F. Swift

发表

Mutual antagonism between IP3RII and miRNA-133a regulates calcium signals and cardiac hypertrophy

K. Mikoshiba, D. Catalucci, M. Bootman, 2012, The Journal of cell biology.

Mutual antagonism between IP3RII and miRNA-133a regulates calcium signals and cardiac hypertrophy

K. Mikoshiba, D. Catalucci, M. Bootman, 2013 .

Hypokalemia induces Ca 2 + overload and Ca 2 + waves in ventricular myocytes by reducing NKA α 2

I. Sjaastad, S. Niederer, N. Smith, 2022 .

Slowed relaxation and preserved maximal force in soleus muscles of mice with targeted disruption of the Serca2 gene in skeletal muscle

Ø. Ellingsen, O. Sejersted, G. Christensen, 2011, The Journal of physiology.

The Na+/K+-ATPase alpha2-isoform regulates cardiac contractility in rat cardiomyocytes.

I. Sjaastad, O. Sejersted, F. Swift, 2007, Cardiovascular research.

Normal contractions triggered by I(Ca,L) in ventricular myocytes from rats with postinfarction CHF.

I. Sjaastad, O. Sejersted, F. Swift, 2002, American journal of physiology. Heart and circulatory physiology.

Extreme sarcoplasmic reticulum volume loss and compensatory T-tubule remodeling after Serca2 knockout

C. Franzini-armstrong, O. Sejersted, G. Christensen, 2012, Proceedings of the National Academy of Sciences.

Serotonin responsiveness through 5-HT2A and 5-HT4 receptors is differentially regulated in hypertrophic and failing rat cardiac ventricle.

K. Krobert, I. Sjaastad, O. Sejersted, 2006, Journal of molecular and cellular cardiology.

Slow diffusion of K (cid:1) in the T tubules of rat cardiomyocytes

I. Sjaastad, T. Strømme, O. Sejersted, 2006 .

Altered Na+/Ca2+-exchanger activity due to downregulation of Na+/K+-ATPase alpha2-isoform in heart failure.

I. Sjaastad, O. Sejersted, J. M. Aronsen, 2008, Cardiovascular research.

Serotonin increases L-type Ca2+ current and SR Ca2+ content through 5-HT4 receptors in failing rat ventricular cardiomyocytes.

I. Sjaastad, O. Sejersted, F. Levy, 2007, American journal of physiology. Heart and circulatory physiology.

rat ventricular cardiomyocytes

I. Sjaastad, O. Sejersted, F. Levy, 2007 .

Dual Serotonergic Regulation of Ventricular Contractile Force Through 5-HT2A and 5-HT4 Receptors Induced in the Acute Failing Heart

K. Krobert, I. Sjaastad, O. Sejersted, 2005, Circulation research.

There Goes the Neighborhood: Pathological Alterations in T-Tubule Morphology and Consequences for Cardiomyocyte Ca2+ Handling

William E. Louch, O. Sejersted, W. Louch, 2010, Journal of biomedicine & biotechnology.

Corrigendum to: Altered Na+/Ca2+-exchanger activity due to downregulation of Na+/K+-ATPase α2-isoform in heart failure

I. Sjaastad, O. Sejersted, J. M. Aronsen, 2008 .

Hypokalaemia induces Ca2+ overload and Ca2+ waves in ventricular myocytes by reducing Na+,K+-ATPase α2 activity.

I. Sjaastad, S. Niederer, N. Smith, 2014, The Journal of physiology.

Restricted diffusion of ions regulates cardiac function

F. Swift, 2007 .

From global to local: a new understanding of cardiac electromechanical coupling.

I. Sjaastad, O. Sejersted, F. Swift, 2012, Tidsskrift for den Norske laegeforening : tidsskrift for praktisk medicin, ny raekke.

Cardiac sodium transport and excitation-contraction coupling.

O. Sejersted, J. M. Aronsen, F. Swift, 2013, Journal of molecular and cellular cardiology.

Functional coupling of alpha(2)-isoform Na(+)/K(+)-ATPase and Ca(2+) extrusion through the Na(+)/Ca(2+)-exchanger in cardiomyocytes.

I. Sjaastad, M. Egger, E. Niggli, 2010, Cell calcium.

Slow diffusion of K+ in the T tubules of rat cardiomyocytes.

I. Sjaastad, T. Strømme, O. Sejersted, 2006, Journal of applied physiology.

Sodium accumulation promotes diastolic dysfunction in end‐stage heart failure following Serca2 knockout

I. Sjaastad, O. Sejersted, G. Christensen, 2010, The Journal of physiology.

Coupling of the Na+/K+-ATPase to Ankyrin B controls Na+/Ca2+ exchanger activity in cardiomyocytes.

C. Carlson, I. Sjaastad, O. Sejersted, 2019, Cardiovascular research.

I(CaL) inhibition prevents arrhythmogenic Ca(2+) waves caused by abnormal Ca(2+) sensitivity of RyR or SR Ca(2+) accumulation.

I. Sjaastad, O. Sejersted, W. Louch, 2013, Cardiovascular research.

Calcium release units in heart failure: that's about the size of it.

G. Christensen, F. Swift, 2012, Cardiovascular research.

There Goes the Neighborhood: Pathological Alterations in T-Tubule Morphology and Consequences for Cardiomyocyte Ca2+ Handling

O. Sejersted, W. Louch, F. Swift, 2010, Journal of biomedicine & biotechnology.

Hypokalaemia induces Ca2+ overload and Ca2+ waves in ventricular myocytes by reducing Na+,K+‐ATPase α2 activity

I. Sjaastad, O. Sejersted, J. M. Aronsen, 2015, The Journal of physiology.

Cardiac sodium transport and excitation-contraction coupling.

O. Sejersted, J. M. Aronsen, F. Swift, 2013, Journal of molecular and cellular cardiology.

Functional coupling of alpha(2)-isoform Na(+)/K(+)-ATPase and Ca(2+) extrusion through the Na(+)/Ca(2+)-exchanger in cardiomyocytes.

I. Sjaastad, M. Egger, E. Niggli, 2010, Cell calcium.