D. Jiang

发表

Epicardial progenitors contribute to the cardiomyocyte lineage in the developing heart

Bin Zhou, William T. Pu, Sadakatsu Ikeda, 2008, Nature.

Modeling the mitochondrial cardiomyopathy of Barth syndrome with iPSC and heart-on-chip technologies

Gang Wang, Ronald J A Wanders, Kevin Kit Parker, 2014 .

Letm1, the mitochondrial Ca2+/H+ antiporter, is essential for normal glucose metabolism and alters brain function in Wolf–Hirschhorn syndrome

D. Clapham, C. Clish, Linlin Zhao, 2013, Proceedings of the National Academy of Sciences.

RyR 2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca 2 release ( SOICR )

Heping Cheng, Dongmei Yang, B. Xiao, 2004 .

Diffusion of single cardiac ryanodine receptors in lipid bilayers is decreased by annexin 12.

N. Publicover, J. Hall, D. Jiang, 2004, Biophysical journal.

Carvedilol and its new analogs suppress arrhythmogenic store overload–induced Ca2+ release

H. Duff, A. Gillis, Heping Cheng, 2011, Nature Medicine.

Functional reconstitution of the mitochondrial Ca2+/H+ antiporter Letm1

D. Clapham, Christopher Miller, Linlin Zhao, 2014, The Journal of general physiology.

Genome-Wide RNAi Screen Identifies Letm1 as a Mitochondrial Ca2+/H+ Antiporter

D. Clapham, Linlin Zhao, D. Jiang, 2009, Science.

Reduced Threshold for Luminal Ca2+ Activation of RyR1 Underlies a Causal Mechanism of Porcine Malignant Hyperthermia*

Huihui Kong, D. Jiang, B. Fruen, 2008, Journal of Biological Chemistry.

Endoplasmic reticulum Ca2+ measurements reveal that the cardiac ryanodine receptor mutations linked to cardiac arrhythmia and sudden death alter the threshold for store-overload-induced Ca2+ release.

N. Demaurex, D. Jiang, D. Hunt, 2008, The Biochemical journal.

Three-dimensional Localization of Divergent Region 3 of the Ryanodine Receptor to the Clamp-shaped Structures Adjacent to the FKBP Binding Sites*

T. Wagenknecht, Fei Li, D. Jiang, 2003, The Journal of Biological Chemistry.

Enhanced Store Overload–Induced Ca2+ Release and Channel Sensitivity to Luminal Ca2+ Activation Are Common Defects of RyR2 Mutations Linked to Ventricular Tachycardia and Sudden Death

Huihui Kong, B. Xiao, D. Jiang, 2005, Circulation research.

Endoplasmic reticulum Ca 2 + measurements reveal that the cardiac ryanodine receptor mutations linked to cardiac arrhythmia and sudden death alter the threshold for store-overload-induced Ca 2 + release

N. Demaurex, D. Jiang, D. Hunt, 2008 .

Loss of luminal Ca2+ activation in the cardiac ryanodine receptor is associated with ventricular fibrillation and sudden death

D. Jiang, Wenqian Chen, Ruiwu Wang, 2007, Proceedings of the National Academy of Sciences.

RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR).

Heping Cheng, Dongmei Yang, B. Xiao, 2004, Proceedings of the National Academy of Sciences of the United States of America.

Enhanced Basal Activity of a Cardiac Ca2+ Release Channel (Ryanodine Receptor) Mutant Associated With Ventricular Tachycardia and Sudden Death

B. Xiao, D. Jiang, S. Chen, 2002, Circulation research.

Characterization of a novel mutation in the cardiac ryanodine receptor that results in catecholaminergic polymorphic ventricular tachycardia

M. Gollob, D. Birnie, D. Jiang, 2010, Channels.

Functional Consequence of Protein Kinase A-dependent Phosphorylation of the Cardiac Ryanodine Receptor

Wenjun Xie, Xianhua Wang, Heping Cheng, 2007, Journal of Biological Chemistry.

Smooth Muscle Tissues Express a Major Dominant Negative Splice Variant of the Type 3 Ca2+ Release Channel (Ryanodine Receptor)*

B. Xiao, D. Jiang, S. Chen, 2003, Journal of Biological Chemistry.

Isoform-dependent Formation of Heteromeric Ca2+ Release Channels (Ryanodine Receptors)*

T. Wagenknecht, T. Murayama, Y. Sei, 2002, The Journal of Biological Chemistry.

Modeling the mitochondrial cardiomyopathy of Barth syndrome with iPSC and heart-on-chip technologies

George A. Truskey, Megan L. McCain, George M. Church, 2014, Nature Medicine.