V. Vanasco

发表

α-Lipoic acid protects kidney from oxidative stress and mitochondrial dysfunction associated to inflammatory conditions.

N. Magnani, P. Evelson, M. C. Cimolai, 2014, Food & function.

Selective TNF-α targeting with infliximab attenuates impaired oxygen metabolism and contractile function induced by an acute exposure to air particulate matter.

M. Garcés, L. Cáceres, N. Magnani, 2015, American journal of physiology. Heart and circulatory physiology.

The oxidative stress and the mitochondrial dysfunction caused by endotoxemia are prevented by α-lipoic acid

P. Evelson, M. C. Cimolai, V. Vanasco, 2008, Free radical research.

Cardiac mitochondrial biogenesis in endotoxemia is not accompanied by mitochondrial function recovery.

D. Corach, M. Vaccaro, N. Magnani, 2014, Free radical biology & medicine.

Cardiac dysfunction, mitochondrial architecture, energy production, and inflammatory pathways: Interrelated aspects in endotoxemia and sepsis.

V. Vanasco, S. Alvarez, T. Vico, 2016, The international journal of biochemistry & cell biology.

In situ and real time muscle chemiluminescence determines singlet oxygen involvement in oxidative damage during endotoxemia.

A. Boveris, P. Evelson, V. Vanasco, 2010, Chemico-biological interactions.

Endotoxemia impairs heart mitochondrial function by decreasing electron transfer, ATP synthesis and ATP content without affecting membrane potential

A. Boveris, N. Magnani, P. Evelson, 2012, Journal of Bioenergetics and Biomembranes.

Alpha-synuclein mitochondrial interaction leads to irreversible translocation and complex I impairment.

A. Alaimo, V. Vanasco, S. Alvarez, 2018, Archives of biochemistry and biophysics.

Mitochondrial bioenergetics links inflammation and cardiac contractility in endotoxemia

D. Corach, M. Garcés, P. Evelson, 2019, Basic Research in Cardiology.

Mitochondrial H2O2 metabolism as central event of heart complex I syndrome in early diabetes.

V. Vanasco, S. Alvarez, L. Valdez, 2023, Free radical biology & medicine.

Energy management and mitochondrial dynamics in cerebral cortex during endotoxemia.

P. Evelson, V. Vanasco, T. Marchini, 2021, Archives of biochemistry and biophysics.

Mitochondrial Dynamics and VMP1-Related Selective Mitophagy in Experimental Acute Pancreatitis

M. Vaccaro, D. Grasso, V. Vanasco, 2021, Frontiers in Cell and Developmental Biology.

Chronic exposure to polluted urban air aggravates myocardial infarction by impaired cardiac mitochondrial function and dynamics

M. Garcés, L. Cáceres, N. Magnani, 2022, Environmental Pollution.

Acute hypobaric hypoxia and cardiac energetic response in prepubertal rats: Role of nitric oxide

V. Vanasco, B. Piotrkowski, L. Costa, 2021, Experimental physiology.

Time course of systemic oxidative stress and inflammatory response induced by an acute exposure to Residual Oil Fly Ash.

N. Magnani, P. Evelson, V. Vanasco, 2014, Toxicology and applied pharmacology.

Urban air pollution induces alterations in redox metabolism and mitochondrial dysfunction in mice brain cortex.

M. Garcés, L. Cáceres, N. Magnani, 2021, Archives of Biochemistry and Biophysics.

Reactive oxygen species produced by NADPH oxidase and mitochondrial dysfunction in lung after an acute exposure to residual oil fly ashes.

N. Magnani, P. Evelson, V. Vanasco, 2013, Toxicology and applied pharmacology.

Oxidative metabolism in the cardiorespiratory system after an acute exposure to nickel-doped nanoparticles in mice.

G. Valacchi, A. Mebert, Maria V. Tuttolomondo, 2021, Toxicology.

The Proinflammatory RAGE/NF-κB Pathway Is Involved in Neuronal Damage and Reactive Gliosis in a Model of Sleep Apnea by Intermittent Hypoxia

Jerónimo Lukin, V. Vanasco, S. Alvarez, 2014, PloS one.