A. Sloetjes

发表

S100A8/A9 increases the mobilization of pro-inflammatory Ly6Chigh monocytes to the synovium during experimental osteoarthritis

T. Vogl, J. Roth, S. V. van Dalen, 2017, Arthritis Research & Therapy.

Role of Activatory FcγRI and FcγRIII and Inhibitory FcγRII in Inflammation and Cartilage Destruction during Experimental Antigen-Induced Arthritis

S. Verbeek, A. Blom, A. Sloetjes, 2001 .

Toll-like receptor 4 induced FcγR expression potentiates early onset of joint inflammation and cartilage destruction during immune complex arthritis: Toll-like receptor 4 largely regulates FcγR expression by interleukin 10

A. Blom, P. V. van Lent, A. Sloetjes, 2006, Annals of the rheumatic diseases.

High LDL levels lessen bone destruction during antigen-induced arthritis by inhibiting osteoclast formation and function.

I. Bot, A. Blom, M. Koenders, 2019, Bone.

Induction of Canonical Wnt Signaling by Synovial Overexpression of Selected Wnts Leads to Protease Activity and Early Osteoarthritis-Like Cartilage Damage.

W. B. van den Berg, A. Blom, M. Koenders, 2015, The American journal of pathology.

01.01 S100A8/a9 increases the mobilisation of LY6C high monocytes to the synovium during experimental osteoarthritis

A. Blom, N. Cremers, A. Sloetjes, 2017 .

S100A8 causes a shift toward expression of activatory Fcγ receptors on macrophages via toll-like receptor 4 and regulates Fcγ receptor expression in synovium during chronic experimental arthritis.

W. B. van den Berg, T. Vogl, J. Roth, 2010, Arthritis and rheumatism.

The inhibitory receptor FcgammaRII reduces joint inflammation and destruction in experimental immune complex-mediated arthritides not only by inhibition of FcgammaRI/III but also by efficient clearance and endocytosis of immune complexes.

K. Nabbe, S. Verbeek, W. B. van den Berg, 2003, The American journal of pathology.

Fcγ receptor-mediated influx of S100A8/A9-producing neutrophils as inducer of bone erosion during antigen-induced arthritis

T. Vogl, J. Roth, A. Blom, 2018, Arthritis Research & Therapy.

Scavenger receptor class A type I/II determines matrix metalloproteinase-mediated cartilage destruction and chondrocyte death in antigen-induced arthritis.

W. B. van den Berg, T. Vogl, L. V. Tits, 2009, Arthritis and rheumatism.

W. Nacken, T. Vogl, J. Roth, 2007, Annals of the rheumatic diseases.

S100A8 enhances osteoclastic bone resorption in vitro through activation of Toll-like receptor 4: implications for bone destruction in murine antigen-induced arthritis.

W. B. van den Berg, T. Vogl, J. Roth, 2011, Arthritis and rheumatism.

Stimulation of chondrocyte-mediated cartilage destruction by S100A8 in experimental murine arthritis.

W. Nacken, H. Freeze, T. Vogl, 2008, Arthritis and rheumatism.

Fcgamma receptors directly mediate cartilage, but not bone, destruction in murine antigen-induced arthritis: uncoupling of cartilage damage from bone erosion and joint inflammation.

K. Nabbe, S. Verbeek, W. B. van den Berg, 2006, Arthritis and rheumatism.

NADPH-oxidase-driven oxygen radical production determines chondrocyte death and partly regulates metalloproteinase-mediated cartilage matrix degradation during interferon-γ-stimulated immune complex arthritis

K. Nabbe, S. Holland, J. Kolls, 2005, Arthritis research & therapy.

Expression and localisation of the new metalloproteinase inhibitor RECK (reversion inducing cysteine-rich protein with Kazal motifs) in inflamed synovial membranes of patients with rheumatoid arthritis

C. Sweep, P. Span, J. Heuvel, 2004, Annals of the rheumatic diseases.

Selectively induced death of macrophages in the synovial lining of murine knee joints using 10B-liposomes and boron neutron capture synovectomy

G. Koning, G. Krijger, W. B. van den Berg, 2009, International journal of radiation biology.

S100A8/A9, a potent serum and molecular imaging biomarker for synovial inflammation and joint destruction in seronegative experimental arthritis

D. Foell, M. Schäfers, T. Vogl, 2016, Arthritis Research & Therapy.

Alarmins S100A8/S100A9 aggravate osteophyte formation in experimental osteoarthritis and predict osteophyte progression in early human symptomatic osteoarthritis

F. Lafeber, T. Vogl, J. Roth, 2014, Annals of the rheumatic diseases.

Active involvement of alarmins S100A8 and S100A9 in regulation of synovial activation and joint destruction during osteoarthrit

W. Nacken, T. Vogl, J. Roth, 2010 .

Intensive cholesterol-lowering treatment reduces synovial inflammation during early collagenase-induced osteoarthritis, but not pathology at end-stage disease in female dyslipidemic E3L.CETP mice.

T. Vogl, E. Pieterman, B. Walgreen, 2023, Osteoarthritis and cartilage.

Different amplifying mechanisms of interleukin-17 and interferon-gamma in Fcgamma receptor-mediated cartilage destruction in murine immune complex-mediated arthritis.

W. B. van den Berg, F. Nimmerjahn, M. Koenders, 2009, Arthritis and rheumatism.

The Inhibitory Receptor FcγRII Reduces Joint Inflammation and Destruction in Experimental Immune Complex-Mediated Arthritides Not Only by Inhibition of FcγRI/III but Also by Efficient Clearance and Endocytosis of Immune Complexes

S. Verbeek, J. Roth, A. Blom, 2003 .

S100A8/A9 increases the mobilization of Ly6C high monocytes to the synovium during experimental osteoarthritis

A. Blom, N. Cremers, A. Sloetjes, 2017 .

Interleukin-1 is not involved in synovial inflammation and cartilage destruction in collagenase-induced osteoarthritis.

W. B. van den Berg, T. Vogl, J. Roth, 2017, Osteoarthritis and cartilage.

Selectively induced death of macrophages in the synovial lining of murine knee joints using 10B-liposomes and boron neutron capture synovectomy

G. Koning, G. Krijger, W. B. van den Berg, 2009, International journal of radiation biology.

Increased expression of CCL18, CCL19, and CCL17 by dendritic cells from patients with rheumatoid arthritis, and regulation by Fc gamma receptors

C. Figdor, M. C. de Waal Malefijt, M. Looman, 2004, Annals of the rheumatic diseases.

Increased FcγRII expression and aberrant tumour necrosis factor α production by mature dendritic cells from patients with active rheumatoid arthritis

C. Figdor, G. Adema, P. Barrera, 2004, Annals of the rheumatic diseases.

K. Nabbe, S. Verbeek, A. Blom, 2003 .