Indroneal Banerjee

发表

Pressure overload induces early morphological changes in the heart.

T. Borg, T. Baudino, Colby A. Souders, 2012, The American journal of pathology.

Disruption of both nesprin 1 and desmin results in nuclear anchorage defects and fibrosis in skeletal muscle.

R. Lieber, Ling T Guo, G. Shelton, 2014, Human molecular genetics.

Transcription factor ISL 1 is essential for pacemaker development and function

C. Glass, Yunfu Sun, Cizhong Jiang, 2022 .

c-Myc Is Required for Proper Coronary Vascular Formation via Cell- and Gene-Specific Signaling

Stephanie L. K. Bowers, S. L. Bowers, T. Baudino, 2012, Arteriosclerosis, thrombosis, and vascular biology.

Lmo7 is dispensable for skeletal muscle and cardiac function.

Yusu Gu, N. Dalton, K. Peterson, 2015, American journal of physiology. Cell physiology.

Lmo 7 is dispensable for skeletal muscle and cardiac function

Yusu Gu, N. Dalton, K. Peterson, 2015 .

RhoA protects the mouse heart against ischemia/reperfusion injury.

G. Dorn, R. Lang, S. Miyamoto, 2011, The Journal of clinical investigation.

muscle: insights from animal models Functional characteristics of dystrophic skeletal

E. Hoffman, R. Lieber, J. Watchko, 2015 .

The Role of Interleukin-6 in the Formation of the Coronary Vasculature

S. L. Bowers, T. Baudino, Colby A. Souders, 2009, Microscopy and Microanalysis.

Altered myocyte contractility and calcium homeostasis in alpha-myosin heavy chain point mutations linked to familial dilated cardiomyopathy.

T. Herron, Matthew Klos, Sherry Morgenstern, 2017, Archives of biochemistry and biophysics.

Thymosin Beta 4 Is Dispensable for Murine Cardiac Development and Function

Yusu Gu, N. Dalton, K. Peterson, 2012, Circulation research.

Loss of FHL1 induces an age-dependent skeletal muscle myopathy associated with myofibrillar and intermyofibrillar disorganization in mice.

J. Seidman, R. Lieber, C. Seidman, 2014, Human molecular genetics.

IL-6 loss causes ventricular dysfunction, fibrosis, reduced capillary density, and dramatically alters the cell populations of the developing and adult heart.

T. Baudino, Indroneal Banerjee, J. Fuseler, 2009, American journal of physiology. Heart and circulatory physiology.

The extracellular matrix: at the center of it all.

Stephanie L. K. Bowers, S. L. Bowers, T. Baudino, 2010, Journal of molecular and cellular cardiology.

Transcription factor ISL1 is essential for pacemaker development and function.

C. Glass, Yunfu Sun, Cizhong Jiang, 2015, The Journal of clinical investigation.

Resident fibroblast lineages mediate pressure overload-induced cardiac fibrosis.

D. Brenner, Yusu Gu, N. Dalton, 2014, The Journal of clinical investigation.

Repression of the Central S plicing Regulator RBFox 2 Is Functionally Linked to Pressure Overload-Induced Heart Failure Graphical

Xiang-Dong Fu, Hai-Ri Li, Long-Sheng Song, 2015 .

Determination of cell types and numbers during cardiac development in the neonatal and adult rat and mouse.

Thomas K Borg, T. Borg, R. Price, 2007, American journal of physiology. Heart and circulatory physiology.

miR-486 is modulated by stretch and increases ventricular growth

K. Peterson, M. Ghassemian, J. Lincoln, 2019, JCI insight.

ALP/Enigma PDZ-LIM domain proteins in the heart.

Hongqiang Cheng, Ju Chen, M. Zheng, 2010, Journal of molecular cell biology.

Neonatal and Adult Cardiovascular Pathophysiological Remodeling and Repair

T. Borg, R. Markwald, T. Baudino, 2008, Annals of the New York Academy of Sciences.

Targeted Ablation of Nesprin 1 and Nesprin 2 from Murine Myocardium Results in Cardiomyopathy, Altered Nuclear Morphology and Inhibition of the Biomechanical Gene Response

Kyle S. Buchholz, L. Gerace, Ju Chen, 2014, PLoS genetics.

Linker of nucleoskeleton and cytoskeleton complex proteins in cardiac structure, function, and disease.

Ju Chen, Indroneal Banerjee, Jennifer Veevers, 2014, Circulation research.

Cyclic stretch of embryonic cardiomyocytes increases proliferation, growth, and expression while repressing Tgf-β signaling.

G. Hardiman, M. Mercola, S. Aceves, 2015, Journal of molecular and cellular cardiology.

Microscopy ambient ionization top-down mass spectrometry reveals developmental patterning

P. Dorrestein, E. Macagno, S. Pfaff, 2013, Proceedings of the National Academy of Sciences.

Integrins protect cardiomyocytes from ischemia/reperfusion injury.

Susan S. Taylor, G. Perkins, J. Copps, 2013, The Journal of clinical investigation.

Thymosin &bgr;4 Is Not Required for Embryonic Viability or Vascular Development

Ju Chen, S. Evans, Indroneal Banerjee, 2013, Circulation research.

Infarct Fibroblasts Do Not Derive From Bone Marrow Lineages

Yusu Gu, N. Dalton, K. Peterson, 2017, Circulation research.

Dynamic Interactions between Myocytes, Fibroblasts, and Extracellular Matrix

Thomas K Borg, T. Borg, T. Baudino, 2006, Annals of the New York Academy of Sciences.

Repression of the Central Splicing Regulator RBFox2 Is Functionally Linked to Pressure Overload-Induced Heart Failure.

Xiang-Dong Fu, Hai-Ri Li, Long-Sheng Song, 2015, Cell reports.

Integrative and Translational Physiology : Integrative Aspects of Energy Homeostasis and Metabolic Diseases Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy

Indroneal Banerjee, S. Ihm, F. Villarreal, 2012 .

The extracellular matrix: at the center of it all.

Stephanie L. K. Bowers, T. Baudino, Indroneal Banerjee, 2010, Journal of molecular and cellular cardiology.

ALP/Enigma PDZ-LIM domain proteins in the heart.

Hongqiang Cheng, Ju Chen, M. Zheng, 2010, Journal of molecular cell biology.

IL-6 loss causes ventricular dysfunction, fibrosis, reduced capillary density, and dramatically alters the cell populations of the developing and adult heart.

T. Baudino, Indroneal Banerjee, J. Fuseler, 2009, American journal of physiology. Heart and circulatory physiology.

CALL FOR PAPERS Integrative and Translational Physiology: Integrative Aspects of Energy Homeostasis and Metabolic Diseases Excess protein O-GlcNAcylation and the progression of diabetic cardiomyopathy

Indroneal Banerjee, S. Ihm, W. Dillmann, 2012 .