Robert N. Correll

发表

Genetic lineage tracing defines myofibroblast origin and function in the injured heart

B. Aronow, J. Molkentin, O. Kanisicak, 2016, Nature Communications.

Thrombospondin-3 augments injury-induced cardiomyopathy by intracellular integrin inhibition and sarcolemmal instability

J. Molkentin, M. Maillet, J. Karch, 2019, Nature Communications.

alpha1G-dependent T-type Ca2+ current antagonizes cardiac hypertrophy through a NOS3-dependent mechanism in mice.

S. Houser, H. Nakayama, J. Molkentin, 2009, The Journal of clinical investigation.

Fibroblast-specific TGF-β-Smad2/3 signaling underlies cardiac fibrosis.

J. Molkentin, O. Kanisicak, R. Vagnozzi, 2017, The Journal of clinical investigation.

Disruption of valosin-containing protein activity causes cardiomyopathy and reveals pleiotropic functions in cardiac homeostasis

J. Molkentin, Robert N. Correll, Matthew J. Brody, 2019, The Journal of Biological Chemistry.

Inhibition of PKC (cid:1) / (cid:2) With Ruboxistaurin Antagonizes Heart Failure in Pigs After Myocardial Infarction Injury

S. Houser, R. Hajjar, J. Molkentin, 2011 .

Integrative Physiology Inhibition of PKC / With Ruboxistaurin Antagonizes Heart Failure in Pigs After Myocardial Infarction Injury Short Communication

S. Houser, R. Hajjar, J. Molkentin, 2011 .

Inhibition of PKC&agr;/&bgr; With Ruboxistaurin Antagonizes Heart Failure in Pigs After Myocardial Infarction Injury

S. Houser, R. Hajjar, J. Molkentin, 2011, Circulation research.

CaMKII does it again: even the mitochondria cannot escape its influence.

J. Molkentin, Robert N. Correll, 2013, Circulation research.

Mitsugumin 29 regulates t-tubule architecture in the failing heart

Didier X. P. Brochet, J. Molkentin, Jianjie Ma, 2017, Scientific Reports.

Genetic Analysis of Connective Tissue Growth Factor as an Effector of Transforming Growth Factor β Signaling and Cardiac Remodeling

J. Molkentin, J. V. van Berlo, J. Rabinowitz, 2015, Molecular and Cellular Biology.

Genetic analysis of CTGF as an effector of TGFβ signaling and cardiac

J. Molkentin, J. Rabinowitz, A. Leask, 2015 .

A Caveolae-Targeted L-Type Ca2+ Channel Antagonist Inhibits Hypertrophic Signaling Without Reducing Cardiac Contractility

S. Houser, J. Molkentin, Hui Gao, 2012, Circulation research.

The Mitochondrial Calcium Uniporter Selectively Matches Metabolic Output to Acute Contractile Stress in the Heart.

Jianyi(Jay) Zhang, D. Bers, J. Molkentin, 2015, Cell reports.

Overlapping and differential functions of ATF6α versus ATF6β in the mouse heart

J. Molkentin, V. Prasad, H. Khalil, 2019, Scientific Reports.

Overexpression of the Na+/K+ ATPase &agr;2 But Not &agr;1 Isoform Attenuates Pathological Cardiac Hypertrophy and Remodeling

D. Bers, J. Molkentin, S. Despa, 2014, Circulation Research.

Rem GTPase interacts with the proximal CaV1.2 C-terminus and modulates calcium-dependent channel inactivation

D. Andres, S. Crump, B. Finlin, 2010, Channels.

The RGK family of GTP-binding proteins: regulators of voltage-dependent calcium channels and cytoskeleton remodeling.

D. Andres, B. Finlin, Robert N. Correll, 2008, Cellular signalling.

L-type calcium channel alpha-subunit and protein kinase inhibitors modulate Rem-mediated regulation of current.

E. Schroder, D. Andres, S. Crump, 2006, American journal of physiology. Heart and circulatory physiology.

Analysis of the Complex between Ca2+ Channel β-Subunit and the Rem GTPase*

D. Andres, S. Crump, B. Finlin, 2006, Journal of Biological Chemistry.

A Tension-Based Model Distinguishes Hypertrophic versus Dilated Cardiomyopathy

J. Seidman, S. Houser, M. Regnier, 2016, Cell.

Y Ears of Study Have Made It Clear That Na Molecular Medicine Rationale: the Na Atpase (nka) Directly Regulates Intracellular Na

D. Bers, J. Molkentin, S. Despa, 2022 .

STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy.

S. Houser, J. Molkentin, J. V. van Berlo, 2015, Journal of molecular and cellular cardiology.

Transient Receptor Potential Channels Contribute to Pathological Structural and Functional Remodeling After Myocardial Infarction

S. Houser, M. Madesh, J. Molkentin, 2014, Circulation research.

CIB1 is a Regulator of Pathological Cardiac Hypertrophy

H. Drexler, Weiping Yuan, J. Molkentin, 2010, Nature Medicine.

Cell-specific ablation of Hsp47 defines the collagen-producing cells in the injured heart

J. Molkentin, R. Vagnozzi, V. Prasad, 2019, JCI insight.

Regulation of L-type Ca2+ Channel Activity and Insulin Secretion by the Rem2 GTPase*

A. Mosley, D. Andres, S. Crump, 2005, Journal of Biological Chemistry.

Overlapping and differential functions of ATF6α versus ATF6β in the mouse heart

J. Molkentin, V. Prasad, H. Khalil, 2019, Scientific Reports.

Cardiac-specific deletion of protein phosphatase 1β promotes increased myofilament protein phosphorylation and contractile alterations.

A. Nairn, J. Molkentin, R. Vagnozzi, 2015, Journal of molecular and cellular cardiology.

Decreased cardiac L-type Ca²⁺ channel activity induces hypertrophy and heart failure in mice.

Donald M Bers, Steven R Houser, Franz Hofmann, 2012, The Journal of clinical investigation.

Plasma Membrane Targeting Is Essential for Rem-mediated Ca2+ Channel Inhibition*

D. Andres, B. Finlin, Robert N. Correll, 2007, Journal of Biological Chemistry.

Mitsugumin 29 regulates t-tubule architecture in the failing heart

Didier X. P. Brochet, J. Molkentin, Jianjie Ma, 2017, Scientific Reports.

Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart

S. Houser, J. Molkentin, Robert N. Correll, 2017, Cardiovascular research.

UltraRapid Communication A Caveolae-Targeted L-Type Ca 2 Channel Antagonist Inhibits Hypertrophic Signaling Without Reducing Cardiac Contractility

S. Houser, J. Molkentin, Hui Gao, 2017 .

Calmodulin binding is dispensable for Rem-mediated Ca2+ channel inhibition

D. Andres, Robert N. Correll, J. Satin, 2008, Molecular and Cellular Biochemistry.

Analysis of the Rem2 - voltage dependant calcium channel beta subunit interaction and Rem2 interaction with phosphorylated phosphatidylinositide lipids.

D. Andres, B. Finlin, Robert N. Correll, 2008, Cellular signalling.

Analysis of the complex between Ca2+ channel beta-subunit and the Rem GTPase.

D. Andres, S. Crump, B. Finlin, 2006, The Journal of biological chemistry.

Assessing wound closure in mice using skin-punch biopsy

Robert N. Correll, Chuong Bui, Mary B. Rowland, 2023, STAR protocols.

Overexpression of the Na+/K+ ATPase &agr;2 But Not &agr;1 Isoform Attenuates Pathological Cardiac Hypertrophy and Remodeling

D. Bers, J. Molkentin, S. Despa, 2014, Circulation research.

CaMKII does it again: even the mitochondria cannot escape its influence.

J. Molkentin, Robert N. Correll, 2013, Circulation research.

CaMKII Determines Mitochondrial Stress CaMKII Does It Again Even the Mitochondria Cannot Escape Its Influence Commentaries on Cutting Edge Science

Robert N. Correll, Jeffery D. Molkentin, Sangeetha, 2013 .

Regulation of cardiac fibroblast cell death by unfolded protein response signaling

Robert N. Correll, Mary B. Rowland, Patrick E. Moore, 2024, Frontiers in physiology.