M. Köhnke

发表

A single nucleotide polymorphism at DBH, possibly associated with attention-deficit/hyperactivity disorder, associates with lower plasma dopamine beta-hydroxylase activity and is in linkage disequilibrium with two putative functional single nucleotide polymorphisms.

I. Waldman, G. Anderson, J. Cubells, 2006, Biological psychiatry.

Methylenetetrahydrofolate reductase C677T-polymorphism and its association with alcohol withdrawal seizure.

A. Batra, F. Machicao, M. Köhnke, 2006, Alcoholism, clinical and experimental research.

The serotonin transporter promotor polymorphism 5-HTTLPR is not associated with alcoholism or severe forms of alcohol withdrawal in a German sample.

A. Batra, M. Köhnke, U. Lutz, 2006, Psychiatric genetics.

Allele distribution of a monoamine oxidase A gene promoter polymorphism in German alcoholics and in subgroups with severe forms of alcohol withdrawal and its influence on plasma homovanillic acid.

A. Batra, M. Köhnke, G. Wiatr, 2006, Psychiatric genetics.

A revised allele frequency estimate and haplotype analysis of the DBH deficiency mutation IVS1+2T → C in African‐ and European‐Americans

J. Gelernter, J. Padbury, R. Romero, 2003, American journal of medical genetics. Part A.

Haplotype‐controlled analysis of the association of a non‐synonymous single nucleotide polymorphism at DBH (+ 1603C → T) with plasma dopamine β‐hydroxylase activity

G. Anderson, J. Cubells, C. Zabetian, 2005, American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics.

The structure of linkage disequilibrium at the DBH locus strongly influences the magnitude of association between diallelic markers and plasma dopamine beta-hydroxylase activity.

R. Elston, J. Gelernter, G. Anderson, 2003, American journal of human genetics.

DBH*444G/A polymorphism of the dopamine-β-hydroxylase gene is associated with alcoholism but not with severe alcohol withdrawal symptoms

A. Batra, M. Köhnke, S. Schick, 2006, Journal of Neural Transmission.

A genotype-controlled analysis of plasma dopamine β-hydroxylase in healthy and alcoholic subjects: evidence for alcohol-related differences in noradrenergic function

R. Vonthein, G. Anderson, J. Cubells, 2002, Biological Psychiatry.

Approach to the genetics of alcoholism: a review based on pathophysiology.

M. Köhnke, 2008, Biochemical pharmacology.

No association between dopamine D2 receptor gene (DRD2) and human intelligence

I. Gottesman, L. Yang, H. W. Moises, 2001, Journal of Neural Transmission.

Treatment of alcoholism in Germany.

M. Köhnke, 2009, Annales Academiae Medicae Stetinensis.

GENETIC STUDY: The polymorphism GABABR1 T1974C [rs29230] of the GABAB receptor gene is not associated with the diagnosis of alcoholism or alcohol withdrawal seizures

A. Batra, R. Vonthein, M. Köhnke, 2006, Addiction biology.

Mitochondrial DNA marker EST00083 is not associated with high vs. average IQ in a German sample

R. Plomin, S. Petrill, M. Köhnke, 1998 .

A Single Nucleotide Polymorphism at DBH, Possibly Associated with Attention-Deficit/Hyperactivity Disorder, Associates with Lower Plasma Dopamine β-Hydroxylase Activity and is in Linkage Disequilibrium with Two Putative Functional Single Nucleotide Polymorphisms

I. Waldman, G. Anderson, J. Cubells, 2006, Biological Psychiatry.

Association of the dopamine transporter gene with alcoholism.

A. Batra, M. Köhnke, S. Schick, 2005, Alcohol and alcoholism.

Plasma Homovanillic Acid: A Significant Association with Alcoholism is Independent of a Functional Polymorphism of the Human Catechol-O-Methyltransferase Gene

R. Vonthein, M. Köhnke, S. Schick, 2003, Neuropsychopharmacology.

Cytochrome P450 2D6 dependent metabolization of risperidone is inhibited by melperone

G. Buchkremer, M. Köhnke, K. Schott, 2006, European Journal of Clinical Pharmacology.

Cytochrome P450 2D6 deficiency and its clinical relevance in a patient treated with risperidone.

G. Barth, M. Köhnke, E. Griese, 2002, Pharmacopsychiatry.