T. Campbell

发表

Human macrophages contain a stretch-sensitive potassium channel that is activated by adherence and cytokines

S. Breit, D. K. Martin, M. Bootcov, 1995, The Journal of Membrane Biology.

Molecular basis of slow activation of the human ether‐á‐go‐go related gene potassium channel

Jamie I Vandenberg, David J Smith, J. Vandenberg, 2004, The Journal of physiology.

Tryptophan scanning mutagenesis of the HERG K+ channel: the S4 domain is loosely packed and likely to be lipid exposed

Jamie I Vandenberg, J. Vandenberg, T. Campbell, 2005, The Journal of physiology.

Effect of S5P α‐helix charge mutants on inactivation of hERG K+ channels

A. Hill, J. Vandenberg, T. Campbell, 2006 .

Effect of S5P alpha-helix charge mutants on inactivation of hERG K+ channels.

J. Zhao, A. Hill, J. Vandenberg, 2006, The Journal of physiology.

The Intracellular Chloride Ion Channel Protein CLIC1 Undergoes a Redox-controlled Structural Transition*

W. D. Fairlie, Matthew Z. DeMaere, M. DeMaere, 2004, Journal of Biological Chemistry.

SELECTIVE DEPRESSION OF MAXIMUM RATE OF DEPOLARIZATION OF GUINEA‐PIG VENTRICULAR ACTION POTENTIALS BY AMIODARONE AND LIGNOCAINE IN SIMULATED ISCHAEMIA: COMPARISON WITH ENCAINIDE

T. Campbell, P. D. Hemsworth, P. Hemsworth, 1990, Clinical and experimental pharmacology & physiology.

Resting, and rate‐dependent depression of of guinea‐pig ventricular action potentials by amiodarone and desethylamiodarone

T. Campbell, R. Pallandi, 1987, British journal of pharmacology.

Not All hERG Pore Domain Mutations Have a Severe Phenotype: G584S Has an Inactivation Gating Defect with Mild Phenotype Compared to G572S, Which Has a Dominant Negative Trafficking Defect and a Severe Phenotype

Jamie I Vandenberg, Adam P Hill, Anthony Varghese, 2009, Journal of cardiovascular electrophysiology.

Importance of physico‐chemical properties in determining the kinetics of the effects of Class I antiarrhythmic drugs on maximum rate of depolarization in guinea‐pig ventricle

T. Campbell, 1983, British journal of pharmacology.

Drug Binding to the Inactivated State Is Necessary but Not Sufficient for High-Affinity Binding to Human Ether-à-go-go-Related Gene Channels

Jamie I Vandenberg, P. Kuchel, J. Vandenberg, 2008, Molecular Pharmacology.

Inherited cardiac arrhythmia syndromes: What have they taught us about arrhythmias and anti‐arrhythmic therapy?

J. Vandenberg, T. Campbell, R. Subbiah, 2004, Clinical and experimental pharmacology & physiology.

Randomized controlled trial of intraaortic balloon counterpulsation in early myocardial infarction with acute heart failure.

M. O'Rourke, N. Sammel, T. Campbell, 1981, The American journal of cardiology.

Update on the Use of Magnesium as an Antiarrhythmic Drug

T. Campbell, 2000 .

Effect of pravastatin compared with placebo initiated within 24 hours of onset of acute myocardial infarction or unstable angina: the Pravastatin in Acute Coronary Treatment (PACT) trial.

I. Meredith, J. Amerena, J. Sloman, 2004, American heart journal.

Blockade by N‐3 polyunsaturated fatty acid of the Kv4.3 current stably expressed in Chinese hamster ovary cells

S. Valenzuela, S. Breit, T. Campbell, 1999, British journal of pharmacology.

Inhibition of HERG potassium channels by the antimalarial agent halofantrine

T J Campbell, S. Valenzuela, S. Breit, 2000, British journal of pharmacology.

Comparative Study of the Effects of Erythromycin and Roxithromycin on Action Potential Duration and Potassium Currents in Canine Purkinje Fibers and Rabbit Myocardium

Donald K. Martin, T. Campbell, J. Bursill, 1998, Journal of cardiovascular pharmacology and therapeutics.

N-3 polyunsaturated fatty acids and cardiac mortality.

T. Campbell, B. Walker, C. B. Singleton, 2000, Australian and New Zealand journal of medicine.

The place of amiodarone: an overview of the four recent large controlled trials.

T. Campbell, Terence J. Campbell, 1997, Australian and New Zealand journal of medicine.

Digitalis for patients with heart failure in sinus rhythm

T. Campbell, 1993, The Medical journal of Australia.

A potassium ion channel is involved in cytokine production by activated human macrophages

S. Breit, T. Campbell, M. R. Qiu, 2002, Clinical and experimental immunology.

Clinical Use of Class Ia Antiarrhythmic Drugs

T. Campbell, 1989 .

Therapeutic drug monitoring: antiarrhythmic drugs.

K. Williams, T. Campbell, Terence J. Campbell, 1998, British journal of clinical pharmacology.

Kinetics of onset of rate-dependent effects of Class I antiarrhythmic drugs are important in determining their effects on refractoriness in guinea-pig ventricle, and provide a theoretical basis for their subclassification.

T. Campbell, 1983, Cardiovascular research.

DIFFERENTIAL EFFECTS OF ANTIARRHYTHMIC AGENTS ON POST‐PAUSE REPOLARIZATION IN CARDIAC PURKINJE FIBRES

T. Campbell, J. Bursill, K. Wyse, 1996, Clinical and experimental pharmacology & physiology.

Inhibition of ATP-sensitive potassium channels in cardiac myocytes by the novel class III antiarrhythmic agent MS-551.

Y. Nakaya, T. Campbell, J. Bursill, 1995, Pharmacology & toxicology.

VOLTAGE‐ AND TIME‐DEPENDENT DEPRESSION OF MAXIMUM RATE OF DEPOLARIZATION OF GUINEA‐PIG VENTRICULAR ACTION POTENTIALS BY TWO STEROIDAL ANTIARRHYTHMIC DRUGS, CCI 22277 AND ORG 6001

T. Campbell, 1982, British journal of pharmacology.

Beta-blockers for ventricular arrhythmias: have we underestimated their value?

T. Campbell, 1996, Australian and New Zealand journal of medicine.

Role of Non-Steroidal Anti-Inflammatory Drugs (NSAIDs) in Modulating Vascular Smooth Muscle Cells by Activating Large-Conductance Potassium Ion Channels

Donald K. Martin, T. Campbell, P. Macdonald, 2012 .

actions ofcaptopril: lack ofdirect antiarrhythmic effects

T. Campbell, 1989 .

Proarrhythmic effects of an oxygen-derived free radical generating system on action potentials recorded from guinea pig ventricular myocardium: a possible cause of reperfusion-induced arrhythmias.

T. Campbell, R. Pallandi, M. Perry, 1987, Circulation research.

The effects of nadolol on various cardiac tissues in normoxia, and on atrial muscle in simulated ischaemia.

T. Campbell, E. Williams, 1982, European journal of pharmacology.

Resting and Rate‐Dependent Depression of Maximum Rate of Depolarisation (Vmax) in Guinea Pig Ventricular Action Potentials by Mexiletine, Disopyramide, and Encainide

T. Campbell, 1983, Journal of cardiovascular pharmacology.

Cellular electrophysiological effects of d‐and dl‐sotalol in guinea‐pig sinoatrial node, atrium and ventricle and human atrium: differential tissue sensitivity

T. Campbell, 1987, British journal of pharmacology.

Reply from Jamie I. Vandenberg, Adam P. Hill, Terence J. Campbell, Catherine E. Clarke

A. Hill, J. Vandenberg, T. Campbell, 2006 .

The HERG K+ channel: progress in understanding the molecular basis of its unusual gating kinetics

P. Kuchel, A. Torres, J. Vandenberg, 2004, European Biophysics Journal.

Clozapine and sudden death.

S. Valenzuela, S. Breit, T. Campbell, 2001, Journal of clinical psychopharmacology.

DIFFERENTIAL EFFECTS ON ACTION POTENTIAL DURATION OF CLASS IA, B AND C ANTIARRHYTHMIC DRUGS: MODULATION BY STIMULATION RATE AND EXTRACELLULAR K+ CONCENTRATION

T. Campbell, R. Pallandi, K. Wyse, 1991, Clinical and experimental pharmacology & physiology.

Long-term survival after counterpulsation for medically refractory heart failure complicating myocardial infarction and cardiac surgery.

M. O'Rourke, D. Kuchar, T. Campbell, 1987, European heart journal.

Basic pharmacology of cardiac drugs

T. Campbell, Aran Maree, 2001, Interventional Cardiology and Cardiac Catheterisation.

Depression of maximum rate of depolarization of guinea‐pig ventricular action potentials by metabolites of encainide

T. Campbell, P. D. Hemsworth, Paul D. Hemsworth, 1989, British journal of pharmacology.

Subclassification of Class I antiarrhythmic drugs: Enhanced relevance after CAST

T. Campbell, 1992, Cardiovascular Drugs and Therapy.

Effect of the class III antiarrhythmic agent E-4031 on the ATP-sensitive potassium channel in rabbit ventricular myocytes.

T. Campbell, J. Bursill, K. Wyse, 1996, Pharmacology & toxicology.

Comparative effects of azimilide and ambasilide on the human ether-a-go-go-related gene (HERG) potassium channel.

T J Campbell, S. Valenzuela, S. Breit, 2000, Cardiovascular research.

Action Potential Prolongation Exhibits Simple Dose‐Dependence for Sotalol, but Reverse

T. Campbell, K. Wyse, V. Ye, 1993, Journal of cardiovascular pharmacology.

Antiarrhythmic drugs

T. Campbell, 1994, The Medical journal of Australia.

Class III Antiarrhythmic Effects of Dofetilide in Rabbit Atrial Myocardium

N. Lovell, T. Campbell, R. Pallandi, 1996 .

The S631A mutation causes a mechanistic switch in the block of hERG channels by CnErg1.

P. Kuchel, A. Hill, J. Vandenberg, 2007, Biophysical journal.

Voltage- and time-dependent depression of maximum rate of depolarisation of guinea-pig ventricular action potentials by two new antiarrhythmic drugs, flecainide and lorcainide.

T. Campbell, E. V. Vaughan Williams, T. Campbell, 1983, Cardiovascular research.

Electrophysiological effects of labetolol on rabbit atrial, ventricular and Purkinje cells, in normoxia and hypoxia.

J. Millar, T. Campbell, E. M. Vaughn Williams, 1982, Cardiovascular research.

Effects of hyperkalaemia on the depression of maximum rate of depolarization by class I antiarrhythmic agents in guinea‐pig myocardium

T. Campbell, K. Wyse, V. Ye, 1993, British journal of pharmacology.

Quinidine but Not Disopyramide Prolongs Cardiac Purkinje Fiber Action Potentials After a Pause

T. Campbell, J. Bursill, K. Wyse, 1994, Journal of cardiovascular pharmacology.

Proarrhythmic actions of antiarrhythmic drugs: a review.

T. Campbell, 1990, Australian and New Zealand journal of medicine.

Nifedipine in the treatment of life threatening Prinzmetal angina.

T. Campbell, G. Michell, J. Hickie, 1979, Australian and New Zealand journal of medicine.

Antiarrhythmic agents

T. Campbell, 1984, The Medical journal of Australia.

Characteristics of cardiac action potentials in marsupials

T. Campbell, 2004, Journal of Comparative Physiology B.

Inhibition of the human ether‐a‐go‐go‐related gene (HERG) potassium channel by cisapride: affinity for open and inactivated states

T J Campbell, S. Breit, T. Campbell, 1999, British journal of pharmacology.

Molecular Cloning and Expression of a Chloride Ion Channel of Cell Nuclei*

S. Valenzuela, S. Breit, M. Bootcov, 1997, The Journal of Biological Chemistry.

Recombinant CLIC1 (NCC27) Assembles in Lipid Bilayers via a pH-dependent Two-state Process to Form Chloride Ion Channels with Identical Characteristics to Those Observed in Chinese Hamster Ovary Cells Expressing CLIC1*

W. D. Fairlie, P. Curmi, J. Matthews, 2002, The Journal of Biological Chemistry.

Functional characterization of the NCC27 nuclear protein in stable transfected CHO‐K1 cells

S. Valenzuela, S. Breit, M. Mazzanti, 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Intravenous sotalol for the treatment of atrial fibrillation and flutter after cardiopulmonary bypass. Comparison with disopyramide and digoxin in a randomised trial.

T. Campbell, J. Morgan, T. Gavaghan, 1985, British heart journal.

Treatment of atrial arrhythmias after cardiac surgery with intravenous disopyramide.

T. Campbell, J. Morgan, 1980, Australian and New Zealand journal of medicine.

Atrial tachyarrhythmias after cardiac surgery: results of disopyramide therapy.

M. Feneley, T. Campbell, J. Morgan, 1985, Australian and New Zealand journal of medicine.

The HERG K+ channel: progress in understanding the molecular basis of its unusual gating kinetics

P. Kuchel, A. Torres, J. Vandenberg, 2004, European Biophysics Journal.

The nuclear chloride ion channel NCC27 is involved in regulation of the cell cycle

E. Musgrove, S. Valenzuela, S. Breit, 2000, The Journal of physiology.

ELECTROPHYSIOLOGICAL AND OTHER EFFECTS ON RABBIT HEARTS OF CCI 22277, A NEW STEROIDAL ANTIARRHYTHMIC DRUG

T. Campbell, E. Williams, 1982, British journal of pharmacology.

Subclassification of Class I Antiarrhythmic Drugs

T. Campbell, 1989 .

Differing electrophysiological effects of class IA, IB and IC antiarrhythmic drugs on guinea‐pig sinoatrial node

T. Campbell, 1987, British journal of pharmacology.

Modulation of the Electrophysiologic Actions of E-4031 and Dofetilide by Hyperkalemia and Acidosis in Rabbit Ventricular Myocytes

Donald K. Martin, T. Campbell, J. Bursill, 1997, Journal of cardiovascular pharmacology and therapeutics.

Flecainide compared with a combination of digoxin and disopyramide for acute atrial arrhythmias after cardiopulmonary bypass.

R. Kelly, C. Thorburn, T. Campbell, 1988, British heart journal.

Extracellular Acidosis Modulates Drug Block of Kv4.3 Currents by Flecainide and Quinidine

S. Breit, J. Vandenberg, T. Campbell, 2003, Journal of cardiovascular electrophysiology.

Inhibition of HERG channels stably expressed in a mammalian cell line by the antianginal agent perhexiline maleate

T J Campbell, S. Breit, T. Campbell, 1999, British journal of pharmacology.

Cardiac electrophysiological actions of captopril: lack of direct antiarrhythmic effects

T. Campbell, Paul D. Hemsworth, Regan T. Pallandi, 1989, British journal of pharmacology.

Treatment of atrial fibrillation: time for change?

T. Campbell, 1992, The Medical journal of Australia.

Crystal Structure of a Soluble Form of the Intracellular Chloride Ion Channel CLIC1 (NCC27) at 1.4-Å Resolution*

Matthew Z. DeMaere, P. Curmi, S. Valenzuela, 2001, The Journal of Biological Chemistry.

Mechanism of block of the hERG K+ channel by the scorpion toxin CnErg1.

M. Sunde, A. Hill, J. Vandenberg, 2007, Biophysical journal.

The prevalence of arrhythmias in hypertrophic cardiomyopathy: role of ambulatory monitoring and signal-averaged electrocardiography.

R. Kelly, D. Kuchar, T. Campbell, 1986, Australian and New Zealand journal of medicine.

Selective depression of conduction of premature action potentials in canine Purkinje fibres by class Ib antiarrhythmic drugs: comparison with Ia and Ic drugs.

T. Campbell, R. Pallandi, 1988, Cardiovascular research.

Effects of components of ischemia on the Kv4.3 current stably expressed in Chinese hamster ovary cells.

S. Valenzuela, S. Breit, T. Campbell, 2002, Journal of molecular and cellular cardiology.

HERG K+ channels: friend and foe.

T J Campbell, J. Vandenberg, T. Campbell, 2001, Trends in pharmacological sciences.

Effects of Hyperkalemia, Acidosis, and Hypoxia on the Depression of Maximum Rate of Depolarization by Class I Antiarrhythmic Drugs in Guinea Pig Myocardium: Differential Actions of Class Ib and Ic Agents

T. Campbell, Kenneth R. Wyse, P. Hemsworth, 1991, Journal of cardiovascular pharmacology.

Structure of the HERG K+ Channel S5P Extracellular Linker

P. Kuchel, M. Sunde, S. Breit, 2003, Journal of Biological Chemistry.

Lidocaine Shows Greater Selective Depression of Depolarized and Acidotic Myocardium than Propafenone: Possible Implications for Proarrhythmia

T. Campbell, K. Wyse, V. Ye, 1993, Journal of cardiovascular pharmacology.

Clinical Efficacy of Cardiac Glycosides for Arrhythmias

T. Campbell, 1989 .

Flecainide compared witha combination ofdigoxin anddisopyramide foracuteatrial arrhythmias after cardiopulmonary bypass

R. Kelly, A. Keogh, C. Thorburn, 1988 .

Kinetics of Drug Interaction with the Kv11.1 Potassium Channel

Stefan A. Mann, A. Hill, J. Vandenberg, 2014, Molecular Pharmacology.

Therapeutic drug monitoring: antiarrhythmic drugs.

K. Williams, T. Campbell, 2008, British journal of clinical pharmacology.

The Novel Class III Antiarrhythmic Agent MS-551 Blocks the Cardiac Inward Rectifier With Greater Potency Than Sotalol or E-4031: Possible Relevance to Reverse Use Dependence

Donald K. Martin, Y. Nakaya, T. Campbell, 1997, Journal of cardiovascular pharmacology and therapeutics.

Effect of Dofetilide and d-Sotalol on the ATP-Sensitive Potassium Channel of Rabbit Ventricular Myocytes

Donald K. Martin, T. Campbell, J. Bursill, 1996, Journal of cardiovascular pharmacology and therapeutics.

Proarrhythmic complications of sotalol.

T. Campbell, 1996, Australian and New Zealand journal of medicine.