Rifdat Aoidi

Lung development requires an active ERK/MAPK pathway in the lung mesenchyme

J. Charron, Nicolas Houde, O. Boucherat, 2017, Developmental dynamics : an official publication of the American Association of Anatomists.

Mek1Y130C mice recapitulate aspects of human cardio-facio-cutaneous syndrome

J. Charron, K. Rauen, Nicolas Houde, 2018, Disease Models & Mechanisms.

Fine-tuning of MEK signaling is pivotal for limiting B and T cell activation.

Laurent Beuret, J. Charron, M. Espéli, 2022, Cell reports.

Functional redundancy of the kinases MEK1 and MEK2: Rescue of the Mek1 mutant phenotype by Mek2 knock-in reveals a protein threshold effect

J. Charron, Rifdat Aoidi, A. Maltais, 2016, Science Signaling.

OCRL-mutated fibroblasts from patients with Dent-2 disease exhibit INPP5B-independent phenotypic variability relatively to Lowe syndrome cells.

R. Salomon, P. Billuart, J. Rendu, 2015, Human molecular genetics.

Maternal iron deficiency perturbs embryonic cardiovascular development in mice

Jacob T. Munro, E. Giannoulatou, V. Tybulewicz, 2021, Nature Communications.

MEK2 Negatively Regulates Lipopolysaccharide-Mediated IL-1β Production through HIF-1α Expression

J. Charron, L. Samavati, Nicolas Houde, 2019, The Journal of Immunology.

Maternal iron deficiency perturbs embryonic cardiovascular development

E. Giannoulatou, Jacob E. Munro, V. Tybulewicz, 2020, bioRxiv.

Cognitive impairments in a Down syndrome model with abnormal hippocampal and prefrontal dynamics and cytoarchitecture

M. Walker, D. Bush, M. Good, 2023, iScience.

Congenital heart defects in Down syndrome are caused by increased dosage of DYRK1A

A. Snijders, Rifdat Aoidi, Ok-Ryul Song, 2023, bioRxiv.

Increased dosage of DYRK1A leads to congenital heart defects in a mouse model of Down syndrome

A. Snijders, M. Gutierrez, Rifdat Aoidi, 2024, Science Translational Medicine.