M. Pett

发表

The two most common histological subtypes of malignant germ cell tumour are distinguished by global microRNA profiles, associated with differential transcription factor expression

Anton J. Enright, A. Enright, S. van Dongen, 2010, Molecular Cancer.

Global microRNA profiles in cervical squamous cell carcinoma depend on Drosha expression levels

Leonard D. Goldstein, N L Barbosa-Morais, N. Barbosa-Morais, 2007, The Journal of pathology.

Changes in cervical keratinocyte gene expression associated with integration of human papillomavirus 16.

Tom Freeman, Nicholas Coleman, M. Stanley, 2002, Cancer research.

In vitro progression of human papillomavirus 16 episome-associated cervical neoplasia displays fundamental similarities to integrant-associated carcinogenesis.

M. Stanley, N. Coleman, I. Roberts, 2010, Cancer research.

Critical evaluation of HPV16 gene copy number quantification by SYBR green PCR

A. Teschendorff, M. Stanley, N. Coleman, 2008, BMC biotechnology.

An increase in DNA double‐strand breaks, induced by Ku70 depletion, is associated with human papillomavirus 16 episome loss and de novo viral integration events

N. Coleman, M. Pett, N. Foster, 2007, The Journal of pathology.

Interferon-beta treatment of cervical keratinocytes naturally infected with human papillomavirus 16 episomes promotes rapid reduction in episome numbers and emergence of latent integrants.

A. Teschendorff, M. Stanley, N. Coleman, 2006, Carcinogenesis.

Lack of correlation between predicted and actual off-target effects of short-interfering RNAs targeting the human papillomavirus type 16 E7 oncogene

Anton J. Enright, S. Dongen, A. Enright, 2013, British Journal of Cancer.

Genomic imbalances in 70 snap-frozen cervical squamous intraepithelial lesions: associations with lesion grade, state of the HPV16 E2 gene and clinical outcome

M. Stanley, R. Moseley, N. Coleman, 2004, British Journal of Cancer.

Virus transcript levels and cell growth rates after naturally occurring HPV16 integration events in basal cervical keratinocytes

N. Coleman, I. Groves, M. Pett, 2014, The Journal of pathology.

Integration of high‐risk human papillomavirus: a key event in cervical carcinogenesis?

N. Coleman, M. Pett, M Pett, 2007, The Journal of pathology.

Selection of cervical keratinocytes containing integrated HPV16 associates with episome loss and an endogenous antiviral response.

M. Stanley, G. Yeo, M. Shivji, 2006, Proceedings of the National Academy of Sciences of the United States of America.

Acquisition of High-Level Chromosomal Instability Is Associated with Integration of Human Papillomavirus Type 16 in Cervical Keratinocytes

David I. Smith, M. Stanley, N. Coleman, 2004, Cancer Research.

Malignant germ cell tumors display common microRNA profiles resulting in global changes in expression of messenger RNA targets.

Anton J. Enright, A. Enright, S. van Dongen, 2010, Cancer research.

Early genetic events in HPV immortalised keratinocytes

M. Stanley, N. Coleman, I. Roberts, 2001, Genes, chromosomes & cancer.

LIN28 Expression in malignant germ cell tumors downregulates let-7 and increases oncogene levels.

Anton J. Enright, A. Enright, S. van Dongen, 2013, Cancer research.

Tumor and Stem Cell Biology LIN 28 Expression in Malignant Germ Cell Tumors Downregulates let-7 and Increases Oncogene Levels

Anton J. Enright, S. Dongen, A. Enright, 2013 .

Overexpression of the oncostatin M receptor in cervical squamous cell carcinoma cells is associated with a pro‐angiogenic phenotype and increased cell motility and invasiveness

N. Coleman, W. English, I. Roberts, 2011, The Journal of pathology.

Papillomavirus-Mediated Neoplastic Progression Is Associated with Reciprocal Changes in Jagged1 and Manic Fringe Expression Linked to Notch Activation

M. Stanley, R. Kumar, P. Nair, 2004, Journal of Virology.

Characterization of naturally occurring HPV16 integration sites isolated from cervical keratinocytes under noncompetitive conditions.

M. Stanley, N. Coleman, I. Roberts, 2008, Cancer research.

Functional evidence that Drosha overexpression in cervical squamous cell carcinoma affects cell phenotype and microRNA profiles

N. Barbosa-Morais, H. Saini, N. Coleman, 2011, The Journal of pathology.

Gain and overexpression of the oncostatin M receptor occur frequently in cervical squamous cell carcinoma and are associated with adverse clinical outcome

N. Coleman, Jingxiang Huang, G. Mukherjee, 2007, The Journal of pathology.

Development of peptide aptamer microarrays for detection of HPV16 oncoproteins in cell extracts.

S. Laurenson, P. Ko Ferrigno, F. Hoppe-Seyler, 2011, Analytical biochemistry.

Amplification of chromosome 5p correlates with increased expression of Skp2 in HPV-immortalized keratinocytes

M. Stanley, P. Stern, N. Coleman, 2003, Oncogene.

HPV: from infection to cancer.

M. Stanley, N. Coleman, M. Pett, 2007, Biochemical Society transactions.

Pediatric malignant germ cell tumors show characteristic transcriptome profiles.

S. Tavaré, N. Barbosa-Morais, N. Thorne, 2008, Cancer research.

Lipoprotein lipase is frequently overexpressed or translocated in cervical squamous cell carcinoma and promotes invasiveness through the non-catalytic C terminus

N. Coleman, I. Roberts, A. Chattopadhyay, 2012, British Journal of Cancer.

Molecular Cytogenetic Analysis of Cervical Squamous Cell Carcinoma Cells Demonstrates Discordant Levels of Numerical and Structural Chromosomal Instability and Identifies ‘Selected’ Chromosome Rearrangements

N. Coleman, I. Roberts, M. Pett, 2010, Cytogenetic and Genome Research.

Depletion of HPV16 early genes induces autophagy and senescence in a cervical carcinogenesis model, regardless of viral physical state

Anton J. Enright, A. Enright, S. van Dongen, 2013, The Journal of pathology.

Depletion of polycistronic transcripts using short interfering RNAs: cDNA synthesis method affects levels of non-targeted genes determined by quantitative PCR

N. Coleman, I. Groves, M. Pett, 2013, Virology Journal.