A. Lewis

发表

Molecular driving forces determining potassium channel slow inactivation

Anthony Lewis, Benoît Roux, Eduardo Perozo, 2007, Nature Structural &Molecular Biology.

RNA Interference Reveals That EndogenousXenopus MinK-related Peptides Govern Mammalian K+ Channel Function in Oocyte Expression Studies*

G. Abbott, A. Anantharam, A. Lewis, 2003, The Journal of Biological Chemistry.

A critical role for the inward rectifying potassium channel Kir7.1 in oligodendrocytes of the mouse optic nerve

A. Butt, A. Lewis, M. Papanikolaou, 2020, Brain Structure and Function.

Oxygen sensing by human recombinant large conductance, calcium-activated potassium channels. Regulation by acute hypoxia.

C. Peers, P. Kemp, A. Lewis, 2003, Advances in experimental medicine and biology.

Spadin Selectively Antagonizes Arachidonic Acid Activation of TREK-1 Channels

A. Lewis, Ruolin Ma, 2020, Frontiers in Pharmacology.

Acute hypoxia occludes hTREK-1 modulation: re-evaluation of the potential role of tandem P domain K+ channels in central neuroprotection.

C. Peers, C. Chapman, H. Meadows, 2003, The Journal of physiology.

Hypoxia inhibits human recombinant large conductance, Ca2+‐activated K+ (maxi‐K) channels by a mechanism which is membrane delimited and Ca2+ sensitive

C. Peers, P. Kemp, A. Lewis, 2002, The Journal of physiology.

Airway chemotransduction: from oxygen sensor to cellular effector.

C. Peers, I. O'kelly, P. Kemp, 2002, American journal of respiratory and critical care medicine.

Regulation of the Kv2.1 Potassium Channel by MinK and MiRP1

G. Abbott, T. K. Roepke, A. Lewis, 2009, Journal of Membrane Biology.

Regulation of recombinant human brain tandem P domain K+ channels by hypoxia: a role for O2 in the control of neuronal excitability?

C. Peers, P. Kemp, A. Lewis, 2004, Journal of cellular and molecular medicine.

Rapid Report

C. Peers, C. Chapman, H. Meadows, 2003 .

Acute oxygen sensing in cellular models: relevance to the physiology of pulmonary neuroepithelial and carotid bodies.

D. Adriaensen, C. Peers, P. Kemp, 2003, The anatomical record. Part A, Discoveries in molecular, cellular, and evolutionary biology.

TOK channels use the two gates in classical K+ channels to achieve outward rectification

S. Goldstein, L. G. Cuello, A. Lewis, 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

O(2) sensing by airway chemoreceptor-derived cells. Protein kinase c activation reveals functional evidence for involvement of NADPH oxidase.

C. Peers, I. O'kelly, P. Kemp, 2000, The Journal of biological chemistry.

Oxygen sensing by human recombinant tandem-P domain potassium channels.

C. Peers, P. Kemp, A. Lewis, 2003, Advances in experimental medicine and biology.

Recombinant hTASK1 is an O(2)-sensitive K(+) channel.

C. Peers, C. Chapman, H. Meadows, 2001, Biochemical and biophysical research communications.

Combined Antisense and Pharmacological Approaches Implicate hTASK as an Airway O2 Sensing K+Channel*

C. Peers, I. O'kelly, P. Kemp, 2001, The Journal of Biological Chemistry.

Store-operated calcium entry is essential for glial calcium signalling in CNS white matter

A. Butt, A. Lewis, M. Papanikolaou, 2017, Brain Structure and Function.

Glial and neuronal expression of the Inward Rectifying Potassium Channel Kir7.1 in the adult mouse brain

A. Butt, A. Lewis, M. Papanikolaou, 2019, Journal of anatomy.

TREK-1 Channel Expression in Smooth Muscle as a Target for Regulating Murine Intestinal Contractility: Therapeutic Implications for Motility Disorders

J. Swinny, A. Lewis, M. Papanikolaou, 2018, Front. Physiol..

Atomic Constraints between the Voltage Sensor and the Pore Domain in a Voltage-gated K+ Channel of Known Structure

B. Roux, L. Blachowicz, V. Jogini, 2008, The Journal of general physiology.

Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Human Lung Microvascular Endothelial Cells Controls Oxidative Stress, Reactive Oxygen-Mediated Cell Signaling and Inflammatory Responses

Z. Saynor, D. Laight, J. Shute, 2020, Frontiers in Physiology.

KCNE1 and KCNE2 provide a checkpoint governing voltage-gated potassium channel α-subunit composition.

G. Abbott, A. Lewis, Vikram A. Kanda, 2011, Biophysical journal.

KCNE1 and KCNE2 inhibit forward trafficking of homomeric N-type voltage-gated potassium channels.

G. Abbott, A. Lewis, Vikram A. Kanda, 2011, Biophysical journal.

Peter N. Jordan, D. Christini, G. Abbott, 2003, The Journal of Neuroscience.

MinK, MiRP1, and MiRP2 Diversify Kv3.1 and Kv3.2 Potassium Channel Gating*

G. Abbott, A. Lewis, Zoe A. McCrossan, 2004, Journal of Biological Chemistry.

O2-sensitive K+ channels controlling cell excitability

A. Lewis, C. Peers, P. Kemp, 2003 .