Joost Schalkwijk

发表

A guiding map for inflammation

Richard Hotchkiss, Michael T Heneka, Alberto Mantovani, 2017, Nature Immunology.

Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity

Matti Pirinen, Christian Gieger, Peter Donnelly, 2012 .

Transcriptional response of bronchial epithelial cells to Pseudomonas aeruginosa: identification of early mediators of host defense.

K. Rabe, N. Datson, J. Schalkwijk, 2005, Physiological genomics.

Hypertrophic scarring is associated with epidermal abnormalities: an immunohistochemical study

P. C. van de Kerkhof, J. Schalkwijk, Joost Schalkwijk, 1998, The Journal of pathology.

Keratinocytes drive melanoma invasion in a reconstructed skin model

J. Schalkwijk, L. V. van Kempen, Joost Schalkwijk, 2010, Melanoma research.

β-Defensin-2 Protein Is a Serum Biomarker for Disease Activity in Psoriasis and Reaches Biologically Relevant Concentrations in Lesional Skin

Andrea W. M. Evers, Edward J. Hollox, Martin den Heijer, 2009, PloS one.

Differential gene expression in premalignant human epidermis revealed by cluster analysis of serial analysis of gene expression (SAGE) libraries

F. Ruissen, J. Schalkwijk, B. J. Jansen, 2002, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Psoriasis is associated with increased beta-defensin genomic copy number.

André Reis, Edward J Hollox, Martin den Heijer, 2008, Nature genetics.

Immortalized N/TERT keratinocytes as an alternative cell source in 3D human epidermal models

J. Schalkwijk, H. Niehues, G. Rikken, 2017, Scientific Reports.

An overview of methods for the in vivo evaluation of tissue-engineered skin constructs.

E. Middelkoop, P. Verhaegen, J. Schalkwijk, 2011, Tissue engineering. Part B, Reviews.

Psoriasis is associated with increased β-defensin genomic copy number

André Reis, Edward J Hollox, M. Heijer, 2008, Nature Genetics.

Duplicated Enhancer Region Increases Expression of CTSB and Segregates with Keratolytic Winter Erythema in South African and Norwegian Families.

Tomasz Stokowy, Torunn Fiskerstrand, Fan Yang, 2017, American journal of human genetics.

Replication of LCE3C-LCE3B CNV as a risk factor for psoriasis and analysis of interaction with other genetic risk factors.

Xavier Estivill, André Reis, Tim Becker, 2010, The Journal of investigative dermatology.

Meta-analysis confirms the LCE3C_LCE3B deletion as a risk factor for psoriasis in several ethnic groups and finds interaction with HLA-Cw6.

Rafael de Cid, Pui-Yan Kwok, Xavier Estivill, 2011, The Journal of investigative dermatology.

Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis

Gonçalo Abecasis, Pui-Yan Kwok, Xavier Estivill, 2009, Nature Genetics.

A genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1

Matti Pirinen, Peter Donnelly, Robert Plomin, 2010, Nature Genetics.

Increased angiogenesis and blood vessel maturation in acellular collagen-heparin scaffolds containing both FGF2 and VEGF.

J. Schalkwijk, T. V. van Kuppevelt, P. Geutjes, 2007, Biomaterials.