D. Menichella

发表

Visualization of Sensory Neurons and Their Projections in an Upper Motor Neuron Reporter Line

A. Paller, B. Genc, P. H. Özdinler, 2015, PloS one.

Protein Zero Is Necessary for E-Cadherin-Mediated Adherens Junction Formation in Schwann Cells

S. Scherer, M. Shy, J. Vallat, 2001, Molecular and Cellular Neuroscience.

Regulation of Myelin‐Specific Gene Expression: Relevance to CMT1

M. Shy, J. Kamholz, R. Awatramani, 1999, Annals of the New York Academy of Sciences.

Restabilization treatment after intravenous immunoglobulin withdrawal in chronic inflammatory demyelinating polyneuropathy: Results from the pre‐randomization phase of the Polyneuropathy And Treatment with Hizentra study

D. Yarnitsky, M. Valis, H. Hartung, 2019, Journal of the peripheral nervous system : JPNS.

Subcutaneous immunoglobulin for maintenance treatment in chronic inflammatory demyelinating polyneuropathy (PATH): a randomised, double-blind, placebo-controlled, phase 3 trial

G. L. Masson, D. Yarnitsky, S. Muley, 2018, The Lancet Neurology.

Heterozygous P0 knockout mice develop a peripheral neuropathy that resembles chronic inflammatory demyelinating polyneuropathy (CIDP).

S. Scherer, M. Shy, W. Xu, 1997, Journal of neuropathology and experimental neurology.

Determining Best or Inferior Drug(s) Using an Adaptive Platform for Cryptogenic Sensory Polyneuropathy

R. Barohn, N. Chahin, K. Sharma, 2022, RRNMF Neuromuscular Journal.

Overcoming Cellular Immunity to Prolong Adenoviral‐Mediated Gene Expression in Sciatic Nerve

M. Shy, J. Kamholz, G. Acsadi, 1999, Annals of the New York Academy of Sciences.

Modulation of cell-mediated immunity prolongs adenovirus-mediated transgene expression in sciatic nerve.

M. Shy, H. Jiang, J. Kamholz, 1999, Human gene therapy.

A subpopulation of peripheral sensory neurons expressing the Mas-related G Protein-Coupled Receptor d (Mrgprd) generates pain hypersensitivity in painful diabetic neuropathy

Richard J. Miller, D. Menichella, A. Malfait, 2022, bioRxiv.

The Absence of Myelin P0 Protein Produces a Novel Molecular Phenotype in Schwann Cell

M. Shy, J. Vallat, P. Baron, 1999, Annals of the New York Academy of Sciences.

Spatial Buffering of Potassium Released during Neuronal Activity Genetic and Physiological Evidence That Oligodendrocyte Gap Junctions Contribute to

J. Trimmer, E. Peles, R. Awatramani, 2015 .

Chemogenetic Inhibition of Pain Neurons in a Mouse Model of Osteoarthritis

Richard J. Miller, A. Delaney, D. Menichella, 2017, Arthritis & rheumatology.

Mitochondrial calcium uniporter deletion prevents painful diabetic neuropathy by restoring mitochondrial morphology and dynamics

J. Savas, S. Edassery, D. Menichella, 2021, Pain.

MicroRNA-Deficient Schwann Cells Display Congenital Hypomyelination

L. Wrabetz, R. Awatramani, D. Menichella, 2010, The Journal of Neuroscience.

Charcot-Marie-Tooth disease type 1: molecular pathogenesis to gene therapy.

S. Scherer, M. Shy, J. Lilien, 2000, Brain : a journal of neurology.

Cutaneous Innervation in Impaired Diabetic Wound Healing.

A. Paller, D. Menichella, Richard Miller, 2021, Translational research : the journal of laboratory and clinical medicine.

Reducing CXCR4-mediated nociceptor hyperexcitability reverses painful diabetic neuropathy

Richard J. Miller, D. Menichella, D. Ren, 2018, The Journal of clinical investigation.

Ganglioside GM3 synthase depletion reverses neuropathic pain and small fiber neuropathy in diet-induced diabetic mice

A. Paller, Richard J. Miller, Xiao-Qi Wang, 2016, Molecular pain.

Genetic and Physiological Evidence That Oligodendrocyte Gap Junctions Contribute to Spatial Buffering of Potassium Released during Neuronal Activity

D. Paul, S. Scherer, R. Awatramani, 2006, The Journal of Neuroscience.

Connexins Are Critical for Normal Myelination in the CNS

D. Paul, S. Scherer, D. Goodenough, 2003, The Journal of Neuroscience.

Regulation of myelin-specific gene expression. Relevance to CMT1.

M. Shy, W. Xu, H. Jiang, 1999, Annals of the New York Academy of Sciences.

CXCR4 chemokine receptor signaling mediates pain in diabetic neuropathy

Richard J. Miller, D. Menichella, D. Ren, 2014, Molecular pain.

Heterozygous null mutation in the P0 gene associated with mild Charcot-Marie-Tooth disease.

D. Pareyson, M. Shy, M. Mora, 1999, Annals of the New York Academy of Sciences.

Efficacy and safety of IVIG in CIDP: Combined data of the PRIMA and PATH studies

G. L. Masson, D. Yarnitsky, M. Valis, 2019, Journal of the peripheral nervous system : JPNS.

Reducing CXCR 4-mediated nociceptor hyperexcitability reverses painful diabetic neuropathy

Richard J. Miller, D. Menichella, D. Ren, 2018 .

Reducing CXCR 4-mediated

Richard J. Miller, D. Menichella, D. Ren, 2018 .

Human cells and networks of pain: Transforming pain target identification and therapeutic development

S. Waxman, C. Stucky, W. Z. Ray, 2021, Neuron.

Heterozygous Null Mutation in the P0 Gene Associated with Mild Charcot‐Marie‐Tooth Disease

D. Pareyson, M. Shy, F. Taroni, 1999, Annals of the New York Academy of Sciences.

The absence of myelin P0 protein produces a novel molecular phenotype in Schwann cells.

M. Shy, W. Xu, H. Jiang, 1999, Annals of the New York Academy of Sciences.

GeneticandPhysiologicalEvidenceThatOligodendrocyte GapJunctionsContributetoSpatialBufferingofPotassium ReleasedduringNeuronalActivity

D. Menichella, 2006 .

Peripheral mechanisms of peripheral neuropathic pain

D. Menichella, James S. Coy-Dibley, Paola Pacifico, 2023, Frontiers in molecular neuroscience.

NaV channel variants in patients with painful and nonpainful peripheral neuropathy

R. Freeman, S. Ajroud‐Driss, S. Kirov, 2017, Neurology: Genetics.

Misregulation of mitochondria–lysosome contact dynamics in Charcot–Marie–Tooth Type 2B disease Rab7 mutant sensory peripheral neurons

D. Krainc, Y. Wong, D. Menichella, 2023, Proceedings of the National Academy of Sciences of the United States of America.

D. Menichella, A. Malfait, D. Ren, 2023, Pain.

Keratinocyte-Derived Exosomes in Painful Diabetic Neuropathy

D. Menichella, D. Ren, Abdelhak A. Belmadani, 2024, bioRxiv.