N. Matsuda

发表

PINK1 stabilized by mitochondrial depolarization recruits Parkin to damaged mitochondria and activates latent Parkin for mitophagy

N. Hattori, Keiji Tanaka, M. Komatsu, 2010, The Journal of cell biology.

Ubiquitin is phosphorylated by PINK1 to activate parkin

T. Hirokawa, Y. Saeki, Keiji Tanaka, 2014, Nature.

RMA1, an Arabidopsis thaliana gene whose cDNA suppresses the yeast sec15 mutation, encodes a novel protein with a RING finger motif and a membrane anchor.

A. Nakano, N. Matsuda, 1998, Plant & cell physiology.

DDB2, the xeroderma pigmentosum group E gene product, is directly ubiquitylated by Cullin 4A-based ubiquitin ligase complex.

Keiji Tanaka, T. Chiba, T. Natsume, 2005, DNA repair.

Structural basis for specific cleavage of Lys6-linked polyubiquitin chains by USP30

Y. Hashimoto, Y. Saeki, Keiji Tanaka, 2017, Nature Structural & Molecular Biology.

The ubiquitin signal and autophagy: an orchestrated dance leading to mitochondrial degradation

Keiji Tanaka, Koji Yamano, N. Matsuda, 2016, EMBO reports.

Direct interactions between NEDD8 and ubiquitin E2 conjugating enzymes upregulate cullin-based E3 ligase activity

Y. Saeki, Keiji Tanaka, T. Chiba, 2007, Nature Structural &Molecular Biology.

Does impairment of the ubiquitin-proteasome system or the autophagy-lysosome pathway predispose individuals to neurodegenerative disorders such as Parkinson's disease?

Keiji Tanaka, N. Matsuda, 2010, Journal of Alzheimer's disease : JAD.

Different dynamic movements of wild‐type and pathogenic VCPs and their cofactors to damaged mitochondria in a Parkin‐mediated mitochondrial quality control system

Keiji Tanaka, Y. Kimura, A. Kakizuka, 2013, Genes to cells : devoted to molecular & cellular mechanisms.

Cleaved PGAM5 dephosphorylates nuclear serine/arginine-rich proteinsduring mitophagy.

T. Natsume, S. Iemura, K. Takeda, 2021, Biochimica et biophysica acta. Molecular cell research.

Mitochondrial hexokinase HKI is a novel substrate of the Parkin ubiquitin ligase.

Keiji Tanaka, T. Natsume, S. Iemura, 2012, Biochemical and biophysical research communications.

Parkin Mediates Apparent E2-Independent Monoubiquitination In Vitro and Contains an Intrinsic Activity That Catalyzes Polyubiquitination

K. Lim, Keiji Tanaka, C. Chai, 2011, PLoS ONE.

Discovery and Optimization of Inhibitors of the Parkinson's Disease Associated Protein DJ-1.

K. Tsumoto, Makoto Nakakido, S. Nagatoishi, 2018, ACS chemical biology.

Parkinson’s disease-related DJ-1 functions in thiol quality control against aldehyde attack in vitro

T. Mizushima, Keiji Tanaka, K. Takagi, 2017, Scientific Reports.

Phospho-ubiquitin: upending the PINK-Parkin-ubiquitin cascade.

N. Matsuda, 2016, Journal of biochemistry.

Molecular mechanisms underlying PINK1 and Parkin catalyzed ubiquitylation of substrates on damaged mitochondria.

N. Matsuda, Fumika Koyano, 2015, Biochimica et biophysica acta.

Rma1, a novel type of RING finger protein conserved from Arabidopsis to human, is a membrane-bound ubiquitin ligase.

A. Nakano, N. Matsuda, T. Suzuki, 2001, Journal of cell science.

JCB_201410050 1..18

Y. Saeki, Keiji Tanaka, H. Kosako, 2015 .

Phosphorylated ubiquitin chain is the genuine Parkin receptor

Y. Saeki, Keiji Tanaka, H. Kosako, 2015, The Journal of cell biology.

The principal PINK1 and Parkin cellular events triggered in response to dissipation of mitochondrial membrane potential occur in primary neurons

G. Sobue, Keiji Tanaka, N. Matsuda, 2013, Genes to cells : devoted to molecular & cellular mechanisms.

PINK1 autophosphorylation upon membrane potential dissipation is essential for Parkin recruitment to damaged mitochondria

N. Hattori, Keiji Tanaka, M. Komatsu, 2012, Nature Communications.

Parkin‐mediated ubiquitylation redistributes MITOL/March5 from mitochondria to peroxisomes

Keiji Tanaka, Y. Kimura, H. Kosako, 2019, EMBO reports.

Diverse Effects of Pathogenic Mutations of Parkin That Catalyze Multiple Monoubiquitylation in Vitro*

N. Hattori, Keiji Tanaka, Toshiaki Suzuki, 2006, Journal of Biological Chemistry.

p62/SQSTM1 cooperates with Parkin for perinuclear clustering of depolarized mitochondria

Nobutaka Hattori, Masaaki Komatsu, Noriyuki Matsuda, 2010, Genes to cells : devoted to molecular & cellular mechanisms.

Cell biology: Tagged tags engage disposal

Keiji Tanaka, N. Matsuda, 2015, Nature.

Parkin recruitment to impaired mitochondria for nonselective ubiquitylation is facilitated by MITOL

Keiji Tanaka, H. Kosako, Koji Yamano, 2019, The Journal of Biological Chemistry.

Cleaved PGAM5 is released from mitochondria depending on proteasome-mediated rupture of the outer mitochondrial membrane during mitophagy

K. Takeda, N. Matsuda, A. Yamaguchi, 2018, Journal of biochemistry.

An Arabidopsis Gene lsolated by a Nove1 Method for Detecting Genetic lnteraction in Yeast Encodes the GDP Dissociation lnhibitor of Ara4 GTPase

H. Tsukaya, T. Ueda, H. Uchimiya, 2002 .

An Arabidopsis gene isolated by a novel method for detecting genetic interaction in yeast encodes the GDP dissociation inhibitor of Ara4 GTPase.

A. Nakano, H. Tsukaya, T. Ueda, 1996, The Plant cell.

Overexpression of PRA2, a Rab/Ypt-family small GTPase from Pea Pisum sativum, aggravates the growth defect of yeast ypt mutants.

A. Nakano, T. Ueda, N. Matsuda, 2000, Cell structure and function.

Ubiquitination of exposed glycoproteins by SCFFBXO27 directs damaged lysosomes for autophagy

Y. Saeki, Keiji Tanaka, K. Iwai, 2017, Proceedings of the National Academy of Sciences.

Molecular mechanisms and physiological functions of mitophagy

Koji Yamano, K. Okamoto, Miyuki Sato, 2021, The EMBO journal.

MG53 nucleates assembly of cell membrane repair machinery

M. Nishi, H. Takeshima, M. Brotto, 2009, Nature Cell Biology.

A Dimeric PINK1-containing Complex on Depolarized Mitochondria Stimulates Parkin Recruitment*

Keiji Tanaka, T. Oka, N. Matsuda, 2013, The Journal of Biological Chemistry.

UV-Induced Ubiquitylation of XPC Protein Mediated by UV-DDB-Ubiquitin Ligase Complex

Keiji Tanaka, K. Sugasawa, G. Chu, 2005, Cell.

Ubiquitin Ligase Activities of Bombyx mori Nucleopolyhedrovirus RING Finger Proteins

A. Nakano, Keiji Tanaka, N. Matsuda, 2003, Journal of Virology.

Ubiquitin Ligase Activities of Bombyx mori Nucleopolyhedrovirus

A. Nakano, Keiji Tanaka, S. Nakano, 2002 .

Parkin-catalyzed Ubiquitin-Ester Transfer Is Triggered by PINK1-dependent Phosphorylation*

Norihiro Suzuki, Noriyuki Matsuda, Keiji Tanaka, 2013, The Journal of Biological Chemistry.

Structural insights into ubiquitin phosphorylation by PINK1

Keiji Tanaka, L. Negishi, S. Fukai, 2018, Scientific Reports.

EL5, a rice N-acetylchitooligosaccharide elicitor-responsive RING-H2 finger protein, is a ubiquitin ligase which functions in vitro in co-operation with an elicitor-responsive ubiquitin-conjugating enzyme, OsUBC5b.

A. Nakano, E. Minami, N. Matsuda, 2002, The Plant journal : for cell and molecular biology.

Endosomal Rab cycles regulate Parkin-mediated mitophagy

Keiji Tanaka, R. Youle, Shireen A. Sarraf, 2018, eLife.

A palmitoylated RING finger ubiquitin ligase and its homologue in the brain membranes

K. Araki, Keiji Tanaka, T. Kumanishi, 2003, Journal of neurochemistry.

Unconventional PINK 1 localization mechanism to the outer membrane of depolarized mitochondria drives

Keiji Tanaka, T. Oka, N. Matsuda, 2014 .

Proteostasis and neurodegeneration: the roles of proteasomal degradation and autophagy.

Keiji Tanaka, N. Matsuda, 2014, Biochimica et biophysica acta.

Unconventional PINK1 localization to the outer membrane of depolarized mitochondria drives Parkin recruitment

Keiji Tanaka, T. Oka, N. Matsuda, 2015, Journal of Cell Science.

Constitutive Activation of PINK1 Protein Leads to Proteasome-mediated and Non-apoptotic Cell Death Independently of Mitochondrial Autophagy*

T. Oka, N. Matsuda, K. Okatsu, 2016, The Journal of Biological Chemistry.

Loss of peptide:N-glycanase causes proteasome dysfunction mediated by a sugar-recognizing ubiquitin ligase

Keiji Tanaka, Tadashi Suzuki, K. Iwai, 2021, Proceedings of the National Academy of Sciences.

Mammalian BCAS3 and C16orf70 associate with the phagophore assembly site in response to selective and non-selective autophagy

Keiji Tanaka, K. Imai, H. Kosako, 2021, Autophagy.

Optineurin provides a mitophagy contact site for TBK1 activation

Keiji Tanaka, Koji Yamano, N. Matsuda, 2023, bioRxiv.

Unfolding is the driving force for mitochondrial import and degradation of the Parkinson's disease-related protein DJ-1

T. Hirokawa, C. Motono, K. Tsumoto, 2021, Journal of cell science.

TIM23 facilitates PINK1 activation by safeguarding against OMA1-mediated degradation in damaged mitochondria.

H. Kosako, T. Endo, T. Oka, 2023, Cell reports.

Phosphorylated ubiquitin chain is the genuine Parkin receptor

Y. Saeki, Keiji Tanaka, H. Kosako, 2015, The Journal of cell biology.

Two sides of a coin: Physiological significance and molecular mechanisms for damage-induced mitochondrial localization of PINK1 and Parkin

Koji Yamano, N. Matsuda, 2020, Neuroscience Research.

Critical role of mitochondrial ubiquitination and the OPTN–ATG9A axis in mitophagy

Keiji Tanaka, M. Naito, Koji Yamano, 2020, The Journal of cell biology.

Site-specific Interaction Mapping of Phosphorylated Ubiquitin to Uncover Parkin Activation*♦

T. Hirokawa, Y. Saeki, Keiji Tanaka, 2015, The Journal of Biological Chemistry.

Uncovering the roles of PINK1 and Parkin in mitophagy

Keiji Tanaka, N. Matsuda, 2010, Autophagy.

Elucidation of ubiquitin-conjugating enzymes that interact with RBR-type ubiquitin ligases using a liquid–liquid phase separation–based method

Keiji Tanaka, K. Imai, Koji Yamano, 2022, The Journal of biological chemistry.

Unexpected mitochondrial matrix localization of Parkinson's disease‐related DJ‐1 mutants but not wild‐type DJ‐1

Keiji Tanaka, Koji Yamano, N. Matsuda, 2016, Genes to cells : devoted to molecular & cellular mechanisms.