Ningyuan Wang

发表

Acceleration of the Healing Process and Myocardial Regeneration May Be Important as a Mechanism of Improvement of Cardiac Function and Remodeling by Postinfarction Granulocyte Colony–Stimulating Factor Treatment

M. Arai, G. Takemura, S. Minatoguchi, 2004, Circulation.

The anti‐diabetic drug miglitol is protective against anginal ischaemia through a mechanism independent of regional myocardial blood flow in the dog

M. Arai, G. Takemura, S. Minatoguchi, 2005, Clinical and experimental pharmacology & physiology.

Antidiabetic drug miglitol inhibits myocardial apoptosis involving decreased hydroxyl radical production and Bax expression in an ischaemia/reperfusion rabbit heart

M. Arai, G. Takemura, S. Minatoguchi, 2004, British journal of pharmacology.

N-Methyl-1-deoxynojirimycin (MOR-14), an α-glucosidase inhibitor, markedly improves postischemic left ventricular dysfunction

M. Arai, G. Takemura, S. Minatoguchi, 2000, Heart and Vessels.

Cilnidipine, an N+L-Type Dihydropyridine Ca Channel Blocker, Suppresses the Occurrence of Ischemia/Reperfusion Arrhythmia in a Rabbit Model of Myocardial Infarction

M. Arai, H. Nagai, G. Takemura, 2005, Hypertension Research.

In vivo hepatocyte growth factor gene transfer reduces myocardial ischemia-reperfusion injury through its multiple actions.

M. Arai, S. Yasuda, H. Ushikoshi, 2007, Journal of cardiac failure.

The role of serotonin in ischemic cellular damage and the infarct size-reducing effect of sarpogrelate, a 5-hydroxytryptamine-2 receptor blocker, in rabbit hearts.

M. Arai, G. Takemura, S. Minatoguchi, 2002, Journal of the American College of Cardiology.

Quinaprilat Reduces Myocardial Infarct Size Involving Nitric Oxide Production and Mitochondrial KATP Channel in Rabbits

M. Arai, G. Takemura, S. Minatoguchi, 2003, Journal of cardiovascular pharmacology.

Benidipine Reduces Myocardial Infarct Size Involving Reduction of Hydroxyl Radicals and Production of Protein Kinase C-Dependent Nitric Oxide in Rabbits

M. Arai, H. Nagai, G. Takemura, 2004, Journal of cardiovascular pharmacology.

Nicorandil improves post-ischemic myocardial dysfunction in association with opening the mitochondrial K(ATP) channels and decreasing hydroxyl radicals in isolated rat hearts.

M. Arai, S. Yasuda, I. Kawamura, 2006, Circulation journal : official journal of the Japanese Circulation Society.

Autologous bone marrow cell transplantation improves left ventricular function in rabbit hearts with cardiomyopathy via myocardial regeneration-unrelated mechanisms

M. Arai, G. Takemura, S. Minatoguchi, 2006, Heart and Vessels.

Celiprolol, a Selective β1-Blocker, Reduces the Infarct Size Through Production of Nitric Oxide in a Rabbit Model of Myocardial Infarction

M. Arai, G. Takemura, S. Minatoguchi, 2007 .

Sheng-Mai-San is protective against post-ischemic myocardial dysfunction in rats through its opening of the mitochondrial KATP channels.

M. Arai, G. Takemura, S. Minatoguchi, 2002, Circulation journal : official journal of the Japanese Circulation Society.

Lindera strychnifolia is protective against post-ischemic myocardial dysfunction through scavenging hydroxyl radicals and opening the mitochondrial KATP channels in isolated rat hearts.

M. Arai, G. Takemura, S. Minatoguchi, 2004, The American journal of Chinese medicine.

Role Of Protein Kinase C, KATP Channels And Dna Fragmentation In The Infarct Size‐Reducing Effects Of The Free Radical Scavenger T‐0970

M. Arai, G. Takemura, S. Minatoguchi, 2001, Clinical and experimental pharmacology & physiology.

Role of protein kinase C in the reduction of infarct size by N‐methyl‐1‐deoxynojirimycin, an α‐1,6‐glucosidase inhibitor

M. Arai, K. Kuwano, G. Takemura, 2001, British journal of pharmacology.

Cross‐Talk among Noradrenaline, Adenosine and Protein Kinase C in the Mechanisms of Ischemic Preconditioning in Rabbits

Yoshihiro Uno, M. Arai, G. Takemura, 2003, Journal of cardiovascular pharmacology.