D. Scott

发表

Salivary antioxidants in patients with type 1 or 2 diabetes and inflammatory periodontal disease: a case-control study.

P. Gümüş, N. Buduneli, Ş. Çetinkalp, 2009, Journal of periodontology.

Effects of smoking and gingival inflammation on salivary antioxidant capacity.

N. Buduneli, Samuel I Hawkins, D. Kinane, 2006, Journal of clinical periodontology.

Presence of Porphyromonas gingivalis in esophagus and its association with the clinicopathological characteristics and survival in patients with esophageal cancer

Xiaoshan Feng, R. Lamont, J. Potempa, 2016, Infectious Agents and Cancer.

Comparison of inherently essential genes of Porphyromonas gingivalis identified in two transposon-sequencing libraries.

R. Lamont, M. Hackett, E. Hendrickson, 2015, Molecular oral microbiology.

Periodontitis-specific molecular signatures in gingival crevicular fluid.

D. Scott, X. Xiang, K. Liu, 2010, Journal of periodontal research.

Tobacco-induced alterations to the Fourier-transform infrared spectrum of serum

D. Scott, A. Man, J. Borden, 2003, Journal of Molecular Medicine.

Fatty Acid Profiles in Smokers with Chronic Periodontitis

J. Bagaitkar, N. Buduneli, D. Renaud, 2011, Journal of dental research.

Tobacco Upregulates P. gingivalis Fimbrial Proteins Which Induce TLR2 Hyposensitivity

J. Bagaitkar, D. Renaud, D. Scott, 2010, PloS one.

Syk negatively regulates TLR4-mediated IFNβ and IL-10 production and promotes inflammatory responses in dendritic cells.

R. Lamont, Yi Kang, J. Shang, 2016, Biochimica et biophysica acta.

Porphyromonas gingivalis-Induced Reactive Oxygen Species Activate JAK2 and Regulate Production of Inflammatory Cytokines through c-Jun

R. Jotwani, R. Lamont, D. Scott, 2014, Infection and Immunity.

Tobacco use increases susceptibility to bacterial infection

David Steven Scott, J. Bagaitkar, D. Scott, 2008, Tobacco induced diseases.

Differentiation of Chronic and Aggressive Periodontitis by FTIR Spectroscopy

F. Severcan, P. Gümüş, N. Buduneli, 2016, Journal of dentistry research.

Increased infection with key periodontal pathogens during gestational diabetes mellitus.

P. Gümüş, N. Buduneli, D. Renaud, 2015, Journal of clinical periodontology.

Resolvin D1, resolvin D2 and maresin 1 activate the GSK3β anti-inflammatory axis in TLR4-engaged human monocytes

S. Uriarte, R. Lamont, D. Scott, 2016, Innate immunity.

The influence of nicotine on granulocytic differentiation – Inhibition of the oxidative burst and bacterial killing and increased matrix metalloproteinase-9 release

R. Palmer, D. Renaud, D. Scott, 2008, BMC Cell Biology.

Cigarette smoke-exposed neutrophils die unconventionally but are rapidly phagocytosed by macrophages

J. Potempa, B. Gajkowska, D. Scott, 2011, Cell Death and Disease.

Oral Endocannabinoids and Receptors

D. Scott, D. Saxena, H. Dukka, 2022 .

Potential Mechanisms Underlying Marijuana-Associated Periodontal Tissue Destruction

D. Saxena, D. Scott, H. Dukka, 2021, Journal of dental research.

Atypical cyclic di-AMP signaling is essential for Porphyromonas gingivalis growth and regulation of cell envelope homeostasis and virulence

R. Lamont, M. F. Moradali, D. Scott, 2022, npj Biofilms and Microbiomes.

This information is current as Production in Innate Immune Cells TLR-Mediated Inflammatory Cytokine The Role of JAK-3 in Regulating

J. Suttles, R. Jotwani, R. Lamont, 2013 .

2-Amino-4-(3,4-(methylenedioxy)benzylamino)-6-(3-methoxyphenyl)pyrimidine is an anti-inflammatory TLR-2, -4 and -5 response mediator in human monocytes

R. Lamont, D. Scott, Huaxin Zhou, 2015, Inflammation Research.

Scratching the surface – tobacco-induced bacterial biofilms

J. Bagaitkar, D. Scott, J. Hutcherson, 2015, Tobacco induced diseases.

Porphyromonas gingivalis genes conferring fitness in a tobacco-rich environment.

R. Lamont, J. Potempa, D. Scott, 2019, Molecular oral microbiology.

Inhibition of GSK3 Abolishes Bacterial-Induced Periodontal Bone Loss in Mice

R. Jotwani, J. Potempa, D. Scott, 2012, Molecular medicine.

Progression of periodontal disease in a maintenance population of smokers and non-smokers: a 3-year longitudinal study.

L. Lix, S. Gelskey, D. Scott, 2008, Journal of periodontology.

Increased local matrix metalloproteinase-8 expression in the periodontal connective tissues of smokers with periodontal disease.

D. Scott, A. Man, K. Liu, 2006, Biochimica et biophysica acta.

Noncanonical Activation of β-Catenin by Porphyromonas gingivalis

R. Lamont, J. Potempa, D. Scott, 2015, Infection and Immunity.

Inherently and Conditionally Essential Protein Catabolism Genes of Porphyromonas gingivalis.

D. Scott, Daniel P. Miller, 2020, Trends in microbiology.

Variation in tobacco use profiles obtained from periodontal maintenance patients.

L. Lix, D. Scott, J. Picard, 2007, Journal of dentistry.

Neutrophils alter epithelial response to Porphyromonas gingivalis in a gingival crevice model.

J. Potempa, F. Veillard, J. Bagaitkar, 2013, Molecular oral microbiology.

The role of smoking and gingival crevicular fluid markers on coronally advanced flap outcomes.

N. Buduneli, D. Renaud, D. Scott, 2014, Journal of periodontology.

Immune Recognition of the Epidemic Cystic Fibrosis Pathogen Burkholderia dolosa

M. Gadjeva, G. Pier, G. Priebe, 2017, Infection and Immunity.

Mechanisms of action of environmental factors--tobacco smoking.

R. Palmer, D. Scott, Ron F. Wilson, 2005, Journal of clinical periodontology.

Kan-Zhi Liu, David A Scott, N. Buduneli, 2010, Diabetology & metabolic syndrome.

Marijuana-Derived Cannabinoids Trigger a CB2/PI3K Axis of Suppression of the Innate Response to Oral Pathogens

R. Lamont, D. Scott, Gwyneth J. Lamont, 2019, Front. Immunol..

Neutrophils in periodontal inflammation.

D. Scott, J. Krauss, D. Scott, 2012, Frontiers of oral biology.

FOXO responses to Porphyromonas gingivalis in epithelial cells

R. Lamont, D. Scott, M. Sztukowska, 2015, Cellular microbiology.

Genes Contributing to Porphyromonas gingivalis Fitness in Abscess and Epithelial Cell Colonization Environments

R. Lamont, J. Potempa, M. Whiteley, 2017, Front. Cell. Infect. Microbiol..

Porphyromonas gingivalis RagB is a proinflammatory signal transducer and activator of transcription 4 agonist.

F. Yoshimura, J. Bagaitkar, D. Scott, 2015, Molecular oral microbiology.

Altered antigenic profiling and infectivity of Porphyromonas gingivalis in smokers and non-smokers with periodontitis.

R. Lamont, N. Buduneli, D. Scott, 2014, Journal of periodontology.

Tobacco Smoke Augments Porphyromonas gingivalis - Streptococcus gordonii Biofilm Formation

J. Bagaitkar, D. Renaud, D. Scott, 2011, PloS one.

Tobacco‐induced suppression of the vascular response to dental plaque

N. Buduneli, D. Scott, D. Scott, 2018, Molecular oral microbiology.

Smoking, Infectious Diseases and Innate Immune (Dys)function

J. Bagaitkar, D. Scott, D. Scott, 2011 .

GSK3β and the control of infectious bacterial diseases.

Richard J Lamont, David A Scott, Akhilesh Kumar, 2014, Trends in microbiology.

The Role of JAK-3 in Regulating TLR-Mediated Inflammatory Cytokine Production in Innate Immune Cells

J. Suttles, R. Jotwani, R. Lamont, 2013, The Journal of Immunology.

Microbiota and Metatranscriptome Changes Accompanying the Onset of Gingivitis

A. Ellington, R. Shroff, R. Lamont, 2018, mBio.

Tobacco-induced alterations to Porphyromonas gingivalis-host interactions.

J. Bagaitkar, D. Renaud, D. Scott, 2009, Environmental microbiology.

Porphyromonas gingivalis infection exacerbates oesophageal cancer and promotes resistance to neoadjuvant chemotherapy

R. Lamont, D. Scott, Lanhai Lu, 2021, British Journal of Cancer.

A putative lateral flagella of the cystic fibrosis pathogen Burkholderia dolosa regulates swimming motility and host cytokine production

J. Lipuma, G. Priebe, D. Renaud, 2018, PloS one.

Exploitation of the nicotinic anti-inflammatory pathway for the treatment of epithelial inflammatory diseases.

David A Scott, D. Scott, Michael Martin, 2006, World journal of gastroenterology.

JAK3 restrains inflammatory responses and protects against periodontal disease through Wnt3a signaling

Lanhai Lü, S. Uriarte, R. Lamont, 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

In vivo determination of multiple indices of periodontal inflammation by optical spectroscopy.

M. Sowa, D. Scott, X. Xiang, 2009, Journal of periodontal research.

Structure of RagB, a major immunodominant outer-membrane surface receptor antigen of Porphyromonas gingivalis.

J. Potempa, T. Goulas, I. Garcia-Ferrer, 2016, Molecular oral microbiology.

Microbiological and biochemical findings in relation to clinical periodontal status in active smokers, non-smokers and passive smokers

N. Buduneli, D. Scott, B. Kanmaz, 2019, Tobacco induced diseases.

Cotinine-induced convergence of the cholinergic and PI3 kinase-dependent anti-inflammatory pathways in innate immune cells.

D. Renaud, D. Scott, Michael Martin, 2008, Biochimica et biophysica acta.

Inhibition of serum‐ and glucocorticoid‐inducible kinase 1 enhances TLR‐mediated inflammation and promotes endotoxin‐driven organ failure

R. Lamont, D. Scott, Huaxin Zhou, 2015, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cigarette smoke reduces short chain fatty acid production by a Porphyromonas gingivalis clinical isolate

D. Renaud, D. Scott, H. Hurst, 2019, Journal of periodontal research.

Atypical cyclic di-AMP signaling is essential for Porphyromonas gingivalis growth and regulation of cell envelope homeostasis and virulence

R. Lamont, M. F. Moradali, D. Scott, 2022, npj Biofilms and Microbiomes.

The RagA and RagB proteins of Porphyromonas gingivalis.

J. Potempa, D. Scott, M. Madej, 2021, Molecular oral microbiology.

Molecular mapping of periodontal tissues using infrared microspectroscopy

Michael G. Sowa, Allan Hynes, David A. Scott, 2005, BMC Medical Imaging.

2-Amino-4-(3,4-(methylenedioxy)benzylamino)-6-(3-methoxyphenyl)pyrimidine is an anti-inflammatory TLR-2, -4 and -5 response mediator in human monocytes

R. Lamont, D. Scott, Huaxin Zhou, 2015, Inflammation Research.

Cotinine inhibits the pro-inflammatory response initiated by multiple cell surface Toll-like receptors in monocytic THP cells

J. Bagaitkar, D. Renaud, D. Scott, 2012, Tobacco induced diseases.

Tobacco Induced Diseases BioMed Central

J. Bagaitkar, D. Scott, D. Demuth, 2008 .

Molecular Mechanisms of Tobacco-induced Diseases

D. Scott, Xingwen Wang, 2006 .

Inhibition of GSK3 Abolishes Bacterial-Induced Periodontal Bone Loss in Mice

R. Jotwani, J. Potempa, D. Scott, 2012, Molecular medicine.

GSK3β and the control of infectious bacterial diseases.

Akhilesh Kumar, R. Lamont, D. Scott, 2014, Trends in microbiology.

Cotinine-induced convergence of the cholinergic and PI3 kinase-dependent anti-inflammatory pathways in innate immune cells.

D. Renaud, D. Scott, Michael Martin, 2008, Biochimica et biophysica acta.