S. Ceryak

发表

Effects of hexavalent chromium on the survival and cell cycle distribution of DNA repair-deficient S. cerevisiae.

S. Patierno, T. O'brien, J. Fornsaglio, 2002, DNA repair.

Selective inhibition of CYP27A1 and of chenodeoxycholic acid synthesis in cholestatic hamster liver.

A. Honda, S. Yoshida, N. Tanaka, 2002, Biochimica et biophysica acta.

Enhanced clonogenic survival induced by protein tyrosine phosphatase (PTP) inhibition after Cr(VI) exposure is mediated by c-Raf and Ras activity.

S. Ceryak, Dongsoon Bae, T. Camilli, 2009, Cellular signalling.

Resistance to apoptosis, increased growth potential, and altered gene expression in cells that survived genotoxic hexavalent chromium [Cr(VI)] exposure

D. Stephan, D. E. Pritchard, S. Patierno, 2005, Molecular and Cellular Biochemistry.

pH‐dependent uptake of irinotecan and its active metabolite, SN‐38, by intestinal cells

S. Kudoh, Y. Matsuzaki, Kunihiko Kobayashi, 1999, International journal of cancer.

Raf-independent, PP2A-dependent MEK activation in response to ERK silencing.

S. Ceryak, Dongsoon Bae, 2009, Biochemical and biophysical research communications.

FANCD2 monoubiquitination and activation by hexavalent chromium [Cr(VI)] exposure: activation is not required for repair of Cr(VI)-induced DSBs.

S. Patierno, T. O'brien, S. Ceryak, 2006, Mutation research.

Induction of pro-apoptotic and cell cycle-inhibiting genes in chromium (VI)-treated human lung fibroblasts: Lack of effect of ERK

S. Patierno, T. O'brien, S. Ceryak, 2004, Molecular and Cellular Biochemistry.

Intestinal alkalization as a possible preventive mechanism in irinotecan (CPT-11)-induced diarrhea.

Y. Matsuzaki, Kunihiko Kobayashi, P. Latham, 2002, Cancer research.

Chromium (VI) Activates Ataxia Telangiectasia Mutated (ATM) Protein

S. Patierno, S. Ceryak, L. Ha, 2003, The Journal of Biological Chemistry.

Exposure to particulate hexavalent chromium exacerbates allergic asthma pathology.

S. Patierno, S. Constant, S. Ceryak, 2012, Toxicology and applied pharmacology.

Lung injury, inflammation and Akt signaling following inhalation of particulate hexavalent chromium.

Kent D. Sugden, L. G. Little, S. Patierno, 2009, Toxicology and applied pharmacology.

Comparative binding of bile acids to serum lipoproteins and albumin.

B. Bouscarel, S. Ceryak, H. Fromm, 1993, Journal of lipid research.

Effect of ursodeoxycholic acid on hepatic LDL binding and uptake in dietary hypercholesterolemic hamsters.

S. Robins, B. Bouscarel, S. Ceryak, 2000, Atherosclerosis.

Comparative effect of ursodeoxycholic acid and calcium antagonists on the binding, uptake and degradation of LDL in isolated hamster hepatocytes.

B. Bouscarel, S. Ceryak, H. Fromm, 1996, Biochimica et biophysica acta.

Modulation of bile secretion by hepatic low-density lipoprotein uptake and by chenodeoxycholic acid and ursodeoxycholic acid treatment in the hamster.

S. Ceryak, H. Fromm, M. Malavolti, 1987, Gastroenterology.

Alteration of cAMP-mediated hormonal responsiveness by bile acids in cells of nonhepatic origin.

F. Noonan, T. Gettys, B. Bouscarel, 1995, The American journal of physiology.

Modulation of low density lipoprotein receptor activity by bile acids: differential effects of chenodeoxycholic and ursodeoxycholic acids in the hamster.

S. Ceryak, I. Roberts, H. Fromm, 1987, Journal of lipid research.

Excision repair is required for genotoxin-induced mutagenesis in mammalian cells.

S. Patierno, T. O'brien, J. Wise, 2008, Carcinogenesis.

Interaction of potentially toxic bile acids with human plasma proteins: Binding of lithocholic (3α-hydroxy-5β-cholan-24-oic) acid to lipoproteins and albumin

S. Ceryak, H. Fromm, M. Malavolti, 1989, Lipids.

Mechanisms of chromium-induced suppression of RNA synthesis in cellular and cell-free systems: Relationship to RNA polymerase arrest

S. Patierno, T. O'brien, F. Manning, 2004, Molecular and Cellular Biochemistry.

Generation of S phase-dependent DNA double-strand breaks by Cr(VI) exposure: involvement of ATM in Cr(VI) induction of gamma-H2AX.

S. Patierno, S. Ceryak, L. Ha, 2004, Carcinogenesis.

Ursodeoxycholic acid increases low-density lipoprotein binding, uptake and degradation in isolated hamster hepatocytes.

B. Bouscarel, S. Ceryak, H. Fromm, 1991, The Biochemical journal.

Effects of bile acid depletion and of ursodeoxycholic and chenodeoxycholic acids on biliary protein secretion in the hamster.

S. Ceryak, H. Fromm, M. Malavolti, 1990, Life sciences.

Dual mode of glucagon receptor internalization: role of PKCα, GRKs and β-arrestins.

N. Lee, T. Miyazaki, Lada Krilov, 2011, Experimental cell research.

Comparative formation of lithocholic acid from chenodeoxycholic and ursodeoxycholic acids in the colon.

F. Bazzoli, S. Ceryak, H. Fromm, 1982, Gastroenterology.

Extrahepatic deposition and cytotoxicity of lithocholic acid: Studies in two hamster models of hepatic failure and in cultured human fibroblasts

B. Bouscarel, S. Ceryak, H. Fromm, 1998, Hepatology.

Cerebral low-density lipoprotein (LDL) uptake is stimulated by acute bile drainage.

S. Ceryak, H. Fromm, M. Malavolti, 1991, Biochimica et biophysica acta.

Chromium genotoxicity: A double-edged sword.

Steven R Patierno, S. Patierno, S. Ceryak, 2010, Chemico-biological interactions.

Complexities of chromium carcinogenesis: role of cellular response, repair and recovery mechanisms.

S. Patierno, T. O'brien, S. Ceryak, 2003, Mutation research.

Enhancement of DNA topoisomerase I inhibitor–induced apoptosis by ursodeoxycholic acid

Y. Matsuzaki, B. Bouscarel, S. Ceryak, 2006, Molecular Cancer Therapeutics.

Acquisition of mitochondrial dysregulation and resistance to mitochondrial-mediated apoptosis after genotoxic insult in normal human fibroblasts: a possible model for early stage carcinogenesis.

G. Gerard, E. Kaldjian, S. Patierno, 2012, Biochimica et biophysica acta.

Mechanism of apoptosis and determination of cellular fate in chromium(VI)-exposed populations of telomerase-immortalized human fibroblasts.

S. K. Hartman, D. E. Pritchard, S. Patierno, 2001, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.

AKT1 mediates bypass of the G1/S checkpoint after genotoxic stress in normal human cells

S. Patierno, S. Ceryak, Madhu Lal, 2009, Cell cycle.

Lung Inflammation, Injury, and Proliferative Response after Repetitive Particulate Hexavalent Chromium Exposure

S. Patierno, S. Constant, L. Beaver, 2009, Environmental health perspectives.

Fanconi anemia complementation group A cells are hypersensitive to chromium(VI)-induced toxicity.

D. E. Pritchard, S. Patierno, T. O'brien, 2002, Environmental health perspectives.

Absence of significant role of bile acids in diarrhea of a heterogeneous group of postcholecystectomy patients

S. Ceryak, H. Fromm, M. Malavolti, 2005, Digestive Diseases and Sciences.

A bioenergetic profile of non-transformed fibroblasts uncovers a link between death-resistance and enhanced spare respiratory capacity.

Steven R Patierno, O. Shirihai, S. Patierno, 2013, Mitochondrion.

Polo-like kinase 1 enhances survival and mutagenesis after genotoxic stress in normal cells through cell cycle checkpoint bypass.

S. Patierno, T. O'brien, S. Ceryak, 2010, Carcinogenesis.

Does metabolic radiolabeling stimulate the stress response? Gene expression profiling reveals differential cellular responses to internal beta vs. external gamma radiation

Nicholas F. Marko, T. McCaffrey, R. Howell, 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Bicarbonate Attenuates Irinotecan-Induced Cytotoxicity through Regulation of Both Extracellular and Intracellular pHs in Intestine Cell Line

T. Miyazaki, Y. Matsuzaki, Kunihiko Kobayashi, 2013 .

Feasibility of an endoscopic secretin test: preliminary results.

W. Steinberg, S. Ceryak, A. Ruiz, 2001, Pancreas.

Gallstone dissolution treatment with a combination of chenodeoxycholic and ursodeoxycholic acids

S. Ceryak, H. Fromm, M. Malavolti, 1986, Digestive Diseases and Sciences.

Protein tyrosine phosphatase (PTP) inhibition enhances chromosomal stability after genotoxic stress: decreased chromosomal instability (CIN) at the expense of enhanced genomic instability (GIN)?

S. Patierno, J. Wise, S. Wise, 2012, Mutation research.

Bypass of hexavalent chromium-induced growth arrest by a protein tyrosine phosphatase inhibitor: enhanced survival and mutagenesis.

S. Patierno, T. O'brien, S. Ceryak, 2009, Mutation research.

Bile acids stimulate PKCalpha autophosphorylation and activation: role in the attenuation of prostaglandin E1-induced cAMP production in human dermal fibroblasts.

Lada Krilov, B. Bouscarel, S. Ceryak, 2006, American journal of physiology. Gastrointestinal and liver physiology.

Biphasic regulation by bile acids of dermal fibroblast proliferation through regulation of cAMP production and COX-2 expression level.

B. Bouscarel, S. Ceryak, L. Epstein, 2006, American journal of physiology. Cell physiology.

Formation, absorption, and biotransformation of delta 6-lithocholenic acid in humans.

K. Setchell, S. Ceryak, H. Fromm, 1993, American Journal of Physiology.

Effect of cholestasis on regulation of cAMP synthesis by glucagon and bile acids in isolated hepatocytes.

Y. Matsuzaki, T. Gettys, B. Bouscarel, 1997, American Journal of Physiology.

Urocanic Acid Does not Photobind to DNA in Mice Irradiated with Immunosuppressive Doses of UVB *

G. B. V. Beijersbergen van Henegouwen, F. Noonan, D. Hug, 1998, Photochemistry and photobiology.

Regulation of Stimulated Cyclic AMP Synthesis by Urocanic Acid

F. Noonan, T. Gettys, B. Bouscarel, 1998, Photochemistry and photobiology.

Acquisition of mitochondrial dysregulation and resistance to mitochondrial-mediated apoptosis after genotoxic insult in normal human fibroblasts: a possible model for early stage carcinogenesis.

G. Gerard, E. Kaldjian, S. Patierno, 2012, Biochimica et biophysica acta.

Raf-independent, PP2A-dependent MEK activation in response to ERK silencing.

S. Ceryak, Dongsoon Bae, 2009, Biochemical and biophysical research communications.

Polo-like kinase 1 enhances survival and mutagenesis after genotoxic stress in normal cells through cell cycle checkpoint bypass.

S. Patierno, T. O'brien, S. Ceryak, 2010, Carcinogenesis.