G. Rebeck

发表

Apolipoprotein E in sporadic Alzheimer's disease: Allelic variation and receptor interactions

B. Hyman, D. Strickland, G. Rebeck, 1993, Neuron.

Modulation of β-Amyloid Precursor Protein Processing by the Low Density Lipoprotein Receptor-related Protein (LRP)

B. Hyman, R. Tanzi, I. Mikhailenko, 2000, The Journal of Biological Chemistry.

Modulation of beta-amyloid precursor protein processing by the low density lipoprotein receptor-related protein (LRP). Evidence that LRP contributes to the pathogenesis of Alzheimer's disease.

B. Hyman, R. Tanzi, I. Mikhailenko, 2000, The Journal of biological chemistry.

A gene−brain−cognition pathway for the effect of an Alzheimer׳s risk gene on working memory in young adults

Adam E. Green, G. Rebeck, Benson W. Stevens, 2014, Neuropsychologia.

No cross-sectional influence of APOE ɛ4 dose on clinical tests in Alzheimer's disease

G. Hancock, P. Aisen, G. Rebeck, 2009, Neurobiology of Aging.

A combined effect of two Alzheimer's risk genes on medial temporal activity during executive attention in young adults

Adam E. Green, J. Gray, C. DeYoung, 2014, Neuropsychologia.

Two Alzheimer’s disease risk genes increase entorhinal cortex volume in young adults

Adam E. Green, G. Rebeck, Benson W. Stevens, 2014, Front. Hum. Neurosci..

Progression of Cerebral Amyloid Angiopathy: Accumulation of Amyloid-ß40 in Affected Vessels

B T Hyman, B. Hyman, S. Greenberg, 1998, Journal of neuropathology and experimental neurology.

Lack of association of trinucleotide repeat polymorphisms in the very‐low‐density lipoprotein receptor gene with Alzhelner's disease

B. Hyman, S. Greenberg, G. Rebeck, 1996 .

Association of membrane-bound amyloid precursor protein APP with the apolipoprotein E receptor LRP.

B. Hyman, R. Tanzi, D. Strickland, 2001, Brain research. Molecular brain research.

ApoE isoforms affect neuronal N-methyl-d-aspartate calcium responses and toxicity via receptor-mediated processes

B. Hyman, G. Rebeck, K. Crutcher, 2003, Neuroscience.

Low‐density lipoprotein receptor‐related protein levels and endocytic function are reduced by overexpression of the FE65 adaptor protein, FE65L1

B. Hyman, R. Tanzi, G. Rebeck, 2002, Journal of neurochemistry.

Evidence against association of the FE65 gene (APBB1) intron 13 polymorphism in Alzheimer's patients

D. Blacker, B. Hyman, R. Tanzi, 2000, Neuroscience Letters.

Selective 5-HT1D alpha serotonin receptor gene expression in trigeminal ganglia: implications for antimigraine drug development.

B. Hyman, K. I. Maynard, G. Rebeck, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Characterization of the major DNA repair methyltransferase activity in unadapted Escherichia coli and identification of a similar activity in Salmonella typhimurium

L. Samson, G. Rebeck, C. M. Smith, 1989, Journal of bacteriology.

A mouse model of chemotherapy-related cognitive impairments integrating the risk factors of aging and APOE4 genotype

Olga C. Rodriguez, G. Rebeck, Yichien Lee, 2020, Behavioural Brain Research.

Role of the low-density lipoprotein receptor-related protein in beta-amyloid metabolism and Alzheimer disease.

B. Hyman, D. Strickland, G. Rebeck, 2000, Archives of neurology.

Elevation of LDL Receptor‐Related Protein Levels via Ligand Interactions in Alzheimer Disease and In Vitro

B. Hyman, D. Strickland, G. Rebeck, 2001, Journal of neuropathology and experimental neurology.

Expression and alternate splicing of apolipoprotein E receptor 2 in brain

B. Hyman, A. Clatworthy, W. Schneider, 1999, Neuroscience.

Demonstration by Fluorescence Resonance Energy Transfer of Two Sites of Interaction between the Low-Density Lipoprotein Receptor-Related Protein and the Amyloid Precursor Protein: Role of the Intracellular Adapter Protein Fe65

B. Hyman, D. Strickland, G. Rebeck, 2001, The Journal of Neuroscience.

Spatial inhibition of return is impaired in mild cognitive impairment and mild Alzheimer’s disease

Raymond Scott Turner, Xiong Jiang, G. Rebeck, 2020, bioRxiv.

Cutting in a lipid bilayer? Keystone Symposium on Molecular Mechanisms of Alzheimer Disease, Taos, New Mexico, USA, 3-8 March 1999.

G. Rebeck, 1999, Trends in genetics : TIG.

A Novel Approach for Studying Endogenous Aβ Processing Using Cultured Primary Neurons Isolated from APP Transgenic Mice

G. Rebeck, G. Rebeck, Z. Qiu, 2001, Experimental Neurology.

Notch1 and Amyloid Precursor Protein Are Competitive Substrates for Presenilin1-dependent γ-Secretase Cleavage*

B. Hyman, O. Berezovska, G. Rebeck, 2001, The Journal of Biological Chemistry.

Modulation of Aβ Deposition in APP Transgenic Mice by an Apolipoprotein E Null Background

B. Hyman, S. Paul, M. Irizarry, 2000, Annals of the New York Academy of Sciences.

No evidence for genetic association or linkage of the cathepsin D (CTSD) exon 2 polymorphism and Alzheimer disease

M. McInnis, D. Blacker, B. Hyman, 2001, Annals of neurology.

The endocytic receptor protein LRP also mediates neuronal calcium signaling via N-methyl-D-aspartate receptors.

B. Hyman, B. Bacskai, D. Strickland, 2000, Proceedings of the National Academy of Sciences of the United States of America.

Apolipoprotein E epsilon 4 allele is not associated with earlier age at onset in amyotrophic lateral sclerosis.

B. Hyman, G. Rebeck, D. McKenna-Yasek, 1995, Annals of neurology.

Pathogenic effects of D23N Iowa mutant amyloid beta -protein.

S. Greenberg, W. V. Van Nostrand, H. Cho, 2001, The Journal of biological chemistry.

Modulators of expression and function of LRP in Alzheimer's disease

B. Hyman, R. Tanzi, D. Strickland, 1997 .

Apolipoprotein E and Apolipoprotein E Receptors in the Central Nervous System: Relationship with Alzheimer’s Disease

B. Hyman, M. Irizarry, D. Strickland, 1996 .

Apolipoprotein E modulates γ‐secretase cleavage of the amyloid precursor protein

B. Hyman, A. Lleó, M. Irizarry, 2004, Journal of neurochemistry.

Confirmation of the genetic association of interleukin-1A with early onset sporadic Alzheimer's disease

G. Rebeck, 2000, Neuroscience Letters.

Hemorrhagic stroke associated with the Iowa amyloid precursor protein mutation

T. Grabowski, J. Baron, S. Greenberg, 2003, Neurology.

Lack of association of two lipoprotein lipase polymorphisms with Alzheimer's disease

G. Rebeck, H. Cho, Matthew D Martin-Rehrmann, 2002, Neuroscience Letters.

alpha 2-Macroglobulin exposure reduces calcium responses to N-methyl-D-aspartate via low density lipoprotein receptor-related protein in cultured hippocampal neurons.

B. Hyman, D. Strickland, G. Rebeck, 2002, The Journal of biological chemistry.

Lack of independent associations of apolipoprotein E promoter and intron 1 polymorphisms with Alzheimer's disease

B. Hyman, S. Greenberg, J. Locascio, 1999, Neuroscience Letters.

Lack of allelic imbalance in APOE epsilon3/4 brain mRNA expression in Alzheimer's disease.

B. Hyman, W. Growdon, G. Rebeck, 1999, Neuroscience letters.

APOE2 is associated with longevity independent of Alzheimer’s disease

Xianlin Han, M. Heckman, G. Bu, 2020, eLife.

A second DNA methyltransferase repair enzyme in Escherichia coli.

L. Samson, G. Rebeck, S. Coons, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Alpha 2-macroglobulin receptor/low density lipoprotein receptor-related protein. Relationship to apolipoprotein E and role in Alzheimer disease senile plaques.

B. Hyman, D. Strickland, G. Rebeck, 1994, Annals of the New York Academy of Sciences.

α2‐Macroglobulin Receptor/Low Density Lipoprotein Receptor—Related Protein

B. Hyman, D. Strickland, G. Rebeck, 1994 .

Plasma beta-amyloid peptide, transforming growth factor-beta 1, and risk for cerebral amyloid angiopathy.

S. Younkin, L. Younkin, S. Greenberg, 2000, Annals of the New York Academy of Sciences.

Expression of the Very Low‐Density Lipoprotein Receptor (VLDL‐r), an Apolipoprotein‐E Receptor, in the Central Nervous System and in Alzheimer's Disease

B. Hyman, D. Strickland, R. Christie, 1996, Journal of neuropathology and experimental neurology.

The effects of AbetaPP mutations and APOE polymorphisms on cerebral amyloid angiopathy.

T. Grabowski, S. Greenberg, H. Cho, 2001, Amyloid: Journal of Protein Folding Disorders.

Genetics ignite focus on microglial inflammation in Alzheimer’s disease

Manasi Malik, D. Fardo, S. Estus, 2015, Molecular Neurodegeneration.

Frequency of the apolipoprotein E epsilon 2 allele is diminished in sporadic Alzheimer disease.

B. Hyman, G. Rebeck, H. West, 1994, Neuroscience letters.

Quantitative analysis ofsenile plaques inAlzheimer disease: Observation oflog-normal sizedistribution andmolecular epidemiology ofdifferences associated withapolipoprotein E genotype andtrisomy 21(Downsyndrome)

B. Hyman, G. Rebeck, H. West, 1995 .

APOE4-specific Changes in Aβ Accumulation in a New Transgenic Mouse Model of Alzheimer Disease*

E. Weeber, S. Estus, G. Bu, 2012, The Journal of Biological Chemistry.

Interaction of Reelin with Amyloid Precursor Protein Promotes Neurite Outgrowth

Guojun Bu, B. Hyman, Jiyeon Lee, 2009, The Journal of Neuroscience.

Multiple, diverse senile plaque–associated proteins are ligands of an apolipoprotein e receptor, the α2‐macroglobulin receptor/low‐density‐lipoprotein receptor—related protein

B. Hyman, D. Strickland, G. Rebeck, 1995, Annals of neurology.

Apolipoprotein E and Alzheimer's disease: the protective effects of ApoE2 and E3.

M. Kindy, M. LaDu, G. Rebeck, 2002, Journal of Alzheimer's disease : JAD.

Increased spontaneous mutation and alkylation sensitivity of Escherichia coli strains lacking the ogt O6-methylguanine DNA repair methyltransferase

L. Samson, G. Rebeck, 1991, Journal of bacteriology.

Potential Biological Mechanisms of ApoE in Alzheimer’s Disease

G. Rebeck, 1997 .

APOE genotype and gender effects on Alzheimer disease in 100 adults with Down syndrome

G. Rebeck, R. Nixon, A. Anderson, 1999, Neurology.

Alzheimer's Disease Genetic Risk Factor APOE-ε4 Also Affects Normal Brain Function.

G. Rebeck, Nicolette M. Heinsinger, Amanda Di Battista, 2016, Current Alzheimer research.

Quantitative analysis of senile plaques in Alzheimer disease: observation of log-normal size distribution and molecular epidemiology of differences associated with apolipoprotein E genotype and trisomy 21 (Down syndrome).

R. Mantegna, H. Stanley, B. Hyman, 1995, Proceedings of the National Academy of Sciences of the United States of America.

Neuropathological changes in Down's syndrome hippocampal formation. Effect of age and apolipoprotein E genotype.

B. Hyman, D. Mann, G. Rebeck, 1995, Archives of neurology.

Structure and Functions of Human Cerebrospinal Fluid Lipoproteins from Individuals of Different APOE Genotypes

J. Growdon, B. Hyman, O. Berezovska, 1998, Experimental Neurology.

Clinical manifestations of cerebral amyloid angiopathy–related inflammation

M. Frosch, S. Greenberg, G. Rebeck, 2004, Annals of neurology.

Novel amyloid precursor protein mutation in an Iowa family with dementia and severe cerebral amyloid angiopathy

T. Grabowski, S. Greenberg, J. Vonsattel, 2001, Annals of neurology.

Neuropathology and apolipoprotein E profile of aged chimpanzees: implications for Alzheimer disease.

B T Hyman, J. Tigges, B. Hyman, 1994, Proceedings of the National Academy of Sciences of the United States of America.

Kinetics of Cerebral Amyloid Angiopathy Progression in a Transgenic Mouse Model of Alzheimer Disease

Rebecca A Betensky, Brian J Bacskai, Matthew P Frosch, 2006, The Journal of Neuroscience.

The Expression of Bacterial DNA Repair Genes in Eukaryotic Cells and Vice Versa

L. Samson, G. Rebeck, B. Derfler, 1991 .

Neuroanatomical connections and specific regional vulnerability in Alzheimer's disease

B. Hyman, G. Rebeck, 1993, Neurobiology of Aging.

B. Hyman, R. Tanzi, A. Bush, 1995, Cell.

Pathogenic Effects of D23N Iowa Mutant Amyloid β-Protein*

S. Greenberg, W. V. Van Nostrand, J. Melchor, 2001, The Journal of Biological Chemistry.

Protective Effects of APOE ε2 Genotype on Cognition in Older Breast Cancer Survivors: The Thinking and Living With Cancer Study

H. Cohen, A. Saykin, B. McDonald, 2021, JNCI cancer spectrum.

Epidemiological, Clinical, and Neuropathological Study of Apolipoprotein E Genotype in Alzheimer's Disease

J. Growdon, B. Hyman, S. Greenberg, 1996, Annals of the New York Academy of Sciences.

Clinical and pathological correlates of apolipoprotein E ε4 in Alzheimer's disease

B T Hyman, J J Locascio, J. Growdon, 1996, Annals of neurology.

Frequency of the apolipoprotein E ε2 allele is diminished in sporadic Alzheimer disease

B. Hyman, G. Rebeck, H. West, 1994, Neuroscience Letters.

Association of apolipoprotein E ϵ2 and vasculopathy in cerebral amyloid angiopathy

S. Greenberg, A. Clatworthy, B. Hyman, 1998, Neurology.

Identification and modification of amyloid-independent phenotypes of APOE4 mice

S. Dumanis, G. Rebeck, G. Rebeck, 2016, Experimental Neurology.

Induction of the Cholesterol Transporter ABCA1 in Central Nervous System Cells by Liver X Receptor Agonists Increases Secreted Aβ Levels*

M. Irizarry, H. Fukumoto, G. Rebeck, 2002, The Journal of Biological Chemistry.

Endocytic disturbances distinguish among subtypes of alzheimer's disease and related disorders

C. van Broeckhoven, C. V. van Duijn, N. Bogdanovic, 2001, Annals of neurology.

Females exhibit more extensive amyloid, but not tau, pathology in an Alzheimer transgenic model

Paul S. Aisen, Nobuhisa Iwata, Mark P. Mattson, 2008, Brain Research.

LRP and senile plaques in Alzheimer's disease: colocalization with apolipoprotein E and with activated astrocytes.

B. Hyman, K. Arélin, M. Irizarry, 2002, Brain research. Molecular brain research.

Elevation of cystatin C in susceptible neurons in Alzheimer's disease.

J. Growdon, R. Nitsch, M. Irizarry, 2001, The American journal of pathology.

Apolipoprotein E affects the amount, form, and anatomical distribution of amyloid β-peptide deposition in homozygous APPV717F transgenic mice

B. Hyman, S. Paul, M. Irizarry, 2000, Acta Neuropathologica.

K. Manaye, P. Mouton, D. Lei, 2011, AGE.

O-glycosylation on cerebrospinal fluid and plasma Apolipoprotein E differs in the lipid binding domain.

Oliver C. Grant, R. Woods, G. Rebeck, 2019, Glycobiology.

Cerebrospinal fluid levels of amyloid β‐protein in alzheimer's disease: Inverse correlation with severity of dementia and effect of apolipoprotein e genotype

J. Growdon, B. Hyman, R. Wurtman, 1995, Annals of neurology.

Iowa variant of familial Alzheimer's disease: accumulation of posttranslationally modified AbetaD23N in parenchymal and cerebrovascular amyloid deposits.

Yun Lu, S. Greenberg, J. Ghiso, 2010, The American journal of pathology.

Lack of association of the cholesterol 24-hydroxylase (CYP46) intron 2 polymorphism with Alzheimer’s disease

B. Hyman, M. Ingelsson, M. Irizarry, 2004, Neuroscience Letters.

High-fat diet increases gliosis and immediate early gene expression in APOE3 mice, but not APOE4 mice

G. Rebeck, Nahdia S Jones, Katarina Q. Watson, 2021, Journal of Neuroinflammation.

Cancer Chemotherapy Related Cognitive Impairment and the Impact of the Alzheimer’s Disease Risk Factor APOE

G. Rebeck, Harvey R. Fernandez, S. Flowers, 2020, Cancers.

Reduced apolipoprotein ϵ4 allele frequency in the oldest old Alzheimer's patients and cognitively normal individuals

Bradley T. Hyman, Thomas T. Perls, B. Hyman, 1994, Neurology.

α2‐Macroglobulin Enhances the Clearance of Endogenous Soluble β‐Amyloid Peptide via Low‐Density Lipoprotein Receptor‐Related Protein in Cortical Neuron

B. Hyman, D. Strickland, G. Rebeck, 1999 .

APOE ε3/ε4 heterozygotes have an elevated proportion of apolipoprotein E4 in cerebrospinal fluid relative to plasma, independent of Alzheimer’s disease diagnosis

B. Hyman, L. Wahlund, M. Ingelsson, 2003, Experimental Neurology.

Association of apolipoprotein E epsilon2 and vasculopathy in cerebral amyloid angiopathy.

B. Hyman, S. Greenberg, J. Vonsattel, 1998, Neurology.

Genetic association of a cystatin C gene polymorphism with late-onset Alzheimer disease.

B T Hyman, J. Growdon, B. Hyman, 2000, Archives of neurology.

Genetic association of an (α2-macroglobulin (Val1000lle) polymorphism and Alzheimer's disease

M. Albert, D. Blacker, J. Growdon, 1998 .

Confirmation of an association between a polymorphism in exon 3 of the low‐density lipoprotein receptor‐related protein gene and Alzheimer's disease

B. Hyman, G. Rebeck, E. Hollenbach, 1998, Neurology.

A newly identified polymorphism in the apolipoprotein E enhancer gene region is associated with Alzheimer's disease and strongly with the epsilon 4 allele

B. Hyman, T. Gómez-Isla, G. Rebeck, 1996, Neurology.

Apolipoprotein E epsilon 4 and cerebral hemorrhage associated with amyloid angiopathy.

B. Hyman, S. Greenberg, T. Gómez-Isla, 1995, Annals of neurology.

Plasma β‐Amyloid Peptide, Transforming Growth Factor‐β1, and Risk for Cerebral Amyloid Angiopathy

S. Younkin, L. Younkin, J. Rosand, 2000 .

Apolipoprotein E Receptors Mediate Neurite Outgrowth through Activation of p44/42 Mitogen-activated Protein Kinase in Primary Neurons*

B. Hyman, G. Rebeck, Z. Qiu, 2004, Journal of Biological Chemistry.

α2-Macroglobulin Exposure Reduces Calcium Responses to N-Methyl-d-Aspartate via Low Density Lipoprotein Receptor-related Protein in Cultured Hippocampal Neurons*

B. Hyman, D. Strickland, G. Rebeck, 2002, The Journal of Biological Chemistry.

Clinical and Neuropathological Correlates of Apolipoprotein E Genotype in Alzheimer's Disease ‐ Window on Molecular Epidemiology a

T. Gómez-Isla, B. Hyman, G. Rebeck, 1996, Annals of the New York Academy of Sciences.

Reduced apolipoprotein epsilon 4 allele frequency in the oldest old Alzheimer's patients and cognitively normal individuals.

B T Hyman, B. Hyman, L. Lipsitz, 1994, Neurology.

Abeta species, including IsoAsp23 Abeta, in Iowa-type familial cerebral amyloid angiopathy.

S. Greenberg, T. Shirasawa, Takahiko Shimizu, 2003, Acta neuropathologica.

The pattern of NADPH-diaphorase staining, a marker of nitric oxide synthase activity, is altered in the perforant pathway terminal zone in Alzheimer's disease

B. Hyman, G. Rebeck, K. Marzloff, 1993, Neuroscience Letters.

Microglial low-density lipoprotein receptor-related protein 1 modulates c-Jun N-terminal kinase activation

I. Mikhailenko, D. Strickland, G. Rebeck, 2009, Journal of Neuroimmunology.

Low CD4 nadir exacerbates the impacts of APOE ε4 on functional connectivity and memory in adults with HIV.

Raymond Scott Turner, Xiong Jiang, R. Ellis, 2021, AIDS.

Quantitation of apoE Domains in Alzheimer Disease Brain Suggests a Role for apoE in Aß Aggregation

B. Hyman, S. Greenberg, G. Rebeck, 2001, Journal of neuropathology and experimental neurology.

Alpha2-macroglobulin enhances the clearance of endogenous soluble beta-amyloid peptide via low-density lipoprotein receptor-related protein in cortical neurons.

B. Hyman, D. Strickland, G. Rebeck, 1999, Journal of neurochemistry.

Vascular Changes in Iowa‐Type Hereditary Cerebral Amyloid Angiopathy

S. Greenberg, Y. Shin, G. Rebeck, 2002, Annals of the New York Academy of Sciences.

Lack of association of a polymorphism in the low-density lipoprotein receptor-related protein gene with Alzheimer disease.

B. Hyman, T. Gómez-Isla, A. Clatworthy, 1997, Archives of neurology.

Lack of allelic imbalance in APOE ϵ3/4 brain mRNA expression in Alzheimer's disease

B. Hyman, W. Growdon, G. Rebeck, 1999, Neuroscience Letters.

Apolipoprotein E4 and beta amyloid in senile plaques and cerebral blood vessels of aged rhesus monkeys.

B T Hyman, J. Tigges, B. Hyman, 1994, The American journal of pathology.

Apolipoprotein E--Correction

B. Hyman, G. Rebeck, 1995, Neurology.

Genetic association of an alpha2-macroglobulin (Val1000lle) polymorphism and Alzheimer's disease.

M. Albert, D. Blacker, J. Growdon, 1998, Human molecular genetics.

Alzheimer's disease with and without coexisting Parkinson's disease changes

B. Hyman, M. Gearing, S. Mirra, 1995, Neurology.

ApoE Lipidation as a Therapeutic Target in Alzheimer’s Disease

G. Rebeck, G. Rebeck, M. F. Lanfranco, 2020, International journal of molecular sciences.

APOE epsilon 3/ epsilon 4 heterozygotes have an elevated proportion of apolipoprotein E4 in cerebrospinal fluid relative to plasma, independent of Alzheimer's disease diagnosis.

B. Hyman, L. Wahlund, M. Ingelsson, 2003, Experimental neurology.

Independent APOE4 knock‐in mouse models display reduced brain APOE protein, altered neuroinflammation, and simplification of dendritic spines

G. Rebeck, G. Rebeck, M. Nelson, 2022, Journal of neurochemistry.

Soluble apoE/Aβ complex: mechanism and therapeutic target for APOE4-induced AD risk

S. Estus, G. Bu, M. LaDu, 2014, Molecular Neurodegeneration.

Iowa Variant of Familial Alzheimer’s Disease Accumulation of Posttranslationally Modified A (cid:1) D23N in Parenchymal and Cerebrovascular Amyloid Deposits

Yun Lu, S. Greenberg, J. Ghiso, 2010 .

Functional integrity of neural systems related to memory in Alzheimer's disease.

B. Hyman, N. Simonian, G. Rebeck, 1994, Progress in brain research.

Aging reduces glial uptake and promotes extracellular accumulation of Aβ from a lentiviral vector

G. Rebeck, G. Rebeck, Wenjuan Zhao, 2014, Front. Aging Neurosci..

APOE in the normal brain

G. Rebeck, G. Rebeck, S. Flowers, 2020, Neurobiology of Disease.

Modulation of beta-amyloid precursor protein processing by the low density lipoprotein receptor-related protein (LRP). Evidence that LRP contributes to the pathogenesis of Alzheimer's disease.

D. Strickland, G. Rebeck, J. Beers, 2000, The Journal of biological chemistry.

Progression of Cerebral Amyloid Angiopathy in Transgenic Mouse Models of Alzheimer Disease

Brian J Bacskai, Matthew P Frosch, B. Hyman, 2005, Journal of neuropathology and experimental neurology.

Apolipoprotein E ϵ4/4 in a Neuropathologically Normal Very Elderly Individual

Bradley T. Hyman, J. Growdon, B. Hyman, 1996 .

Apolipoprotein E epsilon4/4 in a neuropathologically normal very elderly individual.

B T Hyman, J. Growdon, B. Hyman, 1996, Archives of neurology.