N. von der Lehr

c-Myc hot spot mutations in lymphomas result in inefficient ubiquitination and decreased proteasome-mediated turnover.

L. Larsson, F. Bahram, N. von der Lehr, 2000, Blood.

The tumor suppressor gene hCDC4 is frequently mutated in human T-cell acute lymphoblastic leukemia with functional consequences for Notch signaling.

U. Lendahl, D. Grandér, O. Sangfelt, 2007, Cancer research.

The F-box protein Skp2 participates in c-Myc proteosomal degradation and acts as a cofactor for c-Myc-regulated transcription.

K. Nakayama, K. Nakayama, T. Kerppola, 2003, Molecular cell.

Phosphorylation by Cdk2 is required for Myc to repress Ras-induced senescence in cotransformation

M. Henriksson, L. Larsson, A. Grandien, 2009, Proceedings of the National Academy of Sciences.

FBXW7/hCDC4 is a general tumor suppressor in human cancer.

S. Reed, S. Apostolidou, M. Widschwendter, 2007, Cancer research.

Interferon-γ-induced p27KIP1 binds to and targets MYC for proteasome-mediated degradation

O. Frings, L. Larsson, P. Hydbring, 2015, Oncotarget.