H. Imamichi

发表

Parallel evolution of drug resistance in HIV: failure of nonsynonymous/synonymous substitution rate ratio to detect selection.

K. Crandall, H. Lane, C. Kelsey, 1999, Molecular biology and evolution.

HIV-1 infected monozygotic twins: a tale of two outcomes

David Posada, Steven Hirschfeld, Keith A Crandall, 2011, BMC Evolutionary Biology.

Interleukin-27 Enhances the Potential of Reactive Oxygen Species Generation from Monocyte-derived Macrophages and Dendritic cells by Induction of p47phox

H. Lane, T. Imamichi, Deepak Poudyal, 2017, Scientific Reports.

Enhanced effector function of CD8(+) T cells from healthy controls and HIV-infected patients occurs through thrombin activation of protease-activated receptor 1.

D. Follmann, H. Lane, Zonghui Hu, 2013, The Journal of infectious diseases.

IL‐15 acts as a potent inducer of CD4+CD25hi cells expressing FOXP3

H. Lane, I. Sereti, H. Imamichi, 2008, European journal of immunology.

Defective HIV-1 proviruses produce viral proteins

B. Sherman, A. Fauci, J. Adelsberger, 2020, Proceedings of the National Academy of Sciences.

Regulatory T cells in HIV-1 infection: the good, the bad, and the ugly.

H. Lane, H. Imamichi, 2012, The Journal of infectious diseases.

In vivo expansion of CD4CD45RO-CD25 T cells expressing foxP3 in IL-2-treated HIV-infected patients.

J. Metcalf, R. Davey, V. Natarajan, 2005, The Journal of clinical investigation.

Decreased interleukin 7 responsiveness of T lymphocytes in patients with idiopathic CD4 lymphopenia.

R. Dersimonian, J. Brenchley, W. Thompson, 2012, The Journal of infectious diseases.

Cutting Edge: Ku70 Is a Novel Cytosolic DNA Sensor That Induces Type III Rather Than Type I IFN

T. Veenstra, D. Huang, R. Lempicki, 2011, The Journal of Immunology.

I IFN Sensor That Induces Type III Rather Than Type Cutting Edge: Ku70 Is a Novel Cytosolic DNA

T. Veenstra, R. Lempicki, Jun Yang, 2011 .

Than Type I IFN DNA Sensor That Induces Type III Rather Cutting Edge : Ku 70 Is a Novel Cytosolic and Tomozumi Imamichi

T. Veenstra, D. Huang, R. Lempicki, 2011 .

HIV-1 viruses detected during episodic blips following interleukin-7 administration are similar to the viruses present before and after interleukin-7 therapy

M. Lederman, M. Fischl, D. Asmuth, 2011, AIDS.

IL-7-dependent STAT1 activation limits homeostatic CD4+ T cell expansion.

T. Myers, H. Lane, J. O’Shea, 2017, JCI insight.

Cerebrospinal fluid HIV-1 compartmentalization in a patient with AIDS and acute varicella-zoster virus meningomyeloradiculitis.

R. Dewar, H. Lane, D. Hammoud, 2013, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

The CD8+HLA‐DR+ T cells expanded in HIV‐1 infection are qualitatively identical to those from healthy controls

R. Lempicki, H. C. Lane, M. Baseler, 2012, European journal of immunology.

Effects of intermittent interleukin-2 therapy on plasma and tissue human immunodeficiency virus levels and quasi-species expression.

R. Dewar, J. Metcalf, R. Davey, 2000, The Journal of infectious diseases.

Defective HIV-1 proviruses produce novel protein-coding RNA species in HIV-infected patients on combination antiretroviral therapy

R. Dewar, A. Fauci, J. Adelsberger, 2016, Proceedings of the National Academy of Sciences.

The emergence and characterization of macrophage‐tropic SIV/HIV chimeric viruses (SHIVs) present in CD4+ T cell‐depleted rhesus monkeys

Charles R. Brown, H. Imamichi, V. Hirsch, 2003, Journal of leukocyte biology.

Macrophage-Tropic Simian/Human Immunodeficiency VirusChimeras Use CXCR4, Not CCR5, for Infections of Rhesus MacaquePeripheral Blood Mononuclear Cells and AlveolarMacrophages

John P. Moore, H. Lane, A. Suffredini, 2003, Journal of Virology.

Amino acid deletions are introduced into the V2 region of gp120 during independent pathogenic simian immunodeficiency virus/HIV chimeric virus (SHIV) infections of rhesus monkeys generating variants that are macrophage tropic

H. Lane, T. Imamichi, A. Suffredini, 2002, Proceedings of the National Academy of Sciences of the United States of America.

Although Macrophage-Tropic Simian/Human Immunodeficiency Viruses Can Exhibit a Range of Pathogenic Phenotypes, a Majority of Isolates Induce No Clinical Disease in Immunocompetent Macaques

H. Imamichi, A. Buckler-White, T. Igarashi, 2007, Journal of Virology.

Genetic divergence of the dihydrofolate reductase and dihydropteroate synthase genes in Pneumocystis carinii from 7 different host species.

J. Kovacs, L. Ma, A. Sukura, 2001, The Journal of infectious diseases.

Drug resistance during indinavir therapy is caused by mutations in the protease gene and in its Gag substrate cleavage sites

J. Falloon, H. Lane, T. Imamichi, 1997, Journal of virology.

Advances in understanding the evolution of HIV.

D. Posada, K. Crandall, H. Imamichi, 1999, AIDS.

Broad and potent neutralization of HIV-1 by a gp41-specific human antibody

Baoshan Zhang, Peter D. Kwong, John R. Mascola, 2012, Nature.

Decreases in Colonic and Systemic Inflammation in Chronic HIV Infection after IL-7 Administration

M. Lederman, V. Natarajan, J. Routy, 2014, PLoS pathogens.

Loss of Naïve Cells Accompanies Memory CD4+ T-Cell Depletion during Long-Term Progression to AIDS in Simian Immunodeficiency Virus-Infected Macaques

H. Lane, H. Imamichi, V. Hirsch, 2006, Journal of Virology.

The Differential Ability of HLA B*5701+ Long-Term Nonprogressors and Progressors To Restrict Human Immunodeficiency Virus Replication Is Not Caused by Loss of Recognition of Autologous Viral gag Sequences

C. Hallahan, S. Migueles, J. Metcalf, 2003, Journal of Virology.

CD8+ T-cell Cytotoxic Capacity Associated with Human Immunodeficiency Virus-1 Control Can Be Mediated through Various Epitopes and Human Leukocyte Antigen Types

D. Follmann, C. Hallahan, S. Migueles, 2014, EBioMedicine.

Selection Pressure on HIV-1 Envelope by Broadly Neutralizing Antibodies to the Conserved CD4-Binding Site

Richard T. Wyatt, John R. Mascola, Mark Connors, 2012, Journal of Virology.

Chronic Exposure to Type-I IFN under Lymphopenic Conditions Alters CD4 T Cell Homeostasis

H. Lane, Zonghui Hu, S. Durum, 2014, PLoS pathogens.

High-Level Resistance to 3′-Azido-3′-Deoxythimidine due to a Deletion in the Reverse Transcriptase Gene of Human Immunodeficiency Virus Type 1

Tomozumi Imamichi, H. Clifford Lane, Hiromi Imamichi, 2000, Journal of Virology.

Relative Replication Fitness of a High-Level 3′-Azido-3′-Deoxythymidine-Resistant Variant of Human Immunodeficiency Virus Type 1 Possessing an Amino Acid Deletion at Codon 67 and a Novel Substitution (Thr→Gly) at Codon 69

J. Metcalf, J. Falloon, H. Lane, 2000, Journal of Virology.

Rapid Recall Ability of Memory T cells is Encoded in their Epigenome

Suresh Cuddapah, A. Barski, K. Zhao, 2017, Scientific Reports.

STING is an essential mediator of the Ku70-mediated production of IFN-λ1 in response to exogenous DNA

Brad T. Sherman, B. Sherman, H. C. Lane, 2017, Science Signaling.

CD4+ T cell responses to interleukin-2 administration in HIV-infected patients are directly related to the baseline level of immune activation.

R. Dersimonian, J. Metcalf, R. Davey, 2007, The Journal of infectious diseases.

In vivo expansion of CD4+CD45RO–CD25+ T cells expressing foxP3 in IL-2-treated HIV-infected patients

J. Metcalf, R. Davey, V. Natarajan, 2005 .

Resistance to Replication of Human Immunodeficiency Virus Challenge in SCID-Hu Mice Engrafted with Peripheral Blood Mononuclear Cells of Nonprogressors Is Mediated by CD8+T Cells and Associated with a Proliferative Response to p24 Antigen

C. Hallahan, R. Davey, M. Connors, 2000, Journal of Virology.

Quantitation of Viremia and Determination of Drug Resistance in Patients with Human Immunodeficiency Virus Infection

R. Dewar, V. Natarajan, T. Elbeik, 2006 .

Amino Acid Deletion at Codon 67 and Thr-to-Gly Change at Codon 69 of Human Immunodeficiency Virus Type 1 Reverse Transcriptase Confer Novel Drug Resistance Profiles

H. Lane, T. Imamichi, H. Imamichi, 2001, Journal of Virology.

Human immunodeficiency virus type 1 quasi species that rebound after discontinuation of highly active antiretroviral therapy are similar to the viral quasi species present before initiation of therapy.

K. Crandall, R. Dewar, J. Metcalf, 2001, The Journal of infectious diseases.

Prolonged Posttreatment Virologic Control and Complete Seroreversion After Advanced Human Immunodeficiency Virus-1 Infection.

Bryan R. Smith, T. Chun, R. Dewar, 2020, Open forum infectious diseases.

Lack of compartmentalization of HIV-1 quasispecies between the gut and peripheral blood compartments.

R. Dewar, J. Kovacs, P. Mannon, 2011, The Journal of infectious diseases.

Analysis of variation in tandem repeats in the intron of the major surface glycoprotein expression site of the human form of Pneumocystis carinii.

C. Beard, J. Kovacs, G. Kutty, 2002, The Journal of infectious diseases.

Lifespan of effector memory CD4+ T cells determined by replication-incompetent integrated HIV-1 provirus

R. Lempicki, V. Natarajan, J. Adelsberger, 2014, AIDS.

Emergence of protease inhibitor resistance mutations in human immunodeficiency virus type 1 isolates from patients and rapid screening procedure for their detection

J. Falloon, T. Imamichi, N. Salzman, 1996, Antimicrobial agents and chemotherapy.

HIV vaccines induce CD8+ T cells with low antigen receptor sensitivity

N. Frahm, C. Hallahan, S. Migueles, 2023, Science.

IL-7-dependent STAT1 activation limits homeostatic CD4+ T cell expansion.

T. Myers, H. Lane, J. O’Shea, 2017, JCI Insight.

Long-term persistence of transcriptionally active ‘defective’ HIV-1 proviruses: implications for persistent immune activation during antiretroviral therapy

R. Dewar, V. Natarajan, Zonghui Hu, 2023, AIDS.