P. North

发表

Repair and misrepair of site-specific DNA double-strand breaks by human cell extracts.

J. Thacker, A. Ganesh, P. North, 1993, Mutation research.

Phosphorylation of BLM, Dissociation from Topoisomerase IIIα, and Colocalization with γ-H2AX after Topoisomerase I-Induced Replication Damage

Y. Pommier, I. Hickson, Linghua Meng, 2005, Molecular and Cellular Biology.

Role of the Bloom's syndrome helicase in maintenance of genome stability.

I. Hickson, P. North, S. Davies, 2001, Biochemical Society transactions.

Phosphorylation of BLM, Dissociation from Topoisomerase III (cid:2) , and Colocalization with (cid:3) -H2AX after Topoisomerase I-Induced Replication Damage

Y. Pommier, I. Hickson, Linghua Meng, 2005 .

Evidence for the involvement of DNA topoisomerase II in neutrophil-granulocyte differentiation.

P. North, G. Francis, P. Jacobs, 1987, Leukemia.

Mechanisms of Resistance to Ionising Radiations: Genetic and Molecular Studies on Ataxia-Telangiectasia and Related Radiation-Sensitive Mutants

J. Thacker, A. Ganesh, P. North, 1989 .

The Bloom's Syndrome Gene Product Interacts with Topoisomerase III*

K. Gatter, I. Hickson, H. Turley, 2000, The Journal of Biological Chemistry.

Role for BLM in replication-fork restart and suppression of origin firing after replicative stress

Ian D Hickson, I. Hickson, P. North, 2007, Nature Structural &Molecular Biology.

BLM is required for faithful chromosome segregation and its localization defines a class of ultrafine anaphase bridges

Kok-Lung Chan, Ian D Hickson, I. Hickson, 2007, The EMBO journal.

Cyclic AMP-dependent protein kinase type I is involved in hypersensitivity of human breast cells to topoisomerase II inhibitors.

A. Harris, G. Tortora, C. Bianco, 1995, Clinical cancer research : an official journal of the American Association for Cancer Research.

Increased error-prone non homologous DNA end-joining – a proposed mechanism of chromosomal instability in Bloom's syndrome

G. Mufti, I. Hickson, T. Gaymes, 2002, Oncogene.

RUNX1 Is a Key Target in t(4;11) Leukemias that Contributes to Gene Activation through an AF4-MLL Complex Interaction

Thomas A. Milne, Adam C. Wilkinson, A. Wilkinson, 2013, Cell reports.

Topo IIIα and BLM Act within the Fanconi Anemia Pathway in Response to DNA-Crosslinking Agents

R. Moses, I. Hickson, S. Olson, 2009, Cytogenetic and Genome Research.

MLL-Rearranged Acute Lymphoblastic Leukemias Activate BCL-2 through H3K79 Methylation and Are Sensitive to the BCL-2-Specific Antagonist ABT-199

K. Coombes, A. Letai, Nianxiang Zhang, 2015, Cell reports.

A mechanism for deletion formation in DNA by human cell extracts: the involvement of short sequence repeats.

J. Thacker, A. Ganesh, P. North, 1992, Nucleic acids research.

The rejoining of double-strand breaks in DNA by human cell extracts.

J. Thacker, A. Ganesh, P. North, 1990, Nucleic acids research.

Phosphorylation of the Bloom's Syndrome Helicase and Its Role in Recovery from S-Phase Arrest

I. Hickson, N. Lakin, P. North, 2004, Molecular and Cellular Biology.

Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome

S. Cantor, I. Hickson, Yuliang Wu, 2011, The EMBO journal.

Phosphorylation of BLM, dissociation from topoisomerase IIIalpha, and colocalization with gamma-H2AX after topoisomerase I-induced replication damage.

Y. Pommier, I. Hickson, Linghua Meng, 2005, Molecular and cellular biology.

Interaction between the helicases genetically linked to Fanconi anemia group J and Bloom's syndrome

I. Hickson, Yuliang Wu, P. North, 2011, The EMBO journal.

Overexpression of the RIα subunit of Protein Kinase A confers hypersensitivity to topoisomerase II inhibitors and 8-Chloro-cyclic Adenosine 3′,5′-Monophosphate in chinese hamster ovary cells.

A. Harris, G. Tortora, C. Bianco, 1994 .