Rosine Onclercq-Delic

发表

Predisposition to therapy-related acute leukemia with balanced chromosomal translocations does not result from a major constitutive defect in DNA double-strand break end joining.

P. Fenaux, V. Coiteux, Rosine Onclercq-Delic, 2007, Leukemia research : a Forum for Studies on Leukemia and Normal Hemopoiesis.

A Major Role for Mitotic cdc2 Kinase Inactivation in the Establishment of the Mitotic DNA Damage Checkpoint

S. Leibovitch, Rosine Onclercq-Delic, M. Amor-Guéret, 2004, Cancer Research.

Cytidine deaminase deficiency impairs sister chromatid disjunction by decreasing PARP-1 activity

C. Jaulin, Rosine Onclercq-Delic, M. Amor-Guéret, 2017, Cell cycle.

Three new BLM gene mutations associated with Bloom syndrome.

D. Stoppa-Lyonnet, A. Barakat, J. Couturier, 2008, Genetic testing.

Possible anti-recombinogenic role of Bloom's syndrome helicase in double-strand break processing.

C. Delteil, D. Papadopoulo, P. Calsou, 2003, Nucleic acids research.

Cytidine deaminase deficiency in tumor cells is associated with sensitivity to a naphthol derivative and a decrease in oncometabolite levels

G. Fontaine, Rosine Onclercq-Delic, M. Amor-Guéret, 2022, Cellular and Molecular Life Sciences.

A decrease in NAMPT activity impairs basal PARP-1 activity in cytidine deaminase deficient-cells, independently of NAD+

J. Guitton, P. Duchambon, Rosine Onclercq-Delic, 2020, Scientific Reports.

Cytidine deaminase activity increases in the blood of breast cancer patients

S. Vacher, I. Bièche, D. Stoppa-Lyonnet, 2022, Scientific Reports.

The Bloom Syndrome Protein Limits the Lethality Associated with RAD51 Deficiency

Pauline Chabosseau, J. Couturier, F. Cordelières, 2010, Molecular Cancer Research.

A balanced pyrimidine pool is required for optimal Chk1 activation to prevent ultrafine anaphase bridge formation

D. Biard, S. Lambert, Rosine Onclercq-Delic, 2016, Journal of Cell Science.

Bloom’s Syndrome and PICH Helicases Cooperate with Topoisomerase IIα in Centromere Disjunction before Anaphase

L. Magnaghi-Jaulin, I. Hurbain, C. Jaulin, 2012, PloS one.

BLM Deficiency Is Not Associated with Sensitivity to Hydroxyurea-Induced Replication Stress

Rosine Onclercq-Delic, M. Amor-Guéret, Géraldine Buhagiar-Labarchède, 2010, Journal of nucleic acids.

A role for Tau protein in maintaining ribosomal DNA stability and cytidine deaminase-deficient cell survival

Michael R. Green, J. Guitton, Rosine Onclercq-Delic, 2017, Nature Communications.

Cytidine Deaminase Deficiency Reveals New Therapeutic Opportunities against Cancer

W. Reinhold, Y. Pommier, S. Vacher, 2016, Clinical Cancer Research.

Pyrimidine Pool Disequilibrium Induced by a Cytidine Deaminase Deficiency Inhibits PARP-1 Activity, Leading to the Under Replication of DNA

M. Lopes, D. Biard, S. Lambert, 2015, PLoS genetics.

Cytidine deaminase deficiency in mice enhances genetic instability but limits the number of chemically induced colon tumors.

T. Larcher, J. Guitton, S. Leboucher, 2022, Cancer letters.

Dephosphorylation and Subcellular Compartment Change of the Mitotic Bloom's Syndrome DNA Helicase in Response to Ionizing Radiation*

Stéphanie Dutertre, L. Tintignac, C. Jaulin, 2002, The Journal of Biological Chemistry.

Pyrimidine pool imbalance induced by BLM helicase deficiency contributes to genetic instability in Bloom syndrome.

M. Debatisse, Pauline Chabosseau, S. Lambert, 2011, Nature communications.

Topoisomerase IIα prevents ultrafine anaphase bridges by two mechanisms

S. Lambert, G. Fontaine, Rosine Onclercq-Delic, 2020, Open Biology.

Topoisomerase IIα Prevents, But Does Not Resolve, Ultrafine Anaphase Bridges By Two Mechanisms

S. Lambert, Rosine Onclercq-Delic, M. Amor-Guéret, 2019, bioRxiv.

A role for Tau protein in maintaining ribosomal DNA stability and cytidine deaminase-deficient cell survival

Michael R. Green, J. Guitton, Rosine Onclercq-Delic, 2017, Nature Communications.