A. Gosselin

发表

S. Sugden, D. Hazuda, F. Pereyra, 2012, Journal of the International AIDS Society.

Transcriptional profiling reveals molecular signatures associated with HIV permissiveness in Th1Th17 cells and identifies Peroxisome Proliferator-Activated Receptor Gammaas an intrinsic negative regulator of viral replication

C. Tremblay, J. Routy, J. Goulet, 2013, Retrovirology.

Peripheral Blood CCR4CCR6 and CXCR3CCR6 CD4 T Cells Are Highly Permissive to HIV-1 Infection

J. Routy, R. Sékaly, N. Chomont, 2010 .

Permissive to Hiv-1 Infection T Cells Are Highly + Cd4 + Ccr6 + and Cxcr3 + Ccr6 + Peripheral Blood Ccr4

J. Routy, R. Sékaly, F. Diaz-Griffero, 2022 .

IL-17A reprograms intestinal epithelial cells to facilitate HIV-1 replication and outgrowth in CD4+ T cells

C. Tremblay, J. Routy, J. Goulet, 2021, iScience.

Oral abstracts of the 21st International AIDS Conference 18–22 July 2016, Durban, South Africa

Michael I. Jordan, Donald A. Jackson, J. G. Ndirangu, 2016, Journal of the International AIDS Society.

Memory CCR6CD4 T Cells Are Preferential Targets for Productive HIV Type 1 Infection Regardless of Their Expression of Integrin b7

J. Routy, P. Ancuta, N. Grandvaux, 2011 .

LILAC pilot study: Effects of metformin on mTOR activation and HIV reservoir persistence during antiretroviral therapy

J. Routy, J. Goulet, M. Massanella, 2021, EBioMedicine.

Improving HIV Outgrowth by Optimizing Cell-Culture Conditions and Supplementing With all-trans Retinoic Acid

J. Routy, J. Goulet, M. Massanella, 2020, Frontiers in Microbiology.

Supplemental Information Host MicroRNAs-221 and-222 Inhibit HIV-1 Entry in Macrophages by Targeting the CD 4 Viral Receptor

Gilmore, J. Routy, C. Power, 2017 .

Proceedings of the Frontiers of Retrovirology Conference 2016

R. König, D. Elleder, R. Sachidanandam, 2016, Retrovirology.

Nef promotes evasion of human immunodeficiency virus type 1-infected cells from the CTLA-4-mediated inhibition of T-cell activation.

G. Freeman, J. van Grevenynghe, J. Routy, 2015, The Journal of general virology.

Host MicroRNAs-221 and -222 Inhibit HIV-1 Entry in Macrophages by Targeting the CD4 Viral Receptor.

J. Routy, M. Tremblay, C. Power, 2017, Cell reports.

Memory CCR6+CD4+ T Cells Are Preferential Targets for Productive HIV Type 1 Infection Regardless of Their Expression of Integrin β7

J. Routy, N. Grandvaux, A. Gosselin, 2011, The Journal of Immunology.

Expression of Integrin Type 1 Infection Regardless of Their Preferential Targets for Productive HIV T Cells Are + CD 4 + Memory CCR 6 and

J. Routy, N. Grandvaux, A. Gosselin, 2011 .

Identification of novel HIV-1 dependency factors in primary CCR4+CCR6+Th17 cells via a genome-wide transcriptional approach

C. Tremblay, J. Routy, R. Sékaly, 2015, Retrovirology.

Impaired Th17 polarization of phenotypically naive CD4+ T-cells during chronic HIV-1 infection and potential restoration with early ART

C. Tremblay, J. Routy, S. DaFonseca, 2015, Retrovirology.

HIV-1 is rarely detected in blood and colon myeloid cells during viral-suppressive antiretroviral therapy.

J. Routy, É. Cohen, D. Planas, 2019, AIDS.

CD16+ monocytes give rise to CD103+RALDH2+TCF4+ dendritic cells with unique transcriptional and immunological features.

C. Tremblay, J. Routy, J. Goulet, 2018, Blood advances.

New insights into the heterogeneity of Th17 subsets contributing to HIV-1 persistence during antiretroviral therapy

J. Routy, J. Goulet, S. Deeks, 2016, Retrovirology.

Peripheral Blood CCR4+CCR6+ and CXCR3+CCR6+ CD4+ T Cells Are Highly Permissive to HIV-1 Infection

J. Routy, R. Sékaly, F. Diaz-Griffero, 2009, The Journal of Immunology.

HIV persists in CCR6+CD4+ T cells from colon and blood during antiretroviral therapy

J. Routy, É. Cohen, D. Planas, 2016, AIDS.

HIV-1 selectively targets gut-homing CCR6+CD4+ T cells via mTOR-dependent mechanisms.

T. Hope, J. Routy, A. Fassati, 2017, JCI insight.

RALDH Activity Induced by Bacterial/Fungal Pathogens in CD16+ Monocyte-Derived Dendritic Cells Boosts HIV Infection and Outgrowth in CD4+ T Cells

J. Routy, J. Estaquier, É. Cohen, 2021, The Journal of Immunology.

Transcriptional profiling reveals developmental relationship and distinct biological functions of CD16+ and CD16- monocyte subsets

Kuang-Yu Liu, Annie Gosselin, Xiaobo Zhou, 2009, BMC Genomics.

Impaired Th17 polarization of phenotypically naive CD4+ T-cells during chronic HIV-1 infection and potential restoration with early ART

C. Tremblay, J. Routy, S. DaFonseca, 2015, Retrovirology.

Pharmacological Inhibition of PPARy Boosts HIV Reactivation and Th17 Effector Functions, While Preventing Progeny Virion Release and de novo Infection

J. Routy, J. Goulet, J. Richard, 2020, Pathogens & immunity.

Phylogenetic analysis of HIV-1 archived DNA in blood and gut-associated lymphoid tissue in two patients under antiretroviral therapy

J. Routy, H. Fleury, A. Gosselin, 2021, Gut Pathogens.

Identification of novel HIV-1 dependency factors in primary CCR4+CCR6+Th17 cells via a genome-wide transcriptional approach

C. Tremblay, J. Routy, R. Sékaly, 2015, Retrovirology.

Transcriptional profiling reveals molecular signatures associated with HIV permissiveness in Th1Th17 cells and identifies Peroxisome Proliferator-Activated Receptor Gamma as an intrinsic negative regulator of viral replication

C. Tremblay, J. Routy, J. Goulet, 2013, Retrovirology.

The Colocalization Potential of HIV-Specific CD8+ and CD4+ T-Cells is Mediated by Integrin β7 but Not CCR6 and Regulated by Retinoic Acid

C. Tremblay, J. Routy, N. Chomont, 2012, PloS one.

Phylogenetic analysis of HIV-1 archived DNA in blood and gut-associated lymphoid tissue in patients receiving antiretroviral therapy: a study from Provir/Latitude45 project

M. Soares, J. Routy, H. Fleury, 2020, bioRxiv.

Proceedings of the Frontiers of Retrovirology Conference 2016

Sebastian A. Wagner, R. König, D. Elleder, 2016, Retrovirology.

Peripheral Blood CCR4+CCR6+ and CXCR3+CCR6+ CD4+ T Cells Are Highly Permissive to HIV-1 Infection

J. Routy, R. Sékaly, F. Diaz-Griffero, 2009, The Journal of Immunology.

New insights into the heterogeneity of Th17 subsets contributing to HIV-1 persistence during antiretroviral therapy

J. Routy, J. Goulet, S. Deeks, 2016, Retrovirology.

Overt IL-32 isoform expression at intestinal level during HIV-1 infection is negatively regulated by IL-17A

C. Tremblay, J. Routy, A. Landay, 2021, AIDS.

Transcriptional profiling reveals molecular signatures associated with HIV permissiveness in Th1Th17 cells and identifies Peroxisome Proliferator-Activated Receptor Gamma as an intrinsic negative regulator of viral replication

C. Tremblay, J. Routy, J. Goulet, 2013, Retrovirology.

Alterations in Th17 Cells and Non-Classical Monocytes as a Signature of Subclinical Coronary Artery Atherosclerosis during ART-Treated HIV-1 Infection

J. Routy, A. Gosselin, P. Ancuța, 2024, Cells.