J. Neumann

发表

Isolation and characterization of the mouse cardiac myosin heavy chain genes.

J. Gulick, A. Subramaniam, J. Neumann, 1991, The Journal of biological chemistry.

Transgenic analysis of the thyroid-responsive elements in the alpha-cardiac myosin heavy chain gene promoter.

J. Robbins, J. Gulick, A. Subramaniam, 1993, The Journal of biological chemistry.

Rescue of cardiac alpha-actin-deficient mice by enteric smooth muscle gamma-actin.

S. Pawlowski, G. Boivin, T. Doetschman, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Rescue of cardiac α-actin-deficient mice by enteric smooth muscle γ-actin

S. Pawlowski, G. Boivin, T. Doetschman, 1997 .

Abnormal bone growth and selective translational regulation in basic fibroblast growth factor (FGF-2) transgenic mice.

G. Dorn, P. Howles, T. Doetschman, 1995, Molecular biology of the cell.

Identification of Maternally Regulated Fetal Gene Networks in the Placenta using a novel

P. Succop, M. Medvedovic, S. Karyala, 2011 .

Targeted Ablation of Plasma Membrane Ca2+-ATPase (PMCA) 1 and 4 Indicates a Major Housekeeping Function for PMCA1 and a Critical Role in Hyperactivated Sperm Motility and Male Fertility for PMCA4*

T. Doetschman, V. Prasad, G. Shull, 2004, Journal of Biological Chemistry.

Inhibition of type 1 protein phosphatase activity by activation of beta-adrenoceptors in ventricular myocardium.

H. Sabbah, Ramesh C. Gupta, J. Neumann, 2002, Biochemical pharmacology.

Functional roles of the alpha isoforms of the Na,K-ATPase.

J. Lingrel, A. Moseley, J. Neumann, 2003, Annals of the New York Academy of Sciences.

Functional Roles of the α Isoforms of the Na,K‐ATPase

J. Lingrel, A. Moseley, J. Neumann, 2003 .

Renal function in NHE3-deficient mice with transgenic rescue of small intestinal absorptive defect.

J. Lorenz, G. Shull, J. Neumann, 2003, American journal of physiology. Renal physiology.

The presence of both negative and positive elements in the 5'-flanking sequence of the rat Na,K-ATPase alpha 3 subunit gene are required for brain expression in transgenic mice.

J. Lingrel, B. Pathak, J. Neumann, 1994, Nucleic acids research.

The α2 Isoform of Na,K-ATPase Mediates Ouabain-induced Cardiac Inotropy in Mice*

I. Grupp, J. Lingrel, J. Lorenz, 2003, Journal of Biological Chemistry.

Mechanisms of beta-adrenergic stimulation of cardiac Ca2+ channels revealed by discrete-time Markov analysis of slow gating.

J. Neumann, S. Herzig, D. Yue, 1993, Biophysical journal.

Regulated expression of the human beta globin gene in transgenic mice requires an upstream globin or nonglobin promoter.

J. Lingrel, J. Neumann, E. Ponce, 1993, Molecular biology of the cell.

Knockout of the Na,K-ATPase α₂-isoform in the cardiovascular system does not alter basal blood pressure but prevents ACTH-induced hypertension.

J. Lingrel, J. Lorenz, J. Neumann, 2011, American journal of physiology. Heart and circulatory physiology.

Tissue-specific regulation of the alpha-myosin heavy chain gene promoter in transgenic mice.

J. Robbins, J. Gulick, A. Subramaniam, 1991, The Journal of biological chemistry.

The alpha2 isoform of Na,K-ATPase mediates ouabain-induced cardiac inotropy in mice.

I. Grupp, J. Lingrel, J. Lorenz, 2003, The Journal of biological chemistry.

PACAP induces bradycardia in guinea-pig heart by stimulation of atrial cholinergic neurones

H. Kilbinger, J. Neumann, S. Herzig, 1996, Naunyn-Schmiedeberg's Archives of Pharmacology.

Molecular and Physiological Effects of Overexpressing Striated Muscle β-Tropomyosin in the Adult Murine Heart (*)

G. Boivin, I. Grupp, G. Grupp, 1995, The Journal of Biological Chemistry.

Effects of serine/threonine protein phosphatases on ion channels in excitable membranes.

J. Neumann, S. Herzig, Stefan Herzig, 2000, Physiological reviews.

Identification of maternally regulated fetal gene networks in the placenta with a novel embryo transfer system in mice.

P. Succop, M. Medvedovic, S. Karyala, 2011, Physiological genomics.

Mouse phospholamban gene expression during development in vivo and in vitro.

Ha Won Kim, T. Doetschman, I. Grupp, 1992, Circulation research.

Reduced Levels of microRNAs miR-124a and miR-150 Are Associated with Increased Proinflammatory Mediator Expression in Krüppel-like Factor 2 (KLF2)-deficient Macrophages*

J. Lingrel, D. Hui, J. Neumann, 2014, The Journal of Biological Chemistry.

Altered phosphatase activity in heart failure, influence on Ca2+ movement

J. Neumann, 2002, Basic Research in Cardiology.

In vivo regulation of the mouse beta myosin heavy chain gene.

H. Rindt, J. Robbins, J. Neumann, 1994, The Journal of biological chemistry.

In vivo analysis of the murine beta-myosin heavy chain gene promoter.

H. Rindt, J. Robbins, J. Gulick, 1993, The Journal of biological chemistry.

SIMULATION OF SOME DYNAMIC PROPERTIES OF SARCOMERE INSIDE THE CARDIOMYOCYTE

J. Neumann, V. Novak, V. Novák, 2009 .

Mathematical model of the electromechanical heart contractile system--regulatory subsystem physiological considerations.

O Kittnar, M Mlcek, V. Novák, 2001, Physiological research.