like fossils, and weird things from the Wisconsin Ordovician

A. Ladd, D. L. Clark, J. Sorenson, 1999, Journal of Paleontology.

Muscleblind-like 1 is required for normal heart valve development in vivo

Michael W. Jenkins, Maurice S. Swanson, M. Swanson, 2015, BMC Developmental Biology.

Known turnover and translation regulatory RNA-binding proteins interact with the 3’ UTR of SECIS-binding protein 2

Michael E. Budiman, A. Ladd, C. Gerber, 2009, RNA biology.

Repression of nuclear CELF activity can rescue CELF-regulated alternative splicing defects in skeletal muscle models of myotonic dystrophy

A. Ladd, D. Berger, Andrea N. Ladd, 2012, PLoS currents.

Differential susceptibility of muscles to myotonia and force impairment in a mouse model of myotonic dystrophy

E. van Lunteren, A. Ladd, D. Berger, 2011, Muscle & nerve.

Gene Expression Analyses Implicate an Alternative Splicing Program in Regulating Contractile Gene Expression and Serum Response Factor Activity in Mice

A. Ladd, Samantha J. Stillwagon, Andrea N. Ladd, 2013, PloS one.

Diversity and conservation of CELF1 and CELF2 RNA and protein expression patterns during embryonic development

A. Ladd, Y. Blech-Hermoni, Samantha J. Stillwagon, 2013, Developmental dynamics : an official publication of the American Association of Anatomists.