I. Blasko

发表

The amyloid β peptide Aβ (25–35) induces apoptosis independent of p53

P. Jansen-Dürr, I. Blasko, M. Wagner, 2000 .

The amyloid beta peptide abeta (25-35) induces apoptosis independent of p53.

P. Jansen-Dürr, I. Blasko, M. Wagner, 2000, FEBS letters.

Conversion from cognitive health to mild cognitive impairment and Alzheimer's disease: Prediction by plasma amyloid beta 42, medial temporal lobe atrophy and homocysteine

Georg Kemmler, Kurt Jellinger, K. Jellinger, 2008, Neurobiology of Aging.

How chronic inflammation can affect the brain and support the development of Alzheimer's disease in old age: the role of microglia and astrocytes

R. Veerhuis, P. Eikelenboom, I. Blasko, 2004, Aging cell.

Role of the Immune System in the Pathogenesis, Prevention and Treatment of Alzheimer’s Disease

I. Blasko, B. Grubeck‐Loebenstein, 2003, Drugs & aging.

Diseases of aging.

P. Jansen-Dürr, I. Blasko, B. Grubeck‐Loebenstein, 2000, Vaccine.

Mechanisms of immune regulation in Alzheimer's disease: a viewpoint.

F. Marx, I. Blasko, B. Grubeck‐Loebenstein, 1999, Archivum immunologiae et therapiae experimentalis.

Plasma amyloid beta protein 42 in non-demented persons aged 75-years: Effects of concomitant medication and medial temporal lobe atrophy

P. Fischer, K. Tragl, K. Jellinger, 2005, Alzheimer's & Dementia.

An imbalance between pro- and anti-inflammatory cytokines, a characteristic feature of old age.

I. Blasko, B. Grubeck‐Loebenstein, M. Saurwein-Teissl, 2000, Cytokine.

Costimulatory Effects of Interferon-γ and Interleukin-1β or Tumor Necrosis Factor α on the Synthesis of Aβ1-40 and Aβ1-42 by Human Astrocytes

R. Veerhuis, P. Eikelenboom, I. Blasko, 2000, Neurobiology of Disease.

The possible role of the immune system in Alzheimer’s disease

F. Marx, I. Blasko, B. Grubeck‐Loebenstein, 1998, Experimental Gerontology.

The production of the Alzheimer amyloid precursor protein (APP) in extraneuronal tissue does not increase in old age.

T. Schmitt, K. Trieb, I. Blasko, 1998, The journals of gerontology. Series A, Biological sciences and medical sciences.

Tumor necrosis factor α augments amyloid β protein (25–35) induced apoptosis in human cells

T. Schmitt, K. Trieb, I. Blasko, 1997, Neuroscience Letters.

Experimental traumatic brain injury in rats stimulates the expression, production and activity of Alzheimer’s disease β-secretase (BACE-1)

G. Ransmayr, A. Kampfl, R. Schliebs, 2004, Journal of Neural Transmission.

Abnormalities of Amyloid β Precursor Protein Expression in Platelets of Patients With Alzheimer Disease: Do We Understand Them Well Enough?—Reply

J. Marksteiner, F. Marx, I. Blasko, 1999 .

Transfected human B cells: a new model to study the functional and immunostimulatory consequences of APP production

F. Marx, I. Blasko, B. Grubeck‐Loebenstein, 1999, Experimental Gerontology.

TNFalpha plus IFNgamma induce the production of Alzheimer beta-amyloid peptides and decrease the secretion of APPs.

T. Hartmann, F. Marx, I. Blasko, 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Tumor necrosis factor alpha augments amyloid beta protein (25-35) induced apoptosis in human cells.

T. Schmitt, K. Trieb, I. Blasko, 1997, Neuroscience letters.

Cognitive deterioration in Alzheimer's disease is accompanied by increase of plasma neopterin.

D. Fuchs, G. Kemmler, A. Griesmacher, 2007, Journal of psychiatric research.

Costimulatory effects of interferon-gamma and interleukin-1beta or tumor necrosis factor alpha on the synthesis of Abeta1-40 and Abeta1-42 by human astrocytes.

R. Veerhuis, P. Eikelenboom, I. Blasko, 2000, Neurobiology of disease.

Lack of Antibody Production Following Immunization in Old Age: Association with CD8+CD28− T Cell Clonal Expansions and an Imbalance in the Production of Th1 and Th2 Cytokines1

Walther Parson, Beatrix Grubeck-Loebenstein, W. Parson, 2002, The Journal of Immunology.

Bile acid quantification of 20 plasma metabolites identifies lithocholic acid as a putative biomarker in Alzheimer’s disease

G. Kemmler, C. Humpel, J. Marksteiner, 2017, Metabolomics.

TNFα plus IFNγ induce the production of Alzheimer β‐amyloid peptides and decrease the secretion of APPs

T. Hartmann, F. Marx, I. Blasko, 1999 .

Plasma amyloid β protein 42 in non-demented persons aged 75 years: Effects of concomitant medication and medial temporal lobe atrophy

G. Kemmler, P. Fischer, K. Tragl, 2005, Neurobiology of Aging.

Treatment patterns in inpatients with bipolar disorder at a psychiatric university hospital over a 9-year period: focus on mood stabilizers

H. Grunze, G. Kemmler, A. Hausmann, 2012, International clinical psychopharmacology.

Effects of medications on plasma amyloid beta (Abeta) 42: longitudinal data from the VITA cohort.

K. Jellinger, H. Hinterhuber, G. Kemmler, 2008, Journal of psychiatric research.

Prospective study on association between plasma amyloid beta-42 and atherosclerotic risk factors

K. Jellinger, G. Kemmler, P. Fischer, 2011, Journal of Neural Transmission.

Ibuprofen Decreases Cytokine-Induced Amyloid Beta Production in Neuronal Cells

Gerhard Ransmayr, Beatrix Grubeck-Loebenstein, G. Ransmayr, 2001, Neurobiology of Disease.

Severity of Depression Impacts Imminent Conversion from Mild Cognitive Impairment to Alzheimer's Disease.

G. Kemmler, J. Marksteiner, E. Deisenhammer, 2017, Journal of Alzheimer's disease : JAD.

Plasma phosphatidylcholines and vitamin B12/folate levels are possible prognostic biomarkers for progression of Alzheimer's disease

G. Kemmler, C. Humpel, J. Marksteiner, 2021, Experimental Gerontology.

Immunization with β-amyloid: could T-cell activation have a harmful effect?

K. Trieb, F. Marx, I. Blasko, 2000, Trends in Neurosciences.

Efficacy of Donepezil Treatment in Alzheimer Patients with and without Subcortical Vascular Lesions

A. Monsch, H. Hinterhuber, J. Marksteiner, 2004, Pharmacology.

Conversion from mild cognitive impairment to dementia: Influence of folic acid and vitamin B12 use in the vita cohort

G. Kemmler, P. Fischer, K. Tragl, 2012, The journal of nutrition, health & aging.

Measurement of Thirteen Biological Markers in CSF of Patients with Alzheimer’s Disease and Other Dementias

H. Hinterhuber, G. Kemmler, C. Humpel, 2005, Dementia and Geriatric Cognitive Disorders.

Leisure time activities and cognitive functioning in middle European population-based study

G. Kemmler, P. Fischer, K. Tragl, 2014 .

Use of Benzodiazepines in Alzheimer’s Disease: A Systematic Review of Literature

J. Marksteiner, M. Defrancesco, W. Fleischhacker, 2015, The international journal of neuropsychopharmacology.

Analysis of Cerebrospinal Fluid of Alzheimer Patients

H. Hinterhuber, C. Humpel, J. Marksteiner, 2008, Pharmacology.

Plasma amyloid beta-42 independently predicts both late-onset depression and Alzheimer disease.

K. Jellinger, G. Kemmler, P. Fischer, 2010, The American journal of geriatric psychiatry : official journal of the American Association for Geriatric Psychiatry.

Does IFNγ play a role in neurodegeneration?

R. Veerhuis, P. Eikelenboom, G. Ransmayr, 2001, Journal of Neuroimmunology.

Does IFNgamma play a role in neurodegeneration?

R. Veerhuis, P. Eikelenboom, G. Ransmayr, 2001, Journal of Neuroimmunology.

Plasma levels of amyloid ß-protein 42 are increased in women with mild cognitive impairment

K. Jellinger, P. Fischer, I. Blasko, 2018 .

Glial Cells: Astrocytes and Oligodendrocytes during Normal Brain Aging

C. Humpel, I. Blasko, B. Grubeck‐Loebenstein, 2009 .

Sex steroids do not prevent amylin-induced apoptosis in human cells.

P. Pozzilli, M. Cavallo, I. Blasko, 1998, Experimental cell research.

Analysis of Cerebrospinal Fluid of Alzheimer Patients

H. Hinterhuber, C. Humpel, J. Marksteiner, 2008, Pharmacology.