B. Kysela

发表

Identification of a defect in DNA ligase IV in a radiosensitive leukaemia patient

P. Jeggo, S. Jackson, A. Doherty, 1999, Current Biology.

Identification of a DNA Nonhomologous End-Joining Complex in Bacteria

P. Jeggo, K. Devine, S. K. Devine, 2002, Science.

DNA ligase IV mutations identified in patients exhibiting developmental delay and immunodeficiency.

P. Jeggo, A. Gennery, K. Cerosaletti, 2001, Molecular cell.

Potential Role for 53BP1 in DNA End-joining Repair through Direct Interaction with DNA*

P. Jeggo, T. Date, Kuniyoshi Iwabuchi, 2003, Journal of Biological Chemistry.

Relative contributions of levels of initial DNA damage and repair of double strand breaks to the ionizing radiation-sensitive phenotype of the Chinese hamster cell mutant, XR-V15B. Part I. X-rays.

J. Arrand, B. Michael, B. Kysela, 1993, International journal of radiation biology.

Relative contributions of levels of initial damage and repair of double-strand breaks to the ionizing radiation-sensitive phenotype of the Chinese hamster cell mutant, XR-V15B. Part II. Neutrons.

J. Arrand, B. Michael, B. Kysela, 1993, International journal of radiation biology.

Ku Stimulation of DNA Ligase IV-dependent Ligation Requires Inward Movement along the DNA Molecule*

P. Jeggo, B. Vojnovic, S. Ameer-Beg, 2003, Journal of Biological Chemistry.

Analysis of DNA ligase IV mutations found in LIG4 syndrome patients: the impact of two linked polymorphisms.

P. Jeggo, A. Doherty, B. Kysela, 2004, Human molecular genetics.

Defects in the kinetics of the repair of DNA double-strand breaks and inhibition of DNA synthesis in the ataxia telangiectasia AT5BI-VA cell line: comparison to a corrected hybrid, atxbc.

H. Lohrer, J. Arrand, B. Kysela, 1995, Radiation research.

Field-inversion gel electrophoresis analysis of the induction and rejoining of DNA double-strand breaks in cells embedded in agarose.

J. Arrand, B. Michael, B. Kysela, 1993, Radiation research.

Ku70 N-terminal lysines acetylation/deacetylation is required for radiation-induced DNA-double strand breaks repair.

B. Kysela, M. Jeeves, A. Al Emam, 2018, Neoplasma.

Attenuating the DNA damage response to double-strand breaks restores function in models of CNS neurodegeneration

C. Kyriacou, Z. Ahmed, R. Tuxworth, 2019, Brain communications.

Letter: Attenuating the DNA damage response to double strand breaks is neuroprotective

C. Kyriacou, Z. Ahmed, R. Tuxworth, 2018, bioRxiv.

Issues surrounding standard cytotoxicity testing for assessing activity of non-covalent DNA-binding metallo-drugs.

B. Kysela, M. J. Hannon, A. J. Pope, 2010, Dalton transactions.

Flow cytometry analysis of ploidy and proliferation activity in classical and spermatocytic seminoma.

B. Kysela, J. Matoška, 1991, Neoplasma.

Cellular and Biochemical Impact of a Mutation in DNA Ligase IV Conferring Clinical Radiosensitivity*

P. Jeggo, A. Doherty, M. Oettinger, 2001, The Journal of Biological Chemistry.

Mutations in the NHEJ component XRCC4 cause primordial dwarfism.

Edward S. Miller, T. Blundell, M. van der Burg, 2015, American journal of human genetics.

Evolutionary and Functional Conservation of the DNA Non-homologous End-joining Protein, XLF/Cernunnos*

S. Jackson, T. E. Wilson, Julie Bianchi, 2006, Journal of Biological Chemistry.

Association of the thyroid stimulating hormone receptor gene (TSHR) with Graves' disease.

J. Barrett, J. Connell, J. Franklyn, 2009, Human molecular genetics.

Phosphorylation of linker histones by DNA-dependent protein kinase is required for DNA ligase IV-dependent ligation in the presence of histone H1.

P. Jeggo, M. Chovanec, B. Kysela, 2005, Proceedings of the National Academy of Sciences of the United States of America.

The C-terminal conserved domain of DNA-PKcs, missing in the SCID mouse, is required for kinase activity.

P. Jeggo, R. Jessberger, H. Beamish, 2000, Nucleic acids research.

Impact of DNA ligase IV on the fidelity of end joining in human cells.

M. Lieber, P. Jeggo, D. Papadopoulo, 2003, Nucleic acids research.

Nonhomologous end joining and V(D)J recombination require an additional factor

S. West, P. Jeggo, M. Oettinger, 2003, Proceedings of the National Academy of Sciences of the United States of America.

AHNAK interacts with the DNA ligase IV-XRCC4 complex and stimulates DNA ligase IV-mediated double-stranded ligation.

P. Jeggo, E. Shtivelman, B. Kysela, 2004, DNA repair.

Intracellular synchrotron nanoimaging and DNA damage/genotoxicity screening of novel lanthanide-coated nanovectors.

D. J. Lewis, P. Cloetens, S. Bohic, 2010, Nanomedicine.

Evolutionary and Functional Conservation of the DNA Non-homologous End-joining Protein, XLF/Cernunnos*

T. E. Wilson, Julie Bianchi, A. Doherty, 2006, Journal of Biological Chemistry.

Mutations in the NHEJ component XRCC4 cause primordial dwarfism.

Edward S. Miller, T. Blundell, M. van der Burg, 2015, American journal of human genetics.