M. Schröter

发表

APRIL, a New Ligand of the Tumor Necrosis Factor Family, Stimulates Tumor Cell Growth

J. Tschopp, L. French, M. Hahne, 1998, The Journal of experimental medicine.

Inhibition of death receptor signals by cellular FLIP

Margot Thome, Kay Hofmann, Pascal Schneider, 1997, Nature.

The A-kinase anchoring protein ( AKAP ) glycogen synthase kinase 3 { beta } interaction protein ( GSKIP ) regulates { beta }-catenin through its interactions with both protein kinase A ( PKA ) and GSK 3 { beta }

W. Birchmeier, Alessandro Dema, P. Skroblin, 2017 .

The A-Kinase Anchoring Protein (AKAP) Glycogen Synthase Kinase 3β Interaction Protein (GSKIP) Regulates β-Catenin through Its Interactions with Both Protein Kinase A (PKA) and GSK3β *

W. Birchmeier, Alessandro Dema, P. Skroblin, 2016, The Journal of Biological Chemistry.

Timed Action of IL-27 Protects from Immunopathology while Preserving Defense in Influenza

J. Schwab, A. Scheffold, C. Hunter, 2014, PLoS pathogens.

Oncogenic Ras inhibits Fas ligand‐mediated apoptosis by downregulating the expression of Fas

J. Tschopp, M. Hahne, M. Schröter, 1999, The EMBO journal.

FLIP prevents apoptosis induced by death receptors but not by perforin/granzyme B, chemotherapeutic drugs, and gamma irradiation.

J. Tschopp, M. Hahne, M. Irmler, 1998, Journal of immunology.

Identification of two regulatory elements controlling Fucosyltransferase 7 transcription in murine CD4+ T cells.

A. Hamann, U. Syrbe, M. Schröter, 2014, Molecular immunology.

Regulation of Fas(Apo‐1/CD95)‐ and perforinmediated lytic pathways of primary cytotoxic T lymphocytes by the protooncogene bcl‐2

C. Borner, J. Tschopp, M. Schröter, 1995, European journal of immunology.

This information is current as Irradiation Chemotherapeutic Drugs , and Gamma Receptors But Not by Perforin / Granzyme B , FLIP Prevents Apoptosis Induced by Death

J. Tschopp, M. Hahne, M. Irmler, 1998 .

Fas and Fas ligand expression in tumor cells and in vascular smooth‐muscle cells of colonic and renal carcinomas

J. Tschopp, L. French, L. Guillou, 1999, International journal of cancer.

The resolution of lesions induced by Leishmania major in mice requires a functional Fas (APO‐1, CD 95) pathway of cytotoxicity

J. Tschopp, M. Hahne, J. Louis, 1998, European journal of immunology.

Fas-dependent tissue turnover is implicated in tumor cell clearance

J. Tschopp, M. Hahne, M. Schröter, 2000, Oncogene.

Involvement of the proteasome in the programmed cell death of NGF‐deprived sympathetic neurons.

J. Martinou, I. Martinou, M. Irmler, 1996, The EMBO journal.

Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors

J. Tschopp, M. Peter, E. Meinl, 1997, Nature.

TRAMP, a novel apoptosis-mediating receptor with sequence homology to tumor necrosis factor receptor 1 and Fas(Apo-1/CD95).

J. Tschopp, L. French, M. Hahne, 1997, Immunity.

Inhibitionof death receptor signalsbycellular FLIP

J. Tschopp, L. French, M. Hahne, 1997 .

Characterization of the non-functional Fas ligand of gld mice.

M C Peitsch, M. Peitsch, J. Tschopp, 1995, International immunology.

Melanoma Cell Expression of Fas(Apo-1/CD95) Ligand: Implications for Tumor Immune Escape

J. Tschopp, L. French, M. Hahne, 1996, Science.

Core 2 ß1,6-N-acetylglucosaminyltransferase-I, crucial for P-selectin ligand expression is controlled by a distal enhancer regulated by STAT4 and T-bet in CD4+ T helper cells 1.

M. Sperandio, A. Hamann, U. Syrbe, 2016, Molecular immunology.

Core 2 ß1,6-N-acetylglucosaminyltransferase-I, crucial for P-selectin ligand expression is controlled by a distal enhancer regulated by STAT4 and T-bet in CD4+ T helper cells 1.

M. Sperandio, A. Hamann, U. Syrbe, 2016, Molecular immunology.