F. Harris

发表

Neoantigenic Potential of Complex Chromosomal Rearrangements in Mesothelioma

Sarah H. Johnson, J. Cheville, A. Mansfield, 2019, Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer.

Targeting HER2 in patient‐derived xenograft ovarian cancer models sensitizes tumors to chemotherapy

I. V. Kovtun, G. Vasmatzis, S. Weroha, 2018, Molecular oncology.

Shared and unique genomic structural variants of different histological components within testicular germ cell tumours identified with mate pair sequencing

Sarah H. Johnson, J. Cheville, B. Leibovich, 2019, Scientific Reports.

Genomic rearrangements in sporadic lymphangioleiomyomatosis: an evolving genetic story

Sarah H. Johnson, Jesse S. Voss, J. Smadbeck, 2017, Modern Pathology.

Mate-pair sequencing identifies a cryptic BMPR2 mutation in hereditary pulmonary arterial hypertension

J. Smadbeck, G. Vasmatzis, R. Frantz, 2020, Pulmonary circulation.

SVAtools for junction detection of genome-wide chromosomal rearrangements by mate-pair sequencing (MPseq).

Travis M. Drucker, Sarah H. Johnson, Roman M. Ženka, 2018, Cancer genetics.

Renin Production by Juxtaglomerular Cell Tumors and Clear Cell Renal Cell Carcinoma and the Role of Angiotensin Signaling Inhibitors

J. Cheville, V. Reuter, S. Boorjian, 2022, Mayo Clinic proceedings.

Genomic rearrangements define lineage relationships between adjacent lepidic and invasive components in lung adenocarcinoma.

Sarah H. Johnson, D. Wigle, Ping Yang, 2014, Cancer research.

Large Chromosomal Rearrangements Yield Biomarkers to Distinguish Low‐Risk From Intermediate‐ and High‐Risk Prostate Cancer

Sarah H. Johnson, I. V. Kovtun, T. Therneau, 2019, Mayo Clinic proceedings.

Integrated analysis of the genomic instability of PTEN in clinically insignificant and significant prostate cancer

Sarah H. Johnson, J. Cheville, R. Karnes, 2016, Modern Pathology.

Optimizing clinical cytology touch preparations for next generation sequencing.

Sarah H. Johnson, A. Mansfield, D. Wigle, 2020, Genomics.

Integrated Genomic Analysis of Pancreatic Ductal Adenocarcinomas Reveals Genomic Rearrangement Events as Significant Drivers of Disease.

Travis M. Drucker, Fergus J Couch, Steven N Hart, 2016, Cancer research.

Using genomics to differentiate multiple primaries from metastatic lung cancer.

Sarah H. Johnson, A. Mansfield, D. Wigle, 2019, Journal of thoracic oncology : official publication of the International Association for the Study of Lung Cancer.

Chromosomal Junction Detection from Whole Genome Sequencing on Formalin-fixed, Paraffin-embedded Tumors.

Sarah H. Johnson, Jesse S. Voss, J. Cheville, 2020, The Journal of molecular diagnostics : JMD.

Suppression of Somatic Expansion Delays the Onset of Pathophysiology in a Mouse Model of Huntington’s Disease

Amy Holt, C. McMurray, Do Yup Lee, 2015, PLoS genetics.

Quantification of Somatic Chromosomal Rearrangements in Circulating Cell-Free DNA from Ovarian Cancers

Sarah H. Johnson, I. V. Kovtun, Minetta C. Liu, 2016, Scientific Reports.

Chromoplectic TPM3–ALK rearrangement in a patient with inflammatory myofibroblastic tumor who responded to ceritinib after progression on crizotinib

A. Mansfield, R. Marks, D. Wigle, 2016, Annals of oncology : official journal of the European Society for Medical Oncology.

Integration of Comprehensive Genomic Analysis and Functional Screening of Affected Molecular Pathways to Inform Cancer Therapy

Sarah H. Johnson, J. Cheville, Minetta C. Liu, 2019, Mayo Clinic proceedings.

Theragnostic chromosomal rearrangements in treatment‐naive pancreatic ductal adenocarcinomas obtained via endoscopic ultrasound

Sarah H. Johnson, J. Cheville, J. Smadbeck, 2021, Journal of cellular and molecular medicine.

Identification of independent primary tumors and intrapulmonary metastases using DNA rearrangements in non-small-cell lung cancer.

Travis M. Drucker, Sarah H. Johnson, D. Wigle, 2014, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

Chromosomal rearrangements and copy number abnormalities of TP63 correlate with p63 protein expression in lung adenocarcinoma

Sarah H. Johnson, A. Feldman, M. Aubry, 2015, Modern Pathology.

Epigenomics may begin to explain in vitro differential response to hypomethylating agents in MMR-D hypermethylated endometrial cancer.

Sarah H. Johnson, J. Cheville, A. Mansfield, 2023, Epigenomics.

9. Detection of fusion genes from complex rearrangements reported by genome-wide mate-pair sequencing (MPseq)

Sarah H. Johnson, A. Mansfield, J. Smadbeck, 2018, Cancer Genetics.