D. Jollow

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Favism: divicine hemotoxicity in the rat.

D. McMillan, D. Jollow, 1999, Toxicological sciences : an official journal of the Society of Toxicology.

Favism: effect of divicine on rat erythrocyte sulfhydryl status, hexose monophosphate shunt activity, morphology, and membrane skeletal proteins.

D. McMillan, D. Jollow, L. J. Bolchoz, 2001, Toxicological sciences : an official journal of the Society of Toxicology.

Acetaminophen-induced hepatic necrosis. I. Role of drug metabolism.

B B Brodie, J R Gillette, B. Brodie, 1973, The Journal of pharmacology and experimental therapeutics.

Role of detoxifying enzymes in bromobenzene-induced liver necrosis.

J R Gillette, D. Jollow, D J Jollow, 1973, The Journal of pharmacology and experimental therapeutics.

Bromobenzene-induced liver necrosis. Protective role of glutathione and evidence for 3,4-bromobenzene oxide as the hepatotoxic metabolite.

D. Jollow, J. Mitchell, J. Gillette, 1974, Pharmacology.

Acetaminophen-induced hepatic necrosis. II. Role of covalent binding in vivo.

B B Brodie, J R Gillette, B. Brodie, 1973, The Journal of pharmacology and experimental therapeutics.

ACETAMINOPHEN-INDUCED HEPATIC NECROSIS. III. CYTOCHROME P-450-MEDIATED COVALENT BINDING IN VITRO

B. Brodie, W. Potter, D. Jollow, 1973 .

Acetaminophen-induced hepatic necrosis. VI. Metabolic disposition of toxic and nontoxic doses of acetaminophen.

S. Thorgeirsson, W. Potter, D. Jollow, 1974, Pharmacology.

Acetaminophen-induced hepatic necrosis. VI. Metabolic disposition of toxic and nontoxic doses of acetaminophen.

S. Thorgeirsson, W. Potter, D. Jollow, 1974, Pharmacology.

Microsomal N-Hydroxylation of p-Chloroacetanilide

D. Jollow, J. Hinson, Jerry R. Mitchell, 1975 .

N-hydroxylation of p-chloroacetanilde in hamsters.

D. Jollow, J. Hinson, J. Mitchell, 1976, Biochemical pharmacology.

Species differences in hepatic glutathione depletion, covalent binding and hepatic necrosis after acetaminophen.

W. Potter, D. Jollow, J. Mitchell, 1974, Life sciences.

Formation of chemically reactive metabolites of phenacetin and acetaminophen.

G. Mulder, L. Pohl, S. Nelson, 1981, Advances in experimental medicine and biology.

Role of sterols in Saccharomyces cerevisiae.

A. Linnane, D. Jollow, J. W. Proudlock, 1968, Biochimica et biophysica acta.

Lipids versus proteins as major targets of pro-oxidant, direct-acting hemolytic agents.

J. Morrow, C. Powell, D. McMillan, 2005, Toxicological sciences : an official journal of the Society of Toxicology.

Primaquine-Induced Hemolytic Anemia: Role of Membrane Lipid Peroxidation and Cytoskeletal Protein Alterations in the Hemotoxicity of 5-Hydroxyprimaquine

J. Morrow, D. McMillan, D. Jollow, 2005, Journal of Pharmacology and Experimental Therapeutics.

Acetaminophen structure-toxicity studies: in vivo covalent binding of a nonhepatotoxic analog, 3-hydroxyacetanilide.

D. Jollow, S. Roberts, V. Price, 1990, Toxicology and applied pharmacology.

Trichloroethylene risk assessment: A review and commentary

J. Fisher, D. Hoel, L. Mohr, 2009, Critical reviews in toxicology.

Improved method for determination of cellular thiols, disulfides and protein mixed disulfides using HPLC with electrochemical detection.

D. Jollow, S. Grossman, C. B. Jensen, 1986, Advances in experimental medicine and biology.

Drug metabolism as a cause of drug toxicity.

B. Brodie, D. Jollow, J. Mitchell, 1973, Drug metabolism and disposition: the biological fate of chemicals.

Acetaminophen‐induced hepatic injury: Protective role of glutathione in man and rationale for therapy

S. Thorgeirsson, W. Potter, D. Jollow, 1974, Clinical pharmacology and therapeutics.

Acetaminophen-induced hepatic necrosis. V. Correlation of hepatic necrosis, covalent binding and glutathione depletion in hamsters.

S. Thorgeirsson, W. Potter, D. Jollow, 1974, Pharmacology.

Diminished serum Gc (vitamin D‐binding protein) levels and increased Gc:G‐actin complexes in a hamster model of fulminant hepatic necrosis

W. M. Lee, P. Goldschmidt-Clermont, D. Emerson, 1987, Hepatology.

Effect of glucose and gluconeogenic substrates on fasting-induced suppression of acetaminophen glucuronidation in the rat.

D. Jollow, V. Price, Veronica F. Price, 1989, Biochemical pharmacology.

Primaquine-Induced Hemolytic Anemia: Role of Splenic Macrophages in the Fate of 5-Hydroxyprimaquine-Treated Rat Erythrocytes

D. McMillan, D. Jollow, Z. Bowman, 2005, Journal of Pharmacology and Experimental Therapeutics.

Acetaminophen hepatotoxicity: studies on the mechanism of cysteamine protection.

D. Jollow, M. Miller, Marion G. Miller, 1986, Toxicology and applied pharmacology.

Effect of cobalt protoporphyrin on hepatic drug-metabolizing enzymes. Specificity for cytochrome P-450.

D. Jollow, S. Spaethe, 1989, Biochemical pharmacology.

Biological Reactive Intermediates IV

D. Jollow, G. Sipes, J. Kocsis, 1991, Advances in Experimental Medicine and Biology.

Role of UDPGA flux in acetaminophen clearance and hepatotoxicity.

D. Jollow, V. Price, 1984, Xenobiotica; the fate of foreign compounds in biological systems.

Increased resistance of diabetic rats to acetaminophen-induced hepatotoxicity.

D. Jollow, V. Price, 1982, The Journal of pharmacology and experimental therapeutics.

Strain differences in susceptibility of normal and diabetic rats to acetaminophen hepatotoxicity.

D. Jollow, V. Price, 1986, Biochemical pharmacology.

Mechanism of decreased acetaminophen glucuronidation in the fasted rat.

D. Jollow, V. Price, 1988, Biochemical pharmacology.

THE PROPERTIES OF A PURIFIED HEPATIC MICROSOMAL MIXED FUNCTION AMINE OXIDASE

D. Ziegler, D. Jollow, C. Mitchell, 1969 .

Biochemical changes after hepatic injury from toxic doses of acetaminophen or furosemide.

S. Thorgeirsson, W. Potter, D. Jollow, 1976, Pharmacology.

Primaquine-Induced Hemolytic Anemia: Effect of 6-Methoxy-8-hydroxylaminoquinoline on Rat Erythrocyte Sulfhydryl Status, Membrane Lipids, Cytoskeletal Proteins, and Morphology

J. Morrow, D. McMillan, D. Jollow, 2002, Journal of Pharmacology and Experimental Therapeutics.

Role of aniline metabolites in aniline-induced hemolytic anemia.

J. H. Harrison, D. Jollow, 1986, The Journal of pharmacology and experimental therapeutics.

Biological Reactive Intermediates VI

H. Greim, T. Monks, R. Snyder, 2001, Advances in Experimental Medicine and Biology.

Role of metabolites in propanil-induced hemolytic anemia.

D. McMillan, D. Jollow, J. Hinson, 1991, Toxicology and applied pharmacology.

Contribution of 3,4-dichlorophenylhydroxylamine in propanil-induced hemolytic anemia.

J. McMillan, D. McMillan, D. Jollow, 1991, Advances in experimental medicine and biology.

Correlation between extent of liver damage in fulminant hepatic necrosis and complexing of circulating group-specific component (vitamin D-binding protein).

W. M. Lee, P. Goldschmidt-Clermont, D. Emerson, 1987, The Journal of laboratory and clinical medicine.

Formation of free radicals and protein mixed disulfides in rat red cells exposed to dapsone hydroxylamine.

R. Crouch, D. McMillan, D. Jollow, 1997, Free radical biology & medicine.

The role of cytochrome P-450 in N-hydroxylation of 2-acetylaminofluorene.

S. Thorgeirsson, D. Jollow, J. Mitchell, 1973, Molecular pharmacology.

Vasopressin stimulates thromboxane synthesis in isolated hamster hepatocytes: relation to hepatocyte calcium content.

P. Halushka, D. Jollow, R. Burch, 1983, Prostaglandins.

Role of oxidant stress in lawsone-induced hemolytic anemia.

D. McMillan, D. Jollow, Snehal D Sarvate, 2004, Toxicological sciences : an official journal of the Society of Toxicology.

Increased incidence of isoniazid hepatitis in rapid acetylators: possible relation to hydrazine metabolites

U. Thorgeirsson, W. Potter, J. Timbrell, 1975, Clinical pharmacology and therapeutics.

Acetaminophen-induced hepatic necrosis. IV. Protective role of glutathione.

B B Brodie, J R Gillette, B. Brodie, 1973, The Journal of pharmacology and experimental therapeutics.

Induction of peroxisome proliferation in cultured hepatocytes by a series of halogenated acetates.

J. McMillan, D. Jollow, Jennie L. Walgren, 2004, Toxicology.

THE BIOGENESIS OF MITOCHONDRIA

A. Linnane, D. Jollow, D. Jollow, 1968, The Journal of Cell Biology.

Dapsone-induced hemolytic anemia.

D. McMillan, D. Jollow, T. Bradshaw, 1995, Drug metabolism reviews.

Role of dapsone hydroxylamine in dapsone-induced hemolytic anemia.

D. Jollow, S. Grossman, 1988, The Journal of pharmacology and experimental therapeutics.

Effects of sulfur-amino acid-deficient diets on acetaminophen metabolism and hepatotoxicity in rats.

D. Jollow, V. Price, 1989, Toxicology and applied pharmacology.

Rapid and sensitive method for the microassay of nitrosobenzene plus phenylhydroxylamine in blood.

J. H. Harrison, D. Jollow, 1983, Journal of chromatography.

Acetylation rates and monthly liver function tests during one year of isoniazid preventive therapy.

U. Thorgeirsson, D. Jollow, J. Mitchell, 1975, Chest.

Biological Reactive Intermediates

D. Parke, R. Snyder, C. Witmer, 1977, Springer US.

Contribution of aniline metabolites to aniline-induced methemoglobinemia.

J. H. Harrison, D. Jollow, D J Jollow, 1987, Molecular pharmacology.

Dapsone-induced hemolytic anemia: effect of N-hydroxy dapsone on the sulfhydryl status and membrane proteins of rat erythrocytes.

J. Simson, D. Jollow, S. Grossman, 1992, Toxicology and Applied Pharmacology.

Use of the NIH shift to determine the relative contribution of competing pathways of aniline metabolism in the rat.

D. Jollow, S. Grossman, 1986, Drug metabolism and disposition: the biological fate of chemicals.

Application of Cryopreserved Human Hepatocytes in Trichloroethylene Risk Assessment: Relative Disposition of Chloral Hydrate to Trichloroacetate and Trichloroethanol

J. McMillan, D. Hoel, L. Mohr, 2006, Environmental health perspectives.

Galactosamine hepatotoxicity: effect of galactosamine on glutathione resynthesis in rat primary hepatocyte cultures.

J. McMillan, D. Jollow, 1992, Toxicology and applied pharmacology.

Metabolic activation of furosemide to a chemically reactive, hepatotoxic metabolite.

W. L. Nelson, W. Potter, D. Jollow, 1976, The Journal of pharmacology and experimental therapeutics.

Modulation of macrophage functioning abrogates the acute hepatotoxicity of acetaminophen

D. Laskin, D. Jollow, C. Gardner, 1995, Hepatology.

Macrophage enhancement of galactosamine hepatotoxicity using a rat hepatocyte culture system.

J. McMillan, D. Jollow, 1995, Research communications in molecular pathology and pharmacology.

Mechanisms of fasting-induced potentiation of acetaminophen hepatotoxicity in the rat.

D. Jollow, D J Jollow, V F Price, 1987, Biochemical pharmacology.

Biochemical Aspects of Toxic Metabolites: Formation, Detoxication, and Covalent Binding

D. Jollow, Carole L. Smith, 1977 .

Chemical analysis and hemolytic activity of the fava bean aglycon divicine.

K. Schey, D. McMillan, D. Jollow, 1993, Chemical research in toxicology.

Primaquine-Induced Hemolytic Anemia: Susceptibility of Normal versus Glutathione-Depleted Rat Erythrocytes to 5-Hydroxyprimaquine

D. McMillan, D. Jollow, Z. Bowman, 2004, Journal of Pharmacology and Experimental Therapeutics.

Toxin Production by Gambierdiscus toxicus Isolated from the Florida Keys

D. Jollow, T. B. Higerd, J. Babinchak, 2008 .

Resolution of Ciguatera-Associated Toxins Using High-Performance Liquid Chromatography ( HPLC )

P. Scheuer, D. Jollow, T. B. Higerd, 2008 .

Comparative pharmacokinetics and tissue distribution of primaquine enantiomers in mice

B. Tekwani, L. Walker, P. Fasinu, 2022, Malaria Journal.

Hydroxylamines and hemolytic anemia.

J. H. Harrison, D. Jollow, S. Grossman, 1986, Advances in experimental medicine and biology.

Cellular glutathione as a protective agent against 4-hydroperoxycyclophosphamide cytotoxicity in K-562 cells

R. Stuart, D. Jollow, J. Oatis, 2008, Cancer Chemotherapy and Pharmacology.

Oxidative stress, glucose-6-phosphate dehydrogenase and the red cell.

D. McMillan, D. Jollow, 2001, Advances in experimental medicine and biology.

Dapsone-induced hemolytic anemia: effect of dapsone hydroxylamine on sulfhydryl status, membrane skeletal proteins and morphology of human and rat erythrocytes.

R. Budinsky, J. Simson, D. McMillan, 1995, The Journal of pharmacology and experimental therapeutics.

Dapsone-induced hemolytic anemia: role of glucose-6-phosphate dehydrogenase in the hemolytic response of rat erythrocytes to N-hydroxydapsone.

R. Budinsky, D. Jollow, S. Grossman, 1995, The Journal of pharmacology and experimental therapeutics.

Identification of free radicals produced in rat erythrocytes exposed to hemolytic concentrations of phenylhydroxylamine.

R. Crouch, D. McMillan, D. Jollow, 1995, Free radical biology & medicine.

Role of glutathione in the in vitro synergism between 4-hydroperoxy-cyclophosphamide and cisplatin in leukemia cell lines.

R. Stuart, D. Jollow, R H Peters, 1991, Cancer research.

The role of N-hydroxyphenetidine in phenacetin-induced hemolytic anemia.

D. Jollow, C. B. Jensen, 1991, Toxicology and applied pharmacology.

Glutathione thresholds in reactive metabolite toxicity.

D. Jollow, 1980, Archives of toxicology. Supplement. = Archiv fur Toxikologie. Supplement.

Primaquine-induced hemolytic anemia: formation and hemotoxicity of the arylhydroxylamine metabolite 6-methoxy-8-hydroxylaminoquinoline.

R. Budinsky, D. McMillan, D. Jollow, 2001, The Journal of pharmacology and experimental therapeutics.

The mechanisms of cobalt chloride-induced protection against acetaminophen hepatotoxicity.

D. Jollow, S. Roberts, V. Price, 1986, Drug metabolism and disposition: the biological fate of chemicals.

Tolerance to Nitroglycerin in Vascular Smooth Muscle Cells Is Not Affected by the Level of Intracellular Glutathione or L-Cysteine

D. Jollow, M. Castresana, Li-Ming Zhang, 1995, Anesthesia and analgesia.

Comparative metabolism and tolerability of racemic primaquine and its enantiomers in human volunteers during 7-day administration

B. Tekwani, L. Walker, M. Elsohly, 2023, Frontiers in Pharmacology.

Biological Reactive Intermediates V

I. Cohen, S. Zderic, H. Greim, 1996, Advances in Experimental Medicine and Biology.

Effect of L-ascorbic acid on acetaminophen-induced hepatotoxicity and covalent binding in hamsters. Evidence that in vitro covalent binding differs from that in vivo.

D. Jollow, M. Miller, 1984, Drug metabolism and disposition: the biological fate of chemicals.

Mechanism of fasting-induced suppression of acetaminophen glucuronidation in the rat.

D. Jollow, S. Spaethe, V. Price, 1986, Advances in experimental medicine and biology.

Anomalous susceptibility of the fasted hamster to acetaminophen hepatotoxicity.

D. Jollow, V. Price, M. Miller, 1986, Biochemical pharmacology.

Pharmacokinetic considerations in toxicity testing.

D. Jollow, S. Roberts, S. Longacre, 1982, Drug metabolism reviews.

Biochemical basis for dose response relationships in reactive metabolite toxicity.

D. Jollow, S. Roberts, V. Price, 1981, Advances in experimental medicine and biology.

Arylamine-induced hemolytic anemia: electron spin resonance spectrometry studies.

R. Crouch, D. McMillan, D. Jollow, 1991, Advances in experimental medicine and biology.

Acute compartment syndrome of the lower limb following childbirth: a case report

D. Jollow, S. Coulton, Sally Bourne, 2020, Journal of Medical Case Reports.

The biogenesis of mitochondria. 7. The effects of glucose repression on the lipid and enzyme composition of mitochondria from yeast.

A. Linnane, D. Jollow, H. Lukins, 1968, Australian Journal of Experimental Biology and Medical Science.

Biological Reactive Intermediates—II

D. Parke, R. Snyder, D. Jollow, 2012, Advances in Experimental Medicine and Biology.

Primaquine-Induced Hemolytic Anemia: Formation of Free Radicals in Rat Erythrocytes Exposed to 6-Methoxy-8-hydroxylaminoquinoline

D. McMillan, D. Jollow, L. J. Bolchoz, 2002, Journal of Pharmacology and Experimental Therapeutics.

Relationship between sulfotransferase activity and susceptibility to acetaminophen-induced liver necrosis in the hamster.

D. Jollow, M. Miller, 1987, Drug metabolism and disposition: the biological fate of chemicals.

THE BIOGENESIS OF MITOCHONDRIA

A. Linnane, D. Jollow, G. M. Kellerman, 1968, The Journal of cell biology.

Hepatic necrosis caused by furosemide

W. Potter, D. Jollow, J. Hinson, 1974, Nature.

Biological reactive intermediates : formation, toxicity, and inactivation

D. Jollow, 1977 .