Kei-ichi Ishikawa

Evidence that phosphorylated ubiquitin signaling is involved in the etiology of Parkinson’s disease

H. Okano, N. Hattori, Y. Imai, 2016, Human molecular genetics.

Metabolomics‐based identification of metabolic alterations in PARK2

N. Hattori, T. Hatano, R. Mohney, 2019, Annals of clinical and translational neurology.

Identifying Therapeutic Agents for Amelioration of Mitochondrial Clearance Disorder in Neurons of Familial Parkinson Disease

N. Hattori, T. Hatano, Y. Imai, 2020, Stem cell reports.

Autophagy promotes cell survival by maintaining NAD(H) levels

Elsje G. Otten, R. Jaenisch, Y. Buganim, 2021 .

Multi-year whole-blood transcriptome data for the study of onset and progression of Parkinson’s Disease

Piero Carninci, N. Hattori, K. Hashimoto, 2019, Scientific Data.

Reduced astrocytic reactivity in human brains and midbrain organoids with PRKN mutations

G. Oyama, N. Hattori, T. Hatano, 2020, NPJ Parkinson's disease.

IPSC-derived midbrain astrocytes from Parkinson’s disease patients carrying pathogenic SNCA mutations exhibit alpha-synuclein aggregation, mitochondrial fragmentation and excess calcium release

S. Finkbeiner, R. Krüger, N. Hattori, 2020, bioRxiv.

Analyses of the MAPT, PGRN, and C9orf72 mutations in Japanese patients with FTLD, PSP, and CBS.

N. Hattori, M. Hasegawa, H. Yoshino, 2013, Parkinsonism & related disorders.