F. Melet

Fli-1 is required for murine vascular and megakaryocytic development and is hemizygously deleted in patients with thrombocytopenia.

R Favier, A. Hart, R. Favier, 2000, Immunity.

An immunological renal disease in transgenic mice that overexpress Fli-1, a member of the ets family of transcription factor genes.

A. Bernstein, L. Zhang, M. Fritzler, 1995, Molecular and cellular biology.

An immunological renal disease in transgenic mice that overexpress Fli-1, a member of the ets family of transcription factor genes

A. Bernstein, L. Zhang, M. Fritzler, 1995, Molecular and cellular biology.

Negative and Translation Termination-Dependent Positive Control of FLI-1 Protein Synthesis by Conserved Overlapping 5′ Upstream Open Reading Frames in Fli-1 mRNA

F. Morlé, C. Gonnet, J. Starck, 2000, Molecular and Cellular Biology.

Generation of a novel Fli-1 protein by gene targeting leads to a defect in thymus development and a delay in Friend virus-induced erythroleukemia

A. Bernstein, L. Zhang, F. Melet, 1996, Molecular and cellular biology.

The carbonic anhydrase II gene, a gene regulated by thyroid hormone and erythropoietin, is repressed by the v-erbA oncogene in erythrocytic cells.

J. Samarut, B. Pain, F. Melet, 1990, The New biologist.