F. Maher

S. Vannucci, I. Simpson, F. Maher, 1993, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Survival of Cultured Neurons from Amyloid Precursor Protein Knock-Out Mice against Alzheimer’s Amyloid-β Toxicity and Oxidative Stress

C. Masters, G. Multhaup, K. Beyreuther, 1998, The Journal of Neuroscience.

Expression and regulation by insulin of GLUT 3 in UMR 106-01, a clonal rat osteosarcoma cell line.

J. Best, F. Maher, S. Rogers, 1996, Biochemical and biophysical research communications.

Insulin receptor expression in the Burkitt lymphoma cells Daudi and Raji.

J. D. Newman, L. Harrison, I. Campbell, 1989, Molecular endocrinology.

Regulation of cell surface GLUT1, GLUT3, and GLUT4 by insulin and IGF‐I in L6 myotubes

Y. Mitsumoto, A. Klip, F. Maher, 1995, FEBS letters.

The Alzheimer's Disease Amyloid Precursor Protein Modulates Copper-Induced Toxicity and Oxidative Stress in Primary Neuronal Cultures

C. Masters, A. Bush, G. Multhaup, 1999, The Journal of Neuroscience.

Glucose Transporter Expression in Brain: Relationship to Cerebral Glucose Utilization

C. B. Smith, P. Davies, S. Vannucci, 1998, Developmental Neuroscience.

Astrocyte-Neuron Metabolic Pathways

C. B. Smith, P. Davies, S. Vannucci, 1998 .

Expression of two glucose transporters, GLUT1 and GLUT3, in cultured cerebellar neurons: Evidence for neuron-specific expression of GLUT3

R. Henneberry, I. Simpson, F. Maher, 1991, Molecular and Cellular Neuroscience.

Cerebral Glucose Utilization and Glucose Transporter Expression: Response to Water Deprivation and Restoration

C. B. Smith, S. Vannucci, I. Simpson, 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

Non-Aβ Component of Alzheimer's Disease Amyloid (NAC) Revisited: NAC and α-Synuclein Are Not Associated with Aβ Amyloid

Fran Maher, Colin L. Masters, Catriona McLean, 1999 .

Non-Abeta component of Alzheimer's disease amyloid (NAC) revisited. NAC and alpha-synuclein are not associated with Abeta amyloid.

C. Masters, K. Beyreuther, P. Jäkälä, 1999, The American journal of pathology.

Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicity.

K. Beyreuther, R. Cappai, C. Masters, 1999, The American journal of pathology.

Interaction of 7-bromoacetyl-7-desacetylforskolin, and alkylating derivative of forskolin, with bovine brain adenylyl cyclase and human erythrocyte glucose transporter.

I. Simpson, F. Maher, A. Laurenza, 1993, Biochemistry.

Immunolocalization of GLUT1 and GLUT3 glucose transporters in primary cultured neurons and glia

F. Maher, 1995, Journal of neuroscience research.

Diverse fibrillar peptides directly bind the Alzheimer’s amyloid precursor protein and amyloid precursor-like protein 2 resulting in cellular accumulation

C. Masters, G. Multhaup, K. Beyreuther, 2003, Brain Research.

Alterations in brain glucose transporter proteins, GLUT1 and GLUT3, in streptozotocin diabetic rats.

S. Vannucci, I. Simpson, F. Maher, 1993, Advances in experimental medicine and biology.

Modulation of Expression of Glucose Transporters GLUT3 and GLUT1 by Potassium and N-Methyl-d-aspartate in Cultured Cerebellar Granule Neurons

I. Simpson, F. Maher, 1994, Molecular and Cellular Neuroscience.