S. Zinkel

发表

Proapoptotic Bid mediates the Atr-directed DNA damage response to replicative stress

Q. Shi, Y. Liu, C. Bertram, 2011, Cell Death and Differentiation.

THE ROLE OF BAD PHOSPHORYLATION STATUS AND BINDING PARTNERS IN PROMOTING APOPTOSIS By Leta

V. Lavin, S. Zinkel, R. Moser, 2007 .

Proapoptotic BID is required for myeloid homeostasis and tumor suppression.

F. Alt, S. Korsmeyer, J. Kutok, 2003, Genes & development.

Bid Regulates the Pathogenesis of Neurotropic Reovirus

A. Pruijssers, T. Dermody, S. Zinkel, 2010, PLoS pathogens.

Cardiolipin fatty acid remodeling regulates mitochondrial function by modifying the electron entry point in the respiratory chain.

O. Boutaud, L. Roberts, S. Dikalov, 2016, Mitochondrion.

The loss of the BH3-only Bcl-2 family member Bid delays T-cell leukemogenesis in Atm−/− mice

Q. Shi, S. Zinkel, S. Biswas, 2013, Cell Death and Differentiation.

Investigation of the proapoptotic BCL-2 family member bid on the crossroad of the DNA damage response and apoptosis.

S. Zinkel, 2008, Methods in enzymology.

Bid protects the mouse hematopoietic system following hydroxyurea-induced replicative stress

Y. Liu, S. Zinkel, A. Aiello, 2012, Cell Death and Differentiation.

Mechanistic Deep Learning Framework on Cell Traits Derived from Single-Cell Mass Cytometry Data

E. Gamazon, S. Zinkel, Bo Wang, 2022, bioRxiv.

Comparative gel electrophoresis measurement of the DNA bend angle induced by the catabolite activator protein

D. Crothers, S. Zinkel, 1990, Biopolymers.

Identification of a negative regulatory element that inhibits c-mos transcription in somatic cells

G. Cooper, J. Szeberényi, S. Zinkel, 1992, Molecular and cellular biology.

Acetaminophen inhibits cytochrome c redox cycling induced lipid peroxidation.

O. Boutaud, J. Oates, Q. Shi, 2012, Biochemical and biophysical research communications.

MTG16 contributes to colonic epithelial integrity in experimental colitis

Dawn B. Beaulieu, Lori A. Coburn, M. Washington, 2012, Gut.

DNA bend direction by phase sensitive detection

D. Crothers, S. Zinkel, 1987, Nature.

BCL2 family in DNA damage and cell cycle control

E. Yang, A. Gross, A. Gross, 2006, Cell Death and Differentiation.

c-mos expression in mouse oocytes is controlled by initiator-related sequences immediately downstream of the transcription initiation site

G. Cooper, S. Zinkel, A. Kiessling, 1991, Molecular and cellular biology.

Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis

S. Korsmeyer, Yongge Zhao, K. Roth, 1999, Nature.

In aged mice, low surrogate light chain promotes pro-B-cell apoptotic resistance, compromises the PreBCR checkpoint, and favors generation of autoreactive, phosphorylcholine-specific B cells

B. Blomberg, D. Frasca, W. Khan, 2015, Aging cell.

The genetic architecture of human infectious diseases and pathogen-induced cellular phenotypes

A. Hale, E. Gamazon, S. Schiff, 2020, medRxiv.

A Role for Proapoptotic BID in the DNA-Damage Response

S. Korsmeyer, A. Gross, K. Hurov, 2005, Cell.

Death and survival signals determine active/inactive conformations of pro-apoptotic BAX, BAD, and BID molecules.

S. Korsmeyer, J. Zha, M. Wei, 1999, Cold Spring Harbor symposia on quantitative biology.

Conditional MLL‐CBP targets GMP and models therapy‐related myeloproliferative disease

T. Golub, S. Armstrong, P. Febbo, 2005, The EMBO journal.

Transgenic studies with a keratin promoter-driven growth hormone transgene: prospects for gene therapy.

E. Fuchs, K. Polonsky, X. Wang, 1997, Proceedings of the National Academy of Sciences of the United States of America.

AWARD NUMBER: W81XWH-16-1-0057 TITLE: Regulation of Programmed Necrosis and Bone Marrow Failure

Principal Investigator, S. Zinkel, 2017 .

Bid maintains mitochondrial cristae structure and function and protects against cardiac disease in an integrative genomics study

E. Gamazon, O. Boutaud, Q. Wells, 2018, eLife.

Bid Plays a Role in the DNA Damage Response

A. Gross, K. Hurov, S. Zinkel, 2007, Cell.

A role for proapoptotic Bax and Bak in T-cell differentiation and transformation.

Q. Shi, S. Zinkel, S. Cleveland, 2010, Blood.

Rejuvenating Bi(d)ology

A. Gross, A. Gross, S. Zinkel, 2013, Oncogene.

BID mediates neuronal cell death after oxygen/ glucose deprivation and focal cerebral ischemia

N. Plesnila, M. Moskowitz, D. Kohane, 2001, Proceedings of the National Academy of Sciences of the United States of America.

Revisiting the case for genetically engineered mouse models in human myelodysplastic syndrome research.

L. Scott, S. Zinkel, Ting Zhou, 2015, Blood.

BID Binds to Replication Protein A and Stimulates ATR Function following Replicative Stress

W. Chazin, Q. Shi, S. Zinkel, 2011, Molecular and Cellular Biology.

Selective Inhibition of JAK1 Primes STAT5-Driven Human Leukemia Cells for ATRA-Induced Differentiation

James C. Pino, Carlos F. Lopez, Alexander L. R. Lubbock, 2021, Targeted Oncology.

Increased Ripk1-mediated bone marrow necroptosis leads to myelodysplasia and bone marrow failure in mice.

Q. Shi, S. Zinkel, M. Savona, 2019, Blood.

Function of BID – a Molecule of the bcl-2 Family – in Ischemic Cell Death in the Brain

S. Korsmeyer, N. Plesnila, M. Moskowitz, 2002, European Surgical Research.

Catabolite activator protein-induced DNA bending in transcription initiation.

D. Crothers, S. Zinkel, 1991, Journal of molecular biology.

Programmed necroptosis is upregulated in low-grade myelodysplastic syndromes and may play a role in the pathogenesis

S. Zinkel, Qiong Shi, R. Juskevicius, 2021, Experimental hematology.

The broad impact of cell death genes on the human disease phenome

E. Gamazon, S. Zinkel, Phillip Lin, 2023, medRxiv.

Mechanistic Deep Learning Framework on Cell Traits Derived from Single-Cell Mass Cytometry Data

E. Gamazon, S. Zinkel, Bo Wang, 2022, bioRxiv.

Investigation of the proapoptotic BCL-2 family member bid on the crossroad of the DNA damage response and apoptosis.

S. Zinkel, 2008, Methods in enzymology.